Cardiovascular Diseases Flashcards

1
Q

What is dilated cardiomyopathy

A

Second most common cardiac disease in dogs

Common in giant breeds (dobermans and Great Danes)

Weakened heart muscle and stretched chambers > less effective pumping (decreases outflow = decreased perfusion) (back up = pulmonary hypertension)

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2
Q

What are the biggest concerns with dilated cardiomyopathy

A

Pulmonary edema and decreased perfusion

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3
Q

What are the presenting complaints with dilated cardiomyopathy

A

Weak, shaking, lethargy, exercise intolerance, syncope

Coughing and breathing problems

Sudden collapse

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4
Q

What are the treatment goals with dilated cardiomyopathy

A

Increase perfusion
Decrease heart rate
Correct pulmonary hypertension
Never stop meds (Vetmedin increases Contractility strength) (viagra)
Low salt diets
Controlled exercise
Taurine may have an effect

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5
Q

What is the prognosis of dilated cardiomyopathy

A

No cure, only supportive care

Quality of life can be made good but they will die

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6
Q

What is mitral valve disease

A

Degenerative

Number one most common cardiac disease in dogs

Can be made worse with dental disease

Common in small breeds

The valves (most the mitral valve) become thick and enlarged. Blood is unable to pass through which results in back flow and leaking of blood and turbulence (murmur)

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7
Q

Describe the pathology of mitral valve disease

A

Damaged valves don’t close properly

Leaking valve = turbulence = murmur

Heart has to work hard to pump the same amount of blood around which can lead to HCM

Initially there are no signs until it progresses

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8
Q

Why are owners told to journal and monitor resting respiratory rates in patients with mitral valve disease

A

A gradual increase indicates the disease is progressing

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9
Q

What is hypertrophic cardiomyopathy (HCM)

A

The thickening of the heart muscles from the heart having to work harder to pump blood around. Results in smaller chambers

Initially looks normal because thickening of the muscles begins inwards

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10
Q

Who does hypertrophic cardiomyopathy present in

A

Most common cardiac disease in cats (primary disease in main coons and rag dolls or it can be secondary to hyperthyroidism)

Also seen in small dogs secondary to mitral valve disease

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11
Q

Describe the pathology of hypertrophic cardiomyopathy

A

Ventricular volume is reduced, so heart can’t pump out as much blood (decreased perfusion)

Blood backs up (pulmonary hypertension and edema and pleural effusion)

Mostly happens on the left side of the heart

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12
Q

What are the presenting complaints of hypertrophic cardiomyopathy

A

Emergency: exercise intolerance, coughing, weakness, shaking, syncope, collapse, open mouth breathing and increased effort in cats, cyanosis and pale MM

Not common to see syncope in cats

Don’t stress them out they can die

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13
Q

What are the treatment goals of hypertrophic cardiomyopathy

A

Slow down the heart rate

Decrease pulmonary hypertension (meds and decrease stress)

Don’t stop medications

Low salt diets

Oxygenation chambers

Treat hyperthyroidism if it is secondary to it in cats

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14
Q

What is the prognosis of hypertrophic cardiomyopathy

A

If showing signs of heart failure the prognosis is very poor (just prolonging death)

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15
Q

Describe heart worm (dirofilaria immitis)

A

Transmitted through mosquitoes

Can cause damaged and blockage to the heart

Not prevalent in Saskatchewan (seen with travel)

Signalment: dog > cats > ferrets

Worms can migrate to various areas in the body

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16
Q

Describe the pathology of heart worm

A

Worms obstruct pulmonary arteries

Heart worm emboli blocks blood Flow in smaller vessels

Right sided heart failure (ascities, peripheral edema, jugular distension)

17
Q

What is essential for animals in areas with high heart worm prevalence

A

Heart worm preventative medications

18
Q

What is saddle thrombus

A

Thromboembolic disease in cats (can be seen with HCM)

Clot formation coming from the left side of the heart lodges in the distal end of the body casing paralysis in the hind limbs

19
Q

Describe saddle thrombus

A

Extremely painful, no pulse felt in hind limb, cold hind feet, pink pads will be cyanotic and can lead to necrosis

20
Q

What is the clinical presentation of saddle thrombus

A

Depends on where the thromboembolism is lodged (can be anywhere) Most common is branch of iliac artery

Causes hind end weakness and paralysis (emergency)

21
Q

What is the treatment of saddle thrombus

A

Not often successful

Try to manage with analgesia

22
Q

What is the prevention of saddle thrombus

A

Drugs to decrease risk of clot formation (more effective than treatment)