Cardiology Flashcards

1
Q

Sinus Bradycardia: Description/Common Causes/Presentation/Dx/Treatment

A

Description:Regular, sinus, slower than normal heart rate for age
Common Causes:Sleep, athletic habitus, anorexia, breath-holding spells, hypothyroidism, medications, steroids, elevated intracranial pressure, severe hypoxia, sinus node dysfunction
Presentation:Can be asymptomatic in athletic individuals
If significant can have fatigue, exercise intolerance, and syncope
dx: ascultation and ECS
Treatment: May not require treatment
Reversible causes should be treated individually
Bradycardic arrest: CPR and atropine
Sinus node dysfunction can be treated with pacemaker

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2
Q

Sinus Arrhythmia: Description/Common Causes/Presentation/Diagnosis/Treatment

A

Description: Irregular, sinus, varying distance between QRS complexes
(Fig. 8.2)
Common Causes: Respirophasic variation: heart rate increases with inspiration, decreases with expiration

Presentation
▪ Usually asymptomatic
Diagnosis
Auscultation and ECG
Treatment
▪ No treatment required

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3
Q

Sinus Arrhythmia: Second-Degre heart Block (Wenckebach/Mobitz I): Description/Common Causes/Presentation/Diagnosis/Treatment

A

Description Gradual PR interval prolongation followed by dropped QRS complex

Common Causes: Occur during times of high vagal tone, often during sleep, athletic habitus, AV node dysfunction
Presentation
▪ Usually asymptomatic
Diagnosis
ECG
Treatment
▪ Usually transient and does not require treatment

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4
Q

Second-Degree Heart Block (Mobitz II)

A

Description Constant PR interval with abrupt dropped QRS complex

Common causes: Most common cause of myocarditis, postsurgical complications, AV node dysfunction, Lyme disease
Presentation
▪ Can be asymptomatic
▪ Can also have fatigue
Diagnosis
ECG
Treatment
▪ Always pathologic

▪ Treat reversible cause if present

▪ Pacemaker placement if no treatable cause

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5
Q

Third-Degree Heart Block or Complete Heart Block

A

Description: AV dissociation with constant P wave faster than QRS rate
(Fig. 8.3)
Common Causes
Congenital (maternal autoimmune disorders), structural heart disease, postsurgical, Lyme disease
Presentation
▪ Can be asymptomatic

▪ Fatigue

▪ Syncope
Diagnosis
ECG, Holter, exercise test, echocardiogram
Treatment
▪ If reversible, can treat

▪ If symptomatic or hemodynamic compromise, will need pacemaker

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6
Q

Sinus Tachycardia

A

Description: Regular, sinus, faster than normal heart rate for age

Common Causes: Anemia, dehydration, pain/agitation, hyperthyroidism
Presentation:
▪ Usually symptomatic from other causes

▪ May have “palpitations”
Diagnosis:
Auscultation and ECG
Treatment:
▪ Treat reversible causes, rare in isolation

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7
Q

Supraventricular Tachycardia (SVT) and Wolff-Parkinson-White (WPW)

A

Description:
Baseline: sinus, WPW has delta wave causing slurring of P wave into QRS
(Fig. 8.4)
In SVT: narrow QRS complex, much faster than normal heart rate, P waves may not be visible
(Fig. 8.5)
Common Causes:
Accessory conduction pathway
Presentation:
▪ At baseline, may not have any symptoms

▪ Will feel palpitations during tachycardia

▪ Can have syncope and sudden cardiac death (high-risk WPW)
Diagnosis
Baseline ECG,
Holter, Exercise test to determine if high risk for sudden death,
SVT ECG
Treatment
▪ Acute treatment of SVT: breath-holding, adenosine, direct cardioversion

▪ Long-term treatment: beta- blockers, catheter ablation

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8
Q

Atrial Flutter

A

Description: Regular, “sawtooth pattern” with atrial rates faster than QRS waves
Common CausesUncommon in children, can be seen in neonates with no other associations
Presentation:
▪ Can be asymptomatic

▪ If rhythm is very fast, can show hemodynamic compromise
Diagnosis
ECG, Holter, echocardiogram to rule out structural heart disease
Treatment
▪ Direct synchronized cardioversion

▪ Usually neonates do not require further therapy

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9
Q

Atrial Fibrillation

A

Description: Irregularly irregular, P waves are abnormal and different morphologies, QRS similar to sinus rhythm
Common Causes: Very uncommon in children, can occur in patients with WPW who have SVT that degenerates into atrial fibrillation, postsurgical
Presentation
▪ Palpitations

▪ Can be at risk of stroke due to left atrial thrombus formation
Diagnosis
ECG, Holter, echocardiogram to rule out structural heart disease
Treatment
▪ Direct cardioversion (must be anticoagulated first due to risk of stroke)

