Cardiology Flashcards
You arrive approximately 8 minutes after a 51-year-old male collapsed at a family event. After determining that he is unresponsive and apneic, you should:
A) assess for a carotid pulse for 5 to 10 seconds.
B) give 2 rescue breaths and check for a pulse.
C) begin CPR, starting with chest compressions.
D) immediately assess the patient’s cardiac rhythm.
Answer: A
After determining that an adult patient is unresponsive and apneic, you should assess for a carotid pulse for at least 5 seconds but no more than 10 seconds. If the patient has a pulse, open the airway and provide rescue breathing. If the patient does not have a pulse, begin CPR (starting with chest compressions), then open the airway and give 2 rescue breaths. Assess the patient’s cardiac rhythm as soon as an AED is available.
A 57-year-old man complains of a dull pain in his chest. He tells you that he has had two heart attacks within the past 3 years and is currently being treated for hypertension. After administering supplemental oxygen, you should:
A) obtain a complete set of vital signs.
B) administer up to 324 mg of baby aspirin.
C) inquire about any medications he is taking.
D) give him 0.4 mg of nitroglycerin sublingual.
Answer: B
According to current Emergency Cardiac Care guidelines, a patient with a suspected acute coronary syndrome should be given supplemental oxygen (as needed to maintain an SpO2 of greater than 94%) and aspirin (160 to 325 mg) as soon as possible. Aspirin blocks the formation of thromboxane A2, which inhibits platelet aggregation and coronary vasoconstriction. Nitroglycerin (NTG), up to three 0.4-mg sublingual doses, is also indicated for patients with suspected cardiac-related chest pain or discomfort; however, you should first establish intravenous access. NTG is a vasodilator and could cause a drop in his blood pressure. Vital signs should be obtained early; however, oxygen and aspirin are interventions that should be performed immediately.
You analyze a cardiac arrest patient’s rhythm with the automated external defibrillator (AED) and receive a no shock message. What should you do next?
A) Give 2 breaths and resume chest compressions
B) Resume CPR starting with chest compressions
C) Check for a pulse for no longer than 10 seconds
D) Continue rescue breathing and transport at once
Answer: B
If you receive a shock advised message from the AED, you should deliver the shock at once and then begin or resume CPR, starting with chest compressions. If you receive a no shock message from the AED, you should immediately resume CPR, starting with chest compressions, and reanalyze the patient’s cardiac rhythm after 2 minutes. Checking for a pulse immediately after defibrillation causes an unnecessary delay in performing chest compressions. You should continue the cycles of CPR, analyzing the cardiac rhythm and delivering a shock (if indicated), and immediately resuming CPR until paramedics arrive or the patient starts to move.
Pale or ashen skin in a patient experiencing an acute myocardial infarction indicates:
A) poorly oxygenated blood caused by respiratory failure.
B) poor cardiac output and reduced peripheral perfusion.
C) hypertension and increased peripheral vascular resistance.
D) peripheral vasodilation caused by reduced sympathetic tone.
Answer: B
Pale or ashen skin indicates reduced peripheral perfusion. In a patient experiencing an acute myocardial infarction (AMI), this is often caused by poor cardiac output. Cyanosis, a blue or purple tint to the skin, is a sign of poor oxygenation of circulating blood; it occurs in patients with respiratory failure. Hypertension typically causes flushing (red) skin, not pallor. Peripheral vasodilation, as seen in patients with heat-related illnesses, also causes flushing of the skin. AMI is typically associated with increased, not decreased, sympathetic nervous system tone.
In contrast to atherosclerosis, arteriosclerosis:
A) is a thickening of the arterial walls, resulting in a loss of elasticity and a reduction in blood flow.
B) occurs when the coronary arteries constrict, which significantly decreases blood flow to the heart.
C) is a disorder in which cholesterol builds up and forms a plaque inside the walls of blood vessels.
D) occurs when the inner layers of the arterial wall become separated, allowing blood to flow between the layers.
