Cardiology Flashcards
First degree block
Constant prolonged PRI >0.20 sec
Management: none, observation
Associated with: aging, ischemia, inflammation, cardiomyopathies, digitalis
2nd degree Mobitz I Heart Block
Wenckebach
Progressive PRI lengthening —> dropped QRS
Management: Asx: observation, +/- cardiac consult
Sx: ATROPINE (anticholinergic), epinephrine, +/- pacemaker
2nd degree Mobitz II heart block
Constant, prolonged PRI —> dropped QRS
Usually block in bundle of HIS
Management: atropine or temporary pacing
Definitive treatment: permanent pacemaker
3rd degree AV block
AV dissociation: P waves not related to QRS
Decreased output (since all P waves not followed by QRS)
Management: temporary pacing —> permanent pacemaker
Definitive txt: permanent pacemaker
Treatment of choice in antidromic atrioventricular reciprocating tachycardia
Wolff-Parkinson White Wide-complex tachycardia (>120ms) Stable: procainamide, amiodarone Unstable: cardioversion Definitive treatment: ablation of the accessory pathway.
Do NOT use drugs that slow conduction through the AV node, such as adenosine, calcium channel blockers, and beta blockers. May enhance conduction through the accessory tract, leading to vfib.
Mitral stenosis
History of rheumatic heart disease
Sx: exertional dyspnea, hemoptysis
On auscultation: loud S1, opening snap, low-pitched, rumbling diastolic apical murmur
Aortic stenosis
Patient will be older
History of diabetes, hypertension
Sx: dyspnea, chest pain, syncope
PE: harsh crescendo-decrescendo systolic murmur that radiates to the carotids, paradoxically split S2, S4 gallop
Most commonly caused by degenerative calcification
Treatment is aortic valve replacement
Comments: murmur decreases with Valsalva
Which murmur is most commonly associated with Marfan’s syndrome?
Mitral valve prolapse
Mitral regurgitation
Patient with a history of ischemic heart disease, endocarditis, MI, trauma
Acute sx: dyspnea, pulmonary edema, cardiogenic edema
Chronic sx: asymptomatic until CHF onset, S3
PE: blowing holosystolic murmur, best heard at apex with radiation to axilla, pulmonary edema, cardiogenic edema
Diagnosis: TTE echo
Acute txt: nitroprusside, dobutamine, intra-aortic balloon pump, emergency surgery
Chronic txt: anticoagulation, CHF rx, valve replacement
LBBB
large wide R wave in lead I
negative wave (QS or rS) in lead V1.
ST elevation V1-V3
New LBBB + Chest Pain = MI until proven otherwise
RBBB
wide S wave in lead I
Triphasic QRS pattern in lead V1
Acute Rheumatic Fever
Patient with a history of GAS infection
Complaining of fever, red skin lesions on the trunk and proximal extremities, and small, non-tender lumps located over the joints
JONES:
Strep: anti-streptolysin O, anti-DNase B, positive throat culture or positive rapid antigen test
Major: carditis, migrating Arthritis, chorea, subcutaneous nodules, erythema marginatum (non-pruritic rash sparing face)
Minor: fever, arthralgia, elevated ESR or CRP, prolonged PR interval
Txt: abx, NSAIDs
Bacterial Endocarditis
Patient will be complaining of fever, rash, cough, and myalgias
PE: Fever, Roth spots, Osler nodes, Murmur, Janeway lesions, Anemia, Nailbed hemorrhages, Emboli (FROM JANE)
Dx: echocardiography and Duke criteria
Most commonly caused by:
IVDA: S. aureus, tricuspid
Native valve: Staphylococcus aureus, Streptococcus viridans (m/c in previously diseased), mitral
Treatment is antibiotics
Comments: GI malignancy: S. bovis
Dental prophylaxis in some cases (amoxicillin)
Dx of endocarditis
- Blood cultures: 3 sets 1 hour apart
- EKG
- Echo (TTE first, TEE more sensitive)
- Labs: CBC - leukocytosis, anemia; elevated ESR/RF
Duke Criteria:
