CH 11 Apraxia of Speech Flashcards

1
Q

what is apraxia?

A

problem with motor programming of speech movements

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2
Q

what is dysarthria?

A

problems with muscles/movement

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3
Q

what is aphasia?

A

a linguistic/language problem

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4
Q

AOS may co-occur with….?

A

oral and limb apraxia

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5
Q

AOS has also been called….?

A

speech apraxia and oral verbal apraxia

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6
Q

AOS also confused with ____ bc the phonologic impairment similarity and similar sites of lesions

A

Brocas aphasia

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7
Q

what is responsible for programming speech and how?

A

motor speech programmer transforms abstract phonemes to neural code from which motor programming can occur that determines specific muscle movements needed

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8
Q

where are motor speech plans held?

A

buffer area

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9
Q

where is MSP located

A

left hemisphere. structures involved are located in parietal frontal and related subcortical circuits

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10
Q

linguistic input to MSP comes from

A

perisylvan area in l. hemisphere. includes temporoparietal cortex, insula, thalamus, basal ganglia

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11
Q

insula is located where?

A

under temoral and frontal lobes

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12
Q

Dr. Robin has found what area to be involved in apraxia?

A

brodmann’s area 6

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13
Q

areas primarily involved with MSP?

A

premotor area (specifically Brocas area, supplemental motor area, basal ganglia, and cerebellar circuits), parietal lobe somatosensory cortex and supramarginal gyrus, insula, basal ganglia (striatum)

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14
Q

most common sites of lesions in AOS are…..

A

Broca’s area, insula, basal ganglia

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15
Q

nonspeech characteristics that may accompany AOS?

A

right sided weakness, spasticity, Babinski reflex, hyperactive stretch reflexes, limb apraxia

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16
Q

what causes apraxia?

A

tumors, stroke, trauma (primarily stroke)

17
Q

pts with apraxia complain of…..

A

know what they want to say but words don’t come out right

18
Q

is swallowing associated with AOS?

A

no. if it is present, may indicate dysarthria and dysphagia

19
Q

clinical findings for oral mech exam

A

if dysarthria does not co-occur, gag reflex, swallowing, and chewing should be WNL and note wether or not oral sensation is impaired

20
Q

clinical findings for non verbal oral apraxia

A

can perform involuntary movements but not involuntary

21
Q

always assess for what when evaluating apraxia…?

A

limb apraxia, NVOA, aphasia

22
Q

what can cause more severe breakdowns in individuals with AOS and Broca’s than in normal speakers?

A

Delayed Auditory Feedback

23
Q

to assess speech, what tasks would you administer?

A

voluntary (conversational, narratives, reading), SMR’s and multisyllabic words and sentences, increasingly complex words (endear, endearing, endearingly), imitation tasks (to assess aphasia)

24
Q

what to look for when assessing for AOS?

A

pts may do okay of automatic and overlearned (days of week), false articulatory starts (self correct), groping for articulators, articulatory distortions (with paraphasias seen in aphasia there are no distortions), rate and prosodic problems, non fluent

25
Q

according to Julie wambaugh, what are the most important diagnostic criteria for AOS?

A

slowed rate of speech, sound errors (distortions, consistent, predictable), prosodic abnormalities

26
Q

a person with severe AOS may not initially be able to phonate and be mute for 2 weeks. if lasts longer, diagnoses may be…..

A

severe, aphasia, anarthria, psychogenic mutism

27
Q

what are the characteristics of aphasia?

A

telegrammatic speech, free flowing phonemic jargon or phonological errors, semantic/syntactic errors, comprehension, reading, writing affected, diadochokinetic rates normal, variable self monitoring

28
Q

what are the characteristics of apraxia

A

speech halting with disrupted prosody, dysfluent with articulatory erros, no semantic or syntactic errors, no problems with comprehension, reading or writing, diadochokinetic rates usually poor, self monitoring is good