Cardiovascular Flashcards
What is medical management of hypertrophic cardiomyopathy?
Negative inotropic agents (e.g. beta blockers, verapamil)
What is the treatment for sustained ventricular tachycardia in a stable patient?
Antiarrhythmic drugs - usually IV amiodarone
What is alcoholic cardiomyopathy?
Diagnosis of exclusion: LV dysfunction (dilated) in pt with hx of alcohol abuse in whom no other potential causes of cardiomyopathy (e.g. coronary artery disease, valvular heart disease) are identified. Cessation of EtOH use can improve or even normalize LV function over time
What are symptoms of an ateriovenous fistula?
Widened pulse pressure, strong peripheral arterial pulsation (e.g. brisk carotid upstroke), systolic flow murmur, tachycardia, and often flushed extremities if fistula involves the extremity. PMI will be displaced to the left and ECG will show left ventricular hypertrophy
What kind of bundle branch block can severe right heart strain cause (e.g. massive PE)?
RBBB
What is Beck’s triad?
Hypotension, elevated JVP, and muffled heart sounds - signs of cardiac tamponade
What happens to the PMI in pts with large pericardial effusions?
Becomes nonpalpable
What is Dressler syndrome?
A secondary form of pericarditis that occurs in the setting of injury to the heart or the pericardium (e.g. following MI). It consists of fever, pleuritic pain, pericarditis and/or a pericardial effusion. Treated with NSAIDS
What is/are the most common location(s) for the ectopic foci that cause A-fib?
The pulmonary veins
What medication is preferred to manage aortic dissection?
IV beta blocker (e.g. labetalol, proranolol, esmolol) - reduces HR, SBP, and LV contractility putting less stress on the dissection
What EKG findings are associated with LVH?
High voltage QRS complexes, lateral ST segment depression, and lateral T wave inversion
What valvular disease may result from carcinoid syndrome?
Tricuspid regurgitation
What are features of a supravalvular aortic stenosis?
Systolic murmur similar to AS except is best heard at the first right intercostal space, unequal carotid pulses, differential blood pressure in upper extremities (high-pressure jet in ascending aorta), and a palpable thrill in the suprasternal notch
Why does left ventricular hypertrophy lead to increased myocardial oxygen demand with exertion?
Patients end up with coronary artery stenosis as an associated anomaly.
What is LV wall stress a measure of?
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