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BDS2 CLINICAL Caries Symposium > Caries and Oral Biology > Flashcards

Caries and Oral Biology Flashcards

1
Q

What is the etiopathology of caries

A

dental plaque
diet
tooth
time

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2
Q

why was time added to the etiopathology of caries

A

there is a need of time to generate caries but it is not just how long but how frequently is there access to sugar

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3
Q

what is the best adapted bacteria

A

bacteria that is most fit for installing itself onto the surface of the tooth

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4
Q

why are teeth coated in protein

A

otherwise the surface will be too harsh and damage the mucosa

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5
Q

how have bacteria adapted to the surface of the tooth

A

they can attach well to proteins

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6
Q

what are the secondary elements

A 
saliva
buffer capacity
clearance rate
diet composition/frequency
microbial species
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7
Q

what are the external factors that drive secondary elements

A 
social class
education
income
behaviors 
attitude 
knowledge
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8
Q

what is caries defined as

A

demineralization of the tooth surface due to acid produced by acidogenic bacteria.

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9
Q

when does remineralization occur

A

when there is no sugar present

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10
Q

why is an exposed lesion less dangerous than an unexposed one

A

if exposed we can clean the area

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11
Q

why is the surface of a lesion not the most demineralized

A

The area that is first to remineralise is the surface, the sub surface takes time to remineralise while the surface both demineralizes and remineralises very quickly.

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12
Q

why should we not probe white spot lesions

A

can cause a fracture

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13
Q

do white spot lesions have carious dentine

A

no

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14
Q

what happens to the rods in enamel lesions

A

the rods do not dissapaear but they become thinner and the gaps between the rods become more present. The rod length is preserved

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15
Q

what crystallites are more prone to demineralization

A

crystallites that are sideways to the rod

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16
Q

what happens in the first stage of inter proximal caries

A

plaque builds up under the contact area
plaque produce acid which causes a white spot lesion
demineralization establishes a response to dentine and the dentine has an initial sclerosis which is not visible under a radiograph

17
Q

what happens in the second stage of approximal caries

A

demin of enamel and start of demin of dentine
structure has not changed much
sclerosis has started to progress

18
Q

what happens in the third stage of approximal caries

A

continuing demineralisation
initial phase of tertiary dentine
at this stage we have loss of mineral but not loss of tissue

19
Q

what happens in the fourth stage of approximal caries

A

cavitiation
the sclerotic dentine progresses
presence of microorganisms inside dentine

20
Q

what is the appearance of a white spot lesion

A

chalk white colour

not bright but as they remineralize they become brighter

21
Q

what are the proteolytic bacteria

A

s. sanguinis

s. gordonii

22
Q

what is the characteristic of an inactive lesion

A

smooth

23
Q

what is the change in pH of inactive lesions similar to

A

that of sound teeth

24
Q

why is there an ecological shift in active lesions

A

S.mutans has a pump as it is acidogenic bacteria which lives well in acidic conditions. It produces acid and the other bacterias die down overtime.

25
Q

describe the first stage of pits and fissure caries

A

start of demineralization of enamel

26
Q

describe the second stage of pits and fissure caries

A

Demineralisation of enamel goes further

There is already sclerotic dentine

27
Q

describe the third stage of pits and fissure caries

A

Spreads quickly in dentine because the tubules branch at the ADJ (ballooning effect)

28
Q

why are root caries harder to treat

A

Restoring root caries is difficult because restoration teeth is based on the surrounding material being hard enough to glue to our material. On these lesions there is no enamel. The enamel is at the cervical region. Therefore, there is exposed dentine and materials don’t bond well to dentine

29
Q

where is fluoride concentraiton highest

A

surface of the tooth

30
Q

why is pits and fissure caries different

A

the rods in the fissures are organized in a different way

31
Q

how are the rods in the fissures organized

A

perpendicular to the surface

32
Q

where is the buffer in the pits and fissures

A

surface region

33
Q

where is the demineralisation of the the pits and fissures greatest

A

core central regions

highest concentration of acid

BDS2 CLINICAL Caries Symposium (6 decks)

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