cardiovascular Flashcards
define heart rate and give the measurement
number of times the heart contracts per minute- measured in beats/min
define stroke volume and give the measurement
volume of blood ejected from the left ventricle per beat- measured in ml
define cardiac output and give the measurement
volume of blood ejected from the left ventricle per minute- measured in L/min
average HR values for an untrained individual at rest, sub-max and maximal intensity
rest= 70 bpm
sub-max= 100bpm
max= 220-age
average SV values for an untrained individual at rest, sub-max and maximal intensity
rest= 70ml
sub-max= 100ml
max= 100ml but will decrease
average CO values for an untrained individual at rest, sub-max and maximal intensity
rest= 5 L/min
sub-max= 10 L/min
max= 20L/min
average HR values for a trained individual at rest, sub-max and maximal intensity
rest= 50bpm
sub-max= 120bpm
max= 220-age
average SV values for a trained individual at rest, sub-max and maximal intensity
rest= 100ml
sub-max= 200ml
max= 200ml but will decrease
average CO values for a trained individual at rest, sub-max and maximal intensity
rest= 5 L/min
sub-max= 24L/min
max= 40 L/min
define myogenic
the capacity of the heart to generate its own electrical impulse, which causes the cardiac muscles to contract
describe the stages of the conduction system
- the SA node located in the right atrium wall generates the electrical impulse and fires it through the atrial walls causing to to contract
- the AV node receives this impulse and delays it for approx. 0.1 second to allow the atria to finish contracting. before releasing the impulse to the bundle of His
- the bundle of His located in the septum of the heart, splits the impulse in two ready to be distributed through each separate ventricle
- the purkyne fibres distribute the impulse through the ventricle walls causing them to contract
atrial systole
the atria contract, forcing remaining blood into the ventricles
ventricular systole
the ventricles contract, increasing the pressure closing the AV valves to prevent backflow into the atria- SL valves are forced open as blood is ejected from the ventricles into the aorta and pulmonary artery
diastole
as the atria and then the ventricles relax, they expand drawing blood into the atria- pressure in the atria increase which opens the AV valves- blood passively enters the ventricles- SL valves are closed to prevent blood from leaving the heart
how is the conduction system linked to the cardiac cycle during atrial systole
the SA node fires the electrical impulse across the atria to the AV node which delays the impulse causing the atria to contract and as a result the AV valves are forced open and remaining blood is passed into the ventricles ensuring that the semi-lunar valves are shut
how is the conduction system linked to the cardiac cycle during diastole
there is no electrical impulse and the cardiac muscles relax- SL valves close and the atria begin to fill with blood which opens the AV valves allowing the blood to enter the ventricles
how is the conduction system linked to the cardiac cycle during ventricular systole
the bundle of His splits and passes the impulse through two branches to the purkyne fibers in both ventricle walls- ventricular muscles contract which means that the AV valves are shut and blood is pushed into the arteries forcing SL valves open until ventricles finish contracting
what is sub-maximal exercise
low-moderate intensity within a performers aerobic capacity or below the anaerobic threshold
what is maximal exercise
high intensity above a performers aerobic capacity, which will take a performer to exhaustion- associated with anaerobic work and fatigue
how does our heart rate respond to sustained sub-max intensity
-heart rate has an anticipatory rise before the run due to the release of adrenaline
-at the start the performers heart rate will have a steep rise but will then plateau when the intensity has become constant as oxygen supply meets the demand
-if the intensity of the work increases/decreases their heart rate will adjust accordingly
-at the start of recovery, there will be a rapid decrease in heart rate before it begins to gradually lower back to resting value
how is the heart rates response different when performing maximal intensity work
-no steady state is reached as the supply of oxygen will never meet the demands of the performers muscles’
-recovery time takes longer for HR to return to its pre-exercise value due to the higher intensity
how does our stroke volume respond to exercise
stroke volume increases in proportion to exercise intensity until a plateau is reached at a sub-maximal intensity due to:
-increased heart rate towards maximal intensities does not allow enough time for the ventricles to fill with blood completely when in the diastolic phase- limiting the Frank-Starling mechanism
how is stroke volume able to increase
- increased venous return- volume of blood that returns to the heart. during exercise VR increases meaning that there is greater volume of blood returning to the heart and filling the ventricles
- frank-starling mechanism- shows us how SV is dependent on VR. an increased volume of blood returning to the heart leads to an increased end-diastolic volume in the ventricles and therefore greater stretch of the ventricular walls. this greater stretch increases the force of the ventricular contraction, ejecting a larger volume of blood from the ventricles.
how does stroke volume respond during recovery
-SV remains elevated during recovery to maintain blood flow to the muscles in order to remove lactic acid and co2.
-it reduces to its pre-exercise value gradually
-a cool down helps to maintain SV