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10
Q

Ventricular Tachycardia

A

Description: Regular, wide complex, QRS different from baseline
(Fig. 8.6)
Common Causes: Structural heart disease, postsurgical, myocarditis, long QT, medication overdose, cardiomyopathy
Presentation
▪ May be asymptomatic

▪ Palpitations

▪ Fatigue

▪ Syncope

▪ Sudden death
Diagnosis
ECG, Holter, exercise test, echocardiogram to rule out structural heart disease
Treatment
▪ If asymptomatic, can consider medical management with beta-blockers

▪ If syncope or aborted sudden cardiac arrest, implantable cardiac defibrillator (ICD) placement

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11
Q

Torsades de Pointes

A

Description Irregular, fast, wide complex, sinusoidal pattern along baseline
Common Causes
Hypokalemia, hypomagnesemia
Prolonged QTc, Brugada
Presentation
▪ Palpitations

▪ Syncope

▪ Sudden death
Diagnosis
ECG
Treatment
▪ Correct if reversible cause

▪ If progresses to ventricular fibrillation, defibrillate!

▪ May respond to magnesium

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12
Q

Ventricular Fibrillation

A

Description: Irregular, wide complex, can be disorganized
Common Causes: Electrolyte abnormalities, channelopathies, postsurgical
Presentation
▪ Palpitations

▪ Syncope

▪ Sudden death
Diagnosis
ECG
Treatment
▪ Correct if reversible cause

▪ Defibrillate!

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13
Q

Premature Atrial Complex (PAC)

A

Description: Single, early, narrow complex beat
(Fig. 8.7)
Common cause:
Abnormal focus in atria, electrolyte abnormalities, can be normal finding
Presentation
▪ Likely asymptomatic if isolated
Diagnosis
ECG, Holter, electrolytes, echocardiogram if frequent PACs
Treatment
▪ No treatment necessary in isolation

▪ Can treat reversible causes

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14
Q

Premature Ventricular Complex

A

Description: Single, early, wide complex beat, different QRS than baseline
(Fig. 8.8)
Common Cause:
Abnormal focus in ventricle, electrolyte abnormalities, Long QT syndrome, myocarditis, HCM, structural heart disease, can be normal finding
Presentation
▪ Likely asymptomatic if isolated
Diagnosis
ECG, Holter, electrolytes, echocardiogram if frequent PVCs
Treatment
▪ No treatment necessary in isolation

▪ Can treat reversible causes

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15
Q

First Degree Heart Block

A

Description: PR interval prolonged beyond normal for age
Common Causes:
AV node dysfunction, increased vagal tone (e.g., sleep), rheumatic fever, Ebstein’s anomaly, Atrial septal defect (ASDs), AV canal defects, hypocalcemia medications
Presentation:
▪ Likely asymptomatic
Diagnosis
ECG, Holter, echocardiogram if persistent
Treatment
▪ Usually benign and no treatment required

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16
Q

Prolonged QT

A

Description: QT interval is corrected (QTc) for heart rate and is prolonged beyond normal for age
(Fig. 8.9)
Common Causes:
Congenital: familial or de novo genetic mutation
Acquired: electrolyte abnormalities, medications, postsurgical, myocarditis, infections
Presentation
▪ At baseline, likely asymptomatic

▪ Predisposes to ventricular fibrillation and can cause syncope or sudden death
Diagnosis
ECG, Holter, exercise test, echocardiogram to rule out structural heart disease, genetic testing
Treatment
▪ Avoid QTc- prolonging medications

▪ Beta-blocker for genetic cause

▪ ICD if history of syncope or aborted sudden cardiac arrest

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17
Q

Infective Endocarditis: Most Common?

A

alpha-hemolytic (viridans group) streptococci

18
Q

Duke Criteria for Infective Endocarditis: Major

A

Positive blood culture for infective endocarditis
Typical microorganisms consistent with infective endocarditis from two separate blood cultures
OR
Persistently positive blood culture
OR
Single positive blood culture for Coxiella burnetii or phase I IgG antibody titer >1:800
Evidence of endocardial involvement:
Positive echo
Vegetation
OR
Abscess
OR
New partial dehiscence of a prosthetic valve
New valvular regurgitation on echo

