Ch. 40 Minor Burns, Sunburns, and Wounds Flashcards
Minor Burns, Sunburns, and Wounds (MBSW)
- Can speed up healing of cuts, burns, and wounds with proper drug selection
- ~450,000/year need medical treatment for burns
- Proper antiseptic and antibiotic use can help healing, minimize scar formation, and prevent secondary infection
MBSW Pathophysiology
- Acute wounds - burns, abrasions, puncture wounds, and lacerations
- Usually from trauma and heal within 1 month
- Chronic wound - any wound that doesn’t heal through normal stages of wound healing
- Require triage and more intense medical care
- Can self treat wounds that don’t extend beyond dermis
- Homeostasis must return quickly after the wound to allow proper healing
- Then healing phases occur (overlap): inflammatory, proliferative, and maturation (remodeling)
- Local healing factors: Tissue perfusion, oxygenation, infection, wound characteristics
- Systemic healing factors: Poor vascularization, bacterial contamination, inadequate nutrition, medical conditions, and medications
- Elderly: delayed healing but equal quality
- Obese: poor perfusion and delayed healing
- Diabetes (uncontrolled) - decreased collagen synthesis, impaired wound contraction, delayed epidermal migration, and decreased chemotaxis/phagocytosis, SHOULD SEE PCP
Burns
Wounds from thermal, electrical, chemical, or UV exposure
Thermal Burns
Skin contact with flames, scalding liquids, hot objects, or from inhaling smoke or hot vapors
Chemical Burns
- Secondary exposure to corrosive or reactive chemicals that cause tissue damage, ulceration, and sloughing
- Necrotic tissue can then act as a reservoir for the chemical and can cause prolonged cutaneous damage
- Remove chemical exposed clothing
- Report to ER for evaluation
Sunburns
- Too much UVA and UVB light exposure from sunlight or tanning beds
- Similar to photosensitive reactions causes from drugs, similar clinical presentations
Abrasions
Rubbing/friction injury to epidermis that extends to uppermost portion of dermis
Punctures
Sharp object that pierces epidermis and can reach into dermis or deeper tissues
Lacerations
Sharp object cutting through the various layers of skin
Inflammatory Stage
- First healing stage
- Body’s immediate response to injury
- Lasts 3-4 days and includes inflammation and hemostasis
- Hemostasis: release of thromboplastin from injured cells that creates a clot to stop the bleeding
- Inflammation: debris and bacteria removed from wound bed, collagen formed, first layer of epithelial cells go cover wound
Proliferative Stage
- Second stage of healing
- Filled with connective tissue and covered by new epithelium
- Starts ~ day 3 and can last 3 weeks
- Forms granulation tissue including connective tissue, capillaries, inflammatory cells
Maturation/Remodeling Stage
- Last stage of healing
- Longest phase starting at about week 3 and peaking at about 60 days post injury
- Continual collagen synthesis/breakdown process where weak collagen is replaced with high strength collagen
Poor Vascularization
- Delays healing
- Results in poor oxygenation and therefore impaired leukocyte activity, decreased collagen, decreased epithelialization, and decreased resistance to infection
- Conditions that decrease perfusion include diabetes, severe anemia, hypotension, peripheral vascular disease, and CHF
Wound Infection
- Deposition/multiplication of organism that cause a host reaction
- Most caused by bacteria like Strep, Staph, and Enterococcus
- Delays collagen synthesis and epithelialization
- Prolongs inflammation and causes tissue destruction
Adequate Nutrition
- Needed for wound repair
- Proteins, carbs, vitamins, and trace elements are all needed for collagen production/energy
- Vitamins commonly used for healing
Vitamin C
- Many roles in healing
- Deficiencies effect many aspect of tissue repair
- Decrease collagen synthesis, decrease fibroblasts, decrease angiogenesis, and increase capillary fragility
- Impaired immunity can also occur from its deficiency
Vitamin E
- Anti-inflammatory
- Reduces scarring
- Widely used for healing
MBSW Clinical Presentation
- Determined by the depth of damage
- 4 stages of wounds
Stage I
- Only epidermis involved
- No skin layer loss
- Redness, nonblanching, unbroken, nonblistering skin
- Minor sunburn: superficial, erythema, slight edema due to increased blood flow to affected skin
- Starts 4 hours post exposure and peaks between 12-24 hours
- Treatment: Avoid additional injury, symptomatic pain/fever relief, mostly self-treated and heal in 3-6 hours
Stage II
- Blistering, involves all epidermis and some of dermis
- Involves skin breakage, pain, edema, erythema, and possible drainage from the wound
- Severe sunburn: blisters, peeling skin, pain, edema, skin tenderness, possible chance of bacterial infection
- Systemic symptoms: vomiting, low grade fever, chills, weakness, shock when larger body portions involved
- Leaves skin more sensitive to sunburns for weeks
- Painful, sensitive to temperature and air, large blisters, blanching (loss of blood vessels), and more intense pain to no sensation occurs
- More prone to infection which can cause worse severity, depth, or both. Increases likelihood of delayed healing and scarring
- Only self treat when on an adult and cover 1-2% BSA
- Seek medical attention if it hasn’t healed or worsened in 2-3 weeks
Stage III
- Loss of entire epidermis, dermis, and dermal appendages, possibly even subcutaneous tissue
- Causes death of full skin thickness in the area
- Results in dry, leathery area that is painless and insensate
- Painful, bleeds profusely, needs hospitalization
Stage IV
- Extension of stage II
- Involves subcutaneous tissue and underlying muscle, tendon, and bone
- SEE DOCTOR/ER