Case Studies - Psychopathology Flashcards
Ost et al (1991)
Procedure: 81 blood & 56 needle phobics were asked to complete a questionnaire on their memories of possible origins of their phobias.
Results:
- 52% attributed onset to conditioning experiences
- 24% recalled vicarious experiences
- 7% instruction/information
- 17% could not remember any specific onset scenarios
Conclusion: Majority attributed to conditioning (learned response)
Barlow & Durand (1995)
Findings: 50% of people with driving phobia could remember a specific incident which triggered their fear.
Conclusion: Phobias are learned
Bandura & Rosenthal (1966)
Supported Social Learning Explanation
Procedure: Model acted as though he were in pain when a buzzer sounded.
Findings: Participants demonstrated an acquired fear emotional response to buzzer later on.
Conclusion: Phobia can be learnt and therefore unlearnt.
Dinardo (1988)
Found that not everyone who is bitten by a dog develops a fear of dogs - some phobias can be explained by evolution. It makes sense to be afraid of heights, fire, spiders etc. as they are dangerous and avoiding them helps us survive.
Billet et al (1998)
Results: MZ twins are twice as likely to develop OCD if their twin had the disorder than with DZ twins.
Conclusion: There is a genetic explanation for OCD, even if CC rates are never 100%. Shared environment & behaviourism might be better explanations.
It is too simplistic to say there is a gene for OCD - it is more likely to be learnt, maybe by social learning theory.
Soomro et al (2008)
Procedure: Meta-analysis of 17 studies on SSRIs for OCD patients.
Results: SSRIs > placebos in reducing OCD symptoms up to 3 months after treatment
Conclusion: SSRIs are effective long-term.
Koran et al (2007)
Procedure: Meta-analysis of studies into OCD treatments
Results: CBT should be used before drug treatments for long term help; drug therapy is easy but short-term effective - people can relapse within a few weeks if medication is stopped.
Aylward et al (1996)
Aim: Investigated structural or functional abnormality of the caudate nucleus in patients with OCD.
Procedure: 24 patients with adult-onset OCD and 21 matched controls had MRI scans.
Results: No structural/functional differences in the c nucleus found.
Conclusion: No evidence of abnormality discovered in c nucleus as an explanation for OCD.