18
Q

Infective Endocarditis: second most common

A

Staphylococcus aureus

18
Q

Duke Criteria for Infectious Endocarditis: Minor criteria

A

Predisposing factor: IV drug use or presence of a predisposing cardiac condition
Fever ≥38.0°C
Vascular phenomena
Immunologic phenomena
Microbiologic evidence: positive blood cultures that do not meet major criteria, or active infection with an organism consistent with infective endocarditis
Definite Infective Endocarditis
Pathologic Criteria
Pathologic lesions: vegetation or intracardiac abscess demonstrating active endocarditis on histology
OR
Microorganism on culture, or histology of a vegetation or intracardiac abscess
Clinical Criteria
Two major clinical criteria
OR
One major and three minor clinical criteria
OR
Five minor clinical criteria
Possible Infective Endocarditis
Presence of one major and one minor clinical criteria
OR
Presence of three minor clinical criteria
Rejected Infective Endocarditis
A firm alternate diagnosis is made
OR
Resolution of clinical manifestations occurs after ≤4 days of antibiotic therapy
OR
No pathologic evidence of infective endocarditis found at surgery or autopsy after ≤4 days of antibiotic therapy
OR
Clinical criteria for possible or definite infective endocarditis not met

18
Q

Pulmonary Flow Murmur

A

Age: All ages
Timing and Configuration: Early to mid-systolic, crescendo-decrescendo
Intensity: Grades 2–3
Quality:
Rough, dissonant
↑ when supine
Location: Second and third intercostal spaces
Etiology: Audible flow across pulmonary outflow tract

19
Q

Still’s Murmur

A

Age: 2–6 years, may be audible from infancy to adulthood
Timing and Configuration: Early systole
Intensity: Grades 1–3
Quality:
Vibratory “twang” or “musical”
↑ when supine
Location:
LLSB, extends to apex
Etiology
Ventricular false tendons

19
Q

Peripheral pulmonic stenosis

A

Age: 0–6 months
Timing and Configuration: Ejection murmur beginning in mid-systole
Intensity: Grades 1–2
Quality: NA
Location: LUSB, radiates to bilateral axillae and back
Etiology:
Acute takeoff of the branch PAs in neonates
↑ with respiratory infections

20
Q

Venous hum

A

Age: ∼3–8 years
Timing and Configuration: Continuous murmur,
↑ in diastole
Intensity: Grades 1–3
Quality:
Whining, roaring, or whirring
↑ when supine ↑ with head turned away from examiner
↓ with compression of jugular vein
Location:
Low anterior neck, extends to infraclavicular area, R>L Etiology: Turbulence at confluence of jugular and subclavian veins as they enter SVC, or angulation of IJV as it courses over transverse process of atlas

21
Q

Supraclavicular or brachiocephalic systolic murmur

A

Age: Children and teenagers
Timing and Configuration: Brief, crescendo-decrescendo Intensity: Grades 1–3
Quality: Disappears with hyperextension of shoulders Location: Above clavicles, radiates to neck
Etiology: Major brachiocephalic vessels arising from aorta

22
Q

Aortic systolic murmur

A

Age: Older children and adults
Timing and configuration: Ejection
Intensity: Grades 1–3
Quality NA
Location: RUSB
Etiology: ↑ with anxiety, anemia, hyperthyroidism, or fear

23
Q

Mammary artery soufflé

A

Age:Teenagers and pregnant women
Timing and configurationSystolic murmur, extends into diastole Intensity: Grades 1–3
Quality: Varies from day to day
Location: Anterior chest wall over breast
Etiology: Blood flow in arteries and veins leading to and from breasts

24
Q

Coarctation: Radiographic Finding

A

“3” sign

25
Q

Coarctation: Anatomic Correlate for radiographic finding

A

Due to “infolding” at the junction of the ascending aorta and descending aorta

26
Q

Tetralogy of Fallot: radiographic finding

A

“boot-shaped” heart

27
Q

Tetralogy of fallot: anatomic correlate for radiographic finding

A

Due to right ventricular hypertrophy causing elevation of the apex

28
Q

Transposition of the great arteries: radiographic finding

A

“egg-on-a-string” heart

29
Q

Transposition of the great arteries: anatomic correlate for radiographic finding

A

Narrowed mediastinal silhouette because of the parallel position of the great vessels, and the heart is more globular, possibly due to increased pulmonary venous return to the left atrium

30
Q

Truncus radiographic finding

A

narrowed medastinal silhouette

31
Q

truncus anatomic correlate for radiographic finding

A

Single arterial trunk

32
Q

Total anomalous pulmonary venous return (TAPVR): radiographic finding

A

“Snowman in a snowstorm”

33
Q

Total anomalous pulmonary venous return (TAPVR): anatomic correlate for radiographic finding

A

Occurs in supracardiac TAPVR when the vertical vein, superior vena cava, and innominate vein create the “head” and a dilated right atrium creates the “body” of the snowman. In obstructed TAVPR, pulmonary venous blood flow cannot exit the lungs, causing diffuse pulmonary edema, or the “snowstorm”

34
Q

Severe Ebstein anomaly radiographic finding

A

“wall-to-wall” heart

35
Q

Severe Ebstein anomaly: anatomic correlate for radiographic finding

A

Severe cardiomegaly due to an extremely enlarged right atrium

36
Q
A