Major: 1)sustained bacteremia with 2 + blood cultures by organism known to cause endocarditis; 2)endocardial involvement via echo or new valvular regurgitation (AR or MR)
Minor: 1)Predisposing condition (abnormal valves, IVDA, indwelling catheters, etc); 2)Fever; 3)vascular and emboli phenomena; 4)immunologic phenomena; 5)+ blood dx not meeting major criteria; 6)echo not meeting major criteria (worsening of existing murmur)
Tetralogy of Fallot
Patient with a history of episodes of cyanosis (tet spells) and squatting for relief
PE: harsh holosystolic murmur LUSB, RV heave
Echo: gold standard: pulmonic stenosis, RVH, overriding aorta, and VSD (PROVe)
CXR: “boot-shaped” heart
Txt: PGE1 infusion followed by surgery
Comments: Most common cyanotic congenital heart disease
Acute Pericarditis
Sx: pleuritic persistent chest pain radiating to the back, worse when lying back, improved when leaning forward
PE: tachycardia and pericardial friction rub
ECG: PR depression, PR elevation (aVR and V1), diffuse ST-segment elevation (concave)
Most commonly caused by idiopathic, then viral (Coxsackie)
Treatment is NSAIDs, colchicine
Hypertrophic Cardiomyopathy
Sx: dyspnea on exertion (most common presenting symptom)
PE: harsh crescendo-decrescendo systolic murmur which increases in intensity with Valsalva maneuver and decreases with squatting
Diagnosis: echocardiography
Most commonly caused by an autosomal dominant genetic defect
Treatment: refraining from vigorous physical activity, beta-blockers or calcium channel blockers
Hyperkalemia
Patient with a history of renal failure, DKA, rhabdomyolysis, tumor lysis
Complaining of lethargy, weakness, paralysis
PE will show bradycardia, hypotension, cardiac dysrhythmia
ECG will show peaked T waves, prolonged PR, wide QRS
Treatment is calcium gluconate, insulin, albuterol, bicarbonate
Mitral valve prolapse
Patient will be complaining of palpitations, dyspnea, non-exertional CP, and fatigue. Most cases are asymptomatic.
PE will show midsystolic click and late systolic murmur
↑ Preload → ↓ murmur; ↓ preload → ↑ murmur
Marfan syndrome, Ehlers-Danlos
Diagnosis is made by echo
Tricuspid regurgitation
Patient will be complaining of signs of right-sided heart failure - ascites, edema, RUQ pain
PE will show JVD and a blowing holosystolic murmur best heard at the left sternal border that become louder during inspiration
Diagnosis is made by echo
Most commonly caused by RV dilatation
Associated with Ebstein’s Anomaly
Atrial septal defect
PE will show wide, fixed split S2
Most commonly caused by persistent ostium secundum
Comments: May lead to Eisenmengers syndrome
Myocarditis
Patient will be complaining of fatigue, fever, chest discomfort, dyspnea, and palpitations
PE will show tachycardia that is disproportionate to fever or discomfort
Echocardiogram will show decreased ventricular ejection fraction with hypokinesis and wall motion abnormalities
Gold standard for diagnosis is an endomyocardial biopsy
Ventricular tachycardia
> 3 consecutive ectopic ventricular beats
Monomorphic, polymorphic
Bidirectional: digoxin toxicity
Wide complexes
Pulseless: immediate defibrillation
Unstable: synchronized cardioversion
Stable: procainamide, amiodarone, synchronized cardioversion (refractory)
If unsure, manage all wide complex tachycardias as ventricular tachycardia
Acyanotic congenital heart disease
ASD: fixed split S2
VSD: most common congenital heart defect, harsh holosystolic murmur at LLSB, complication: Eisenmenger syndrome (longstanding left-to-right shunt becomes a right-to-left shunt)
PDA: machine-like murmur, Rx: indomethacin
Coarctation of the aorta: upper extremity > lower extremity BP/pulse, CXR: rib notching, Rx: PGE1
Diagnosis is made by echocardiography