Answer: A
Arteriosclerosis is a thickening of the arterial walls, which causes a loss of elasticity (hardening of the arteries) and a subsequent reduction in blood flow. Atherosclerosis is a process in which cholesterol and other fatty substances build up and form a plaque inside the walls of blood vessels, which obstructs blood flow and interferes with their ability to dilate or contract. Prinzmetal angina, also called variant or vasospastic angina, occurs when the coronary arteries constrict and significantly decrease blood flow to the heart. Dissection is the process in which the inner layers of the arterial wall, such as the aorta, become separated and allow blood to flow (at high pressures) in between the layers.
Which of the following cardiac dysrhythmias has the GREATEST potential for deteriorating to ventricular fibrillation?
A) Asystole
B) Sinus tachycardia
C) Sinus bradycardia
D) Ventricular tachycardia
Answer: D
Ventricular tachycardia is a dysrhythmia that indicates significant ventricular irritability; therefore, it has the greatest potential for deteriorating to ventricular fibrillation (V-Fib). Asystole, the absence of all electrical and mechanical activity in the heart, is often the result of uncorrected V-Fib.
Survival from out-of-hospital cardiac arrest is MOST dependent on:
A) cardiac drug administration.
B) advanced airway management.
C) aggressive paramedic care.
D) early CPR and defibrillation.
Answer: D
Early CPR and defibrillation (if indicated) have consistently shown to be the two most critical interventions for a cardiac arrest patient with regard to survival. High-quality CPR helps to keep the vital organs viable, and early defibrillation can terminate ventricular fibrillation (V-Fib), which is present as the initial dysrhythmia in about 75% of adult out-of-hospital cardiac arrests. Delayed or inadequate CPR has been directly linked to poor outcomes. For each minute that V-Fib persists, the patient’s chance of survival decreases by 7% to 10%. Early advanced cardiac life support (advanced airway management, paramedic care, cardiac drug administration) is an important component in the treatment of the cardiac arrest patient; however, early CPR and defibrillation are the two most vital components in the chain of survival.
Which of the following cardiac valves are referred to as semilunar valves?
A) Aortic and mitral
B) Aortic and pulmonic
C) Tricuspid and mitral
D) Tricuspid and pulmonic
Answer: B
The pulmonic (right side of the heart) and aortic (left side of the heart) valves are referred to as semilunar valves. The tricuspid (right side of the heart) and mitral (left side of the heart) valves are referred to as atrioventricular valves.
You are transporting a 57-year-old man with chest pain. He is receiving supplemental oxygen and has an intravenous line in place. Suddenly, the patient becomes unresponsive. You should:
A) immediately insert a supraglottic airway device.
B) quickly assess for breathing and check for a pulse.
C) open his airway and look, listen, and feel for breathing.
D) insert an oral airway and begin positive-pressure ventilation.
Answer: B
If you find a patient who is unresponsive, or if a patient becomes unresponsive in your presence, you should quickly assess for breathing and then check for a pulse. Assess for breathing by quickly visualizing the chest for obvious movement. If the patient is not breathing or is not breathing normally (agonal gasps), check for a pulse. If the patient has a pulse, open the airway and provide rescue breathing. If the patient does not have a pulse, begin CPR starting with chest compressions. The technique of look, listen, and feel is no longer recommended; it delays chest compressions if they are needed. The patient may require advanced airway management, but not before performing CPR (if indicated) and establishing a patent airway with basic means first.
You are called to a local supermarket where a customer collapsed. The man is approximately 50-years-old and appears to weigh about 180 lb. When you arrive, two bystanders are performing CPR. Your first action should be to:
A) verify the effectiveness of the bystanders’ CPR.
B) apply the AED and analyze the patient’s cardiac rhythm.
C) insert a supraglottic airway without interrupting CPR.
D) assess the patient to confirm that he is in cardiac arrest.
