Cardiovascular Flashcards
Precordium
area on anterior chest overlying the heart and lower thorax
Order of heart valves
tricuspid, pulmonic, mitral, aortic
Try Pulling My Arm
Systole
S1 (tricuspid, mitral valves close) aortic and pulmonic valves open, ventricles contract
Diastole
S2 (pulmonic, aortic valves close), tricuspid and mitral valves open, blood fills ventricles, atria contract
Preload
end of diastolic pressure, ventricles stretched to greatest dimensions
Afterload
the stress /tension developed in wall of left ventricle during ejection
S1
tricuspid, mitral valves close; loudest at apex of heart
S2
pulmonic, aortic valves close; loudest at base of heart (Erb’s point in particular)
S3
blood fills ventricles
S4
atria contract
Mumur
extra heart sounds, disruption of blood flow
Point of maximal impulse (PMI)
generated by the apex maybe produced by enlarged ventricle, dilated aorta/dialted pulmonary artery, or an LV wall motion abnormality. Should be less than 1 cm in diameter
Thrill
a fine, plapable, rushing vibration (a palpable murmur usually over the base of the heart, indicates turbulence or disruption of expected blood flow). Signifies that the murmur is grade 4 or higher
Heave
a more pronunced lift /lift- RV hypertrophy
Gallop
S3, when you hear this third heart sound it mimics a “gallop”
Rub
friction rub, inflammation of pericardial sac (pericarditis), heard toward apex
Click
high pitched sounds that occur at maximal opening of aortic/pulmonary valves, heard just after S1
Snap
opening of mitral valve
Aortic surface point
Right 2nd ICS; hear S2
Pulmonic surface point
Left 2nd and 3rd ICS; hear S2
Erb’s point
Left 3rd ICS; BEST hearing of S2
Tricuspid surface point
Left 4th ICS/Lower left sternal border
Mitral surface point
Left 4-5th ICS in midclavicular line; S1 heard here at apex
Physiologic splitting
i. During expiration, the pulmonic valve closes only 30 ms after the aortic valve, S2 is heard as one sound
ii. During inspiration, the chest expands (vacuum -> inspiration), this decrease in pressure increases venous return to the right atrium and ventricle, the extra blood volume causes a delay in ejection, and the pulmonic valve closes later. The delay can be heard as a split S2 - especially common in young people.