CARDIOTOCOGRAPHY Flashcards
Contributory factors to fetal morbidity and mortality following CTG
- CTG technique/equipment
- Errors of interpretation of CTG
- Failure to act upon suspicion of pathological CTG
- Fetal blood sampling
- CTG and blood sampling–other
T/F: Staff tasked with CTG interpretation must have evidence of annual training
T
T/F: Key management decisions should not be based on CTG alone
T
Monitoring tool to identify hypoxia
`Cardiotocograph
T/F: CTG during labour is associated with reduced rates of neonatal seizures
T
T/F: increase in caesarean sections and instrumental vaginal births with CTG in labour
T
T/F: with CTG there is no clear differences in cerebral palsy, infant mortality or other standard measures of neonatal wellbeing.
T
CTG pattern with stress
- Hypoxia begins with deceleration
- Acceleration disappear
- Baseline HR increases
CTG pattern with compensated stress
- Stable baseline
- normal variability
- deceleration deeper and wider
CTG pattern with decompensation
- Unstable Baseline
- changes in variability
4features of fetal heart rate
- Baseline rate
- Baseline variability
- Presence or absence of decelerations (and concerning characteristics of variable decelerations if present)
- Presence of accelerations
T/F: If there is a stable baseline fetal heart rate between 110and 160beats/minute and normal variability, continue usual care as the risk of fatal acidosis is low
T
T/F:The presence of fetal heart rate accelerations, even with reduced baseline variability, is generally a sign that the baby is healthy
T
Normalbaseline rate is
110 – 160 bpm
T/F: Baseline HR is gestation dependent
T
Non reassuring baseline rate are –and –
baseline bradycardia and baseline tachycardia
Baseline bradycardia is
100 - 109 bpm
Baseline tachycardia is
160 - 179bpm
AbnormalHR
<100 bpm
>180 bpm
Acceleration is
Increase in FHR above baseline of at least 15bpm for at least 15seconds
T/F: Absence of FHR acceleration is a good predictor of fetal metabolic acidemia or hypoxic injury
F. Poor predictor