Answer: D
On arriving at a scene where bystander CPR is in progress, your first action should be to confirm that the patient is in cardiac arrest. Quickly confirm that he is unresponsive and not breathing and check for a pulse for no longer than 10 seconds. If cardiac arrest is confirmed, resume CPR and apply the automated external defibrillator (AED) as soon as possible. Advanced airway management is not an immediate priority during the initial treatment of cardiac arrest.
Which of the following clinical presentations is consistent with left-sided heart failure?
A) History of a heart murmur, jugular venous distention, and tachycardia
B) History of diabetes, edema to the ankles, and abdominal distention
C) History of hypertension, dried blood around the mouth, and agitation
D) History of angina, swollen hands and feet, and excessive urination
Answer: C
Congestive heart failure occurs when the lungs or systemic circulation become congested with blood secondary to failure of the heart to effectively pump blood. Common causes of congestive heart failure include acute myocardial infarction, heart valve disease, and chronic hypertension. In left-sided heart failure, the left ventricle is unable to pump blood effectively enough so blood backs up in the lungs. Signs and symptoms include dyspnea; orthopnea (difficulty breathing while lying down); a productive cough with pink frothy sputum (dried blood around the mouth is evidence of this); and restlessness or agitation secondary to reduced cerebral blood flow. Right-sided heart failure occurs when the right ventricle is unable to effectively pump blood to the lungs and results in blood backing up in the systemic circulation. Signs and symptoms include peripheral edema and jugular venous distention. In more severe cases, fluid can accumulate in the peritoneal cavity (ascites), resulting in a painful, distended abdomen.
A 61-year-old woman presents with acute chest pain and shortness of breath that woke her from her sleep. She took two of her prescribed nitroglycerin tablets before your arrival, but is still in significant pain. Her BP is 86/52 mm Hg, her heart rate is 110 beats/min, and her respirations are 24 breaths/min and labored. In addition to administering supplemental oxygen, you should:
A) give her up to 325 mg of aspirin, monitor her vital signs, and transport at once.
B) start an IV line, give her a 2 liter normal saline bolus, and transport immediately.
C) place her in a supine position, elevate her legs, and give her 325 mg of aspirin.
D) give her one more NTG tablet, reassess her blood pressure, and transport at once.
Answer: A
The patient’s clinical presentation is consistent with acute myocardial infarction, which is possibly complicated by left-sided congestive heart failure. Her systolic BP is less than 90 mm Hg, which contraindicates further NTG administration. You should give her up to 325 mg of aspirin, monitor her vital signs, and transport at once. Establish vascular access en route and administer enough fluid to maintain adequate perfusion; 2 L of normal saline is a lot of fluid and could easily exacerbate any pulmonary edema she may have. The patient may be a candidate for continuous positive airway pressure (CPAP); however, you must recall that any form of positive-pressure ventilation has a negative effect on cardiac output, and CPAP could cause a further drop in her BP. A supine position should be avoided in patients with labored breathing and possible pulmonary edema because this only makes it more difficult for them to breathe.
You and your partner witness the collapse of a middle-aged man. Which of the following represents the MOST correct sequence for assessing and providing care to him?
A) Immediately begin CPR, starting with chest compressions, while your partner quickly retrieves the AED from the ambulance
B) Assess responsiveness and visualize the chest for signs of breathing, check for a pulse, and begin chest compressions if needed
C) Gently tap the patient to see if he responds; open his airway; look, listen, and feel for breathing; and give two rescue breaths if needed
D) Open the airway while assessing responsiveness, give two rescue breaths if needed, and check for a carotid pulse for up to 10 seconds
Answer: B
If you encounter a patient who seems to be unresponsive or if you witness the collapse of a patient, you should assess responsiveness while quickly visualizing the chest for obvious signs of breathing. If the patient is not breathing or is not breathing normally (agonal gasps), assess for a carotid pulse for up to 10 seconds and begin chest compressions if the patient is pulseless. As you begin CPR (30 chest compressions to two rescue breaths), your partner should retrieve the AED and apply it as soon as possible. The technique of look, listen, and feel is no longer recommended because it causes an unnecessary delay in providing chest compressions if the patient is pulseless.
