Cardiology Flashcards
S4 coincides with which part of the ECG
P wave
S4 is due to atrial contraction against a stiff, non-compliant ventricle
e.g. Aortic stenosis
GI bleeding + murmur
What murmur?
Aortic stenosis
Heyde’s syndrome = GI bleeding (colonic angiodysplasia) + Aortic stenosis
What coronary artery supplies posterior heart
posterior heart
Posterior interventricular artery (PIVA)
RCA (90%)
or LCx (10%)
Also supplies AVN
What coronary artery supplies SAN and AVN
What is coronary artery dominance
Coronary artery dominance = determined by which artery gives rise to posterior interventricular artery (PIVA)
- Right Coronary Artery (90%) ==> Right dominance
- LCx (10%) ==> Left dominance
- Equal contribution ==> Equal dominance
What is the flow of coronary veins
All cardiac veins* meet at the coronary sinus –> empties into right atrium
*Except anterior cardiac veins which empty directly into the RA
What are the waves of the JVP
What causes the following JVP pathology
large a waves
Cannon a waves
Absent a wave
Prominent X descent
Giant V waves
Slow Y desscent
What is Kussmaul’s sign
What causes it
Kussmaul’s sign = paradoxical ↑ JVP during inspiration
Caused by Constrictive pericarditis
ACS
DVLA rules?
Acute coronary syndrome
- If PCI –> 1 week off driving
- If CABG or conservatively managed –> 4 weeks off driving
Wellens syndrome
What is it?
ECG findings?
Osborn wave
What is it?
What causes it?
Epsilon wave
What is it?
What causes it?
ECG changes in hyperkalaemia
hyperkalaemia
Peaked T waves
Loss of P waves
Prolonged PR interval
Bradycardia
Broad QRS –> BBB –> Sine wave –> VF –> Asysole
If bradycardia, blocks (AV block, BBB), bizarre QRS complexes –> think Hyperkalaemia
ECG changes in hypokalaemia
hypokalaemia
TWI
U waves
Prolonged PR interval
ST depression
Long QU interval
ECG changes in hypercalcaemia
hypercalcaemia
Shortening of QTc
Osborn waves / J waves
ECG changes in hypocalcaemia
hypocalcaemia
Prolonged QTc
ECG changes in hypomagnesaemia
hypomagnesaemia
Prolonged QTc
ECG changes in TCA overdose
Treatment
TCA overdose
Broad QRS complex
Dominant secondary R’ wave in aVR (M appearance in aVR)
Sinus tachycardia
Treatment –> IV Sodium Bicarbonate + Activated Charcoal
Digoxin toxicity
Sx
Tx
Digoxin toxicity
Abdominal pain
Yellow-hue vision
Haloes
Ventricular ectopics
Bradycardia
Tx: Digibind (Digoxin-specific antibody)
ECG changes in incomplete and complete tri-fascicular block
ECG changes in bifascicular block
ECG criteria for Bifascicular block:
- RBBB
- Either Right axis deviation or Left axis deviation
Prolonged QTc
- Causes
- Treatment
Tip: “Hypos, Anti(medication), MI and Congenital”
Tx: β blockers
Treatment of tachyarrhythmia
Prophylaxis for recurrent SVT
Prophylaxis for recurrent SVT
(1) β blockers
(1) Catheter ablation
Tx of atrial flutter
(1) Rate control, Anticoagulation, Rhythmn control
(2) Catheter ablation of cavotricuspid isthmus (curative)
What is Ebstein’s anomaly
What is it caused by
Presentation
Associations
Ebstein’s anomaly = congenital defect where tricuspid valve is found lower in the R ventricle –> large RA, small RV = “atrialisation of right ventricle”
Caused by Lithium exposure in utero (Bipolar MUM)
Features
- Tricuspid regurgitation / Pansystolic murmur
Associations
- PFO
- ASD
- WPW syndrome
Features of VT
Broad complex tachycardia
Capture beats
Fusion beats
Tx for VT
- Pulseless VT
- –> Cardiac Arrest
- VT with pulse
- Haemodynamically unstable
- Synchronised DC cardioversion
- Haemodynamically stable
- IV Amiodarone
- Haemodynamically unstable
What is C/I in VT
Avoid CCBs (Verapamil) in VT
Blocking the AVN may trigger VF –> Cardiac arrest
Tx for Torsades de Pointes
IV Magnesium sulphate 1-2g
ECG: Convex / coved ST elevation in V1-V3 followed by TWI
Diagnosis? Cause?
How to make ECG signs more apparent
Treatment?
Brugada syndrome
Mutation in SCN5A gene
ECG changes more apparent post-flecainide or post-ajmaline administration
Tx: ICD
Cardiac MRI: fibrofatty tissue within the myocardium
Diagnosis? ECG?
Treatment?
Complications?
Arrhythmogenic right ventricular cardiomyopathy
Right ventricular myocardium is replaced by fatty and fibrofatty tissue
ECG: Epsilon wave, TWI V1-V3
Management: Treat arrhythias, Catheter ablation to prevent VT, ICD
Complications: 2nd most common cause of sudden cardiac death in the young (HCM is 1st)
Exercise or emotion-induced VT
Diagnosis?
Cause?
Catecholaminergic polymorphic ventricular tachycardia
Autosomal dominant mutation in RYR2
Onset < 20 years old
Exercise/Emotion –> VT
Bradyarrhythmias - tx
If adverse features –> IV Atropine 500 micrograms +/- Repeat +/- Transcutaneous pacing +/- IV Isoprenaline +/- IV Adrenaline
If no adverse features
- If at risk of asystole (2nd/3rd AV block) –> See above
- If low risk –> Observation
Tx of AV block