A 70-year-old woman was suddenly awakened with the feeling that she was suffocating. She is anxious, is laboring to breathe, and has dried blood on her lips. Which of the following pathophysiologies BEST explains her clinical presentation?
A) Decreased stroke volume with left heart failure
B) Increased preload with left heart failure
C) Decreased preload with right heart failure
D) Increased stroke volume with right heart failure
Answer: A
Paroxysmal nocturnal dyspnea (PND), the sudden awakening from sleep with the feeling of being suffocated, along with the dried blood around the patient’s lips (likely due to coughing up blood-tinged sputum), are classic indicators of left-sided congestive heart failure (CHF). In left-sided CHF, stroke volume (the amount of blood ejected from the ventricle per contraction) is decreased secondary to a weakened or damaged myocardium. Decreased stroke volume causes blood to regurgitate into the upper chamber of the heart and ultimately backs up into the lungs and causes pulmonary edema.
The purpose of defibrillation is to:
A) stop all electrical activity in the heart.
B) convert asystole to a perfusing rhythm
C) decrease an excessively rapid heart rate.
D) increase the rate of a slow beating heart.
Answer: A
To defibrillate means to shock the heart with a specialized electrical current (direct current) to stop all electrical activity and restore a normal, rhythmic beat. Defibrillation induces a period of asystole, an absence of all electrical activity, so that a pacemaker in the heart may resume normal cardiac electrical activity. Defibrillation is used to terminate ventricular fibrillation and pulseless ventricular tachycardia; defibrillation is not used to treat asystole. Excessively rapid heart rates are not treated with defibrillation (unsynchronized shock); instead, they are treated with cardioversion (synchronized shock) if the patient is unstable or medication therapy if the patient is stable. Excessively slow heart rates are treated with transcutaneous cardiac pacing, a procedure in which the heart rate is increased by passing small electrical currents through the heart at a preset rate, or with various medications.
A 50-year-old man experiences chest discomfort after exerting himself. After resting for 10 minutes and taking one of his prescribed nitroglycerin tablets, the pressure in his chest completely subsides. This scenario MOST accurately describes:
A) unstable angin
B) cardiac necrosis.
C) stable angina.
D) myocardial infarction.
Answer: C
When, for a brief period, the heart muscle is not getting enough oxygen (ischemia), the resultant chest pain, pressure, or discomfort is called angina pectoris. Angina that follows a predictable pattern and is promptly relieved with rest and/or NTG is called stable angina. By contrast, unstable angina does not follow a predictable pattern and is often not relieved by rest and/or NTG. Unstable angina is also referred to as preinfarction angina. Acute myocardial infarction (AMI) occurs when a portion of the heart muscle dies (undergoes necrosis) because of a completely blocked coronary artery. Although the pain characteristics of angina and AMI are the same, angina is often relieved by rest and/or NTG, whereas the pain from an AMI is not.
What is the ejection fraction if the ventricle fills with 120 mL of blood and has a stroke volume of 80 mL?
A) 0.59
B) 0.67
C) 0.74
D) 0.8
Answer: B
Ejection fraction is the percentage of blood ejected from a filled ventricle during a single contraction. If the ventricle contains 120 mL before a contraction, and then ejects 80 mL during the contraction (the stroke volume), the ejection fraction is 67% (80 mL / 120 mL = 66.6 [67%]). Normal left ventricular ejection fraction is 55% to 70%.
The ability of cardiac cells to respond to electrical impulses is called:
A) excitability.
B) automaticity.
C) conductivity.
D) contractility.
Answer: A
Excitability refers to the cardiac cells’ ability to respond to electrical impulses. Cardiac muscle cells have a special characteristic called automaticity, which is not found in any other type of muscle in the body. Automaticity allows a cardiac muscle cell to contract spontaneously without a stimulus from a nerve cell. The ability of the cardiac cells to conduct electrical impulses is called conductivity. Contractility refers to the force with which the heart muscle contracts.
Which of the following is NOT a contraindication for administering nitroglycerin?
A) Systolic BP less than 90 mm Hg
B) History of a prior ischemic stroke
C) Recent use of Cialis or Levitra
D) Heart rate less than 50 beats/min
Answer: B
Contraindications to the administration of nitroglycerin (NTG) include a systolic blood pressure less than 90 mm Hg, significant bradycardia (heart rate
A 66-year-old woman is diagnosed with cardiomyopathy. What does this indicate?
A) An enlarged myocardium
B) Strengthening of the ventricles
C) Progressive cardiac weakening
D) An occluded coronary artery
Answer: C
Cardiomyopathy is a progressive weakening of the myocardium. This condition is commonly the result of chronic hypertension, multiple myocardial infarctions, or congestive heart failure. An enlarged myocardium is called cardiomegaly.
Sudden cardiac arrest in the adult population is MOST often secondary to:
A) a cardiac arrhythmia.
B) myocardial infarction.
C) massive hypovolemia.
D) respiratory failure.
Answer: A
Approximately 75% of adult sudden cardiac arrests are secondary to a cardiac arrhythmia, most frequently ventricular fibrillation (V-Fib). Respiratory failure is the most common cause of cardiac arrest in infants and children.
Shortly after administering nitroglycerin to your 51-year-old patient with chest pain, he becomes lightheaded and pale. You reassess his BP and note that it is 84/56 mm Hg. A patent IV line has been established and the patient is receiving supplemental oxygen. You should immediately:
A) place him supine and elevate his legs.
B) give a 20 mL/kg bolus of normal saline.
C) give him 324 mg of aspirin and reassess him.
D) assist his ventilations with a bag-mask device.
Answer: A
Nitroglycerin is a vasodilator, and in some cases, it can cause hypotension. Hypotension in a patient who may be experiencing an acute myocardial infarction could be disastrous. If hypotension occurs after administrating NTG, you should immediately place the patient supine and elevate his or her legs 6 to 12 inches to improve perfusion. An IV fluid bolus may also be indicated, but elevating the patient’s legs is faster than infusing 500 mL of normal saline. Aspirin is not a vasodilator and has no effect on the blood pressure. Furthermore, aspirin would have been given prior to NTG anyway. There is no evidence in this scenario that indicates the patient is breathing inadequately; therefore, assisted ventilation is not necessary at this time.
Which of the following statements regarding the chest pain or discomfort associated with acute myocardial infarction is correct?
A) It is severe, but generally subsides within 30 minutes
B) It does not change with breathing or body movement
C) It is typically described by the patient as a sharp feeling
D) It is often pleuritic in nature and is made worse by breathing
Answer: B
The chest pain or discomfort associated with acute myocardial infarction (AMI) is often described by the patient as pressure, crushing, or squeezing, and is not affected by breathing or other body movement. Pleuritic (sharp) chest pain that is made worse by breathing is common with a spontaneous pneumothorax or pulmonary embolism; it is not common in patients experiencing an AMI. Unlike anginal chest pain, which often subsides within 15 minutes after rest or nitroglycerin, the pain associated with an AMI can last from 30 minutes to several hours.
Common signs of acute left heart failure with pulmonary edema include:
A) jugular vein distention.
B) ascites and bradycardia.
C) hypertension and tachycardia.
D) peripheral edema and hypotension.
Answer: C
Hypertension and tachycardia are commonly associated with acute left heart failure and pulmonary edema. These signs are the result of a sympathetic discharge of epinephrine to increase cardiac contractility and improve cardiac output. Jugular vein distention, peripheral edema, and ascites (fluid accumulation in the peritoneum) are common findings associated with chronic right-sided heart failure.