Cardio Images Flashcards

1
Q

Name the pathologic process and how it results/examples.

A

Concentric hypertrophy

Increased systolic loads (increased afterloads)

Examples: aortic stenosis, pulmonic stenosis, pulmonary hypertension in PDA, cats with hyperthyroidism (systemic hypertension)

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2
Q

Name the pathologic process and how it results/examples.

A

Eccentric hypertrophy & dilation

Increase in diastolic load (increased preload)

Examples: AV or semilunar insufficiencies, arteriovenous shunts

Mass is usually increased, but walls are thin

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3
Q

Name the pathologic process and what does this indicate.

What can this be seen with?

A

Subendocardial fibrosis

Best indicator or dilation in atria

Seen with either type of cardiac hypertrophy

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4
Q

This image shows three sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: pulmonary congestion & edema, LA enlargement

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5
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: pulmonary edema

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6
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: hemosiderosis “heart failure cells”

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7
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

RHF

Sequelae: passive hepatic congestion “nutmeg liver”

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8
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

RHF

Sequelae: hydrothorax

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9
Q

This image shows what pathologic process?

Define the pathologic process.

A

RHF secondary to pulmonary disease (dirofilariasis) = “Cor pulmonale”

(other causes - chronic obstructive pulmonary dz, PTE, neoplasia)

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10
Q

Name the congenital abnormality indicated by the arrow.

What are the consequences?

Name the congenital abnormality indicated by the star.

A

Atrial septal defect

Consequences: excessive flow from LA to RA in neonate → RV dilation → elevated CVP & blood in lungs → pulmonary hypertension → RV hypertrophy → reversal of flow through defect → cyanosis

High ventricular septal defect

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11
Q

Name the congenital abnormality, where is it located?

What are the consequences?

A

Ventricular septal defect in membranous portion

Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death

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12
Q

Name the congenital abnormality, where is it located?

What are the consequences?

A

Ventricular septal defect in muscular portion

Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death

(none in neonate)

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13
Q

Name the congenital abnormality.

How long after birth is this considered normal?

A

PDA

Normally closes within 5 days after birth

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14
Q

Name the congenital abnormality.

Which arrow indicates the abnormality (top, middle or bottom)?

A

PDA

Middle

Top = aorta

Bottom = pulmonary artery

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15
Q

Name the pathologic process.

What diseases does this commonly accompany?

A

Subendocardial hemorrhage

Associated diseases: septicemia, endotoxemia, anoxia, electrocution, trauma, agonal change

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16
Q

Name the pathologic process.

What are 6 causes of this process?

A

Subendocardial mineralization

Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease

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17
Q

Name the pathologic process.

What are 6 causes of this process?

A

Subendocardial mineralization

Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease

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18
Q

Name this pathologic process.

What are 3 causes, in order of decreasing incidence?

What are some gross findings that are characteristic of this process?

Which anatomic location is most commonly affected?

A

Vegetative valvular and mural endocarditis

Causes: bacteria, parasites, fungi

Characteristics: large yellow/gray friable masses of fibrin on valves “vegetations” (fibrin, leukocytes, bacteria, granulation tissue microscopically)

Mitral > aortic > tricuspid > pulmonary

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19
Q

Name this pathologic process.

Which portion of the heart is affected with this process?

A

Uremic endocarditis

Affected: left atrium

Ulcerative

20
Q

Name this pathologic process.

What is it characterized by and which structure is most commonly affected?

A

Endocardiosis

Myxomatous valvular degeneration

Mitral > tricuspid

21
Q

Name this pathologic process.

What is it characterized by and which structure is most commonly affected?

A

Endocardiosis

Myxomatous valvular degeneration

Mitral > tricuspid

22
Q

This image shows a possible sequalae of endocardiosis, name the sequalae.

What are the other 6 sequelae of endocardiosis?

A

Left atrial rupture (also shown here)

Others: valvular incompetency, congestive HF (R or L), atrial dilation, jet lesions, hemopericardium, chordae tendinae rupture

23
Q

Name this pathologic process.

What species is this usually seen in?

Why are these not of concern?

A

Hematocyst

Ruminants

Do not cause problems, regress within a few months after birth

24
Q

Name this pathologic process.

What species is this usually seen in?

Why are these not of concern?

A

Lymphocyst

Ruminants

Do not cause problems, regress within a few months after birth

25
Q

Name the pathologic process and specific cause.

What species are affected?

What do the arrows indicate?

A

Myocardial necrosis

Cause: nutritional - vitamin E/selenium deficiency

Species: lambs, calves, swine, horses

LEFT: areas of necrosis

RIGHT: mineralization of necrotic fibers

26
Q

Name the pathologic process and specific cause.

What is this disease process called?

A

Myocardial necrosis

Cause: nutritional - vitamin E/selenium deficiency

“Mulberry heart disease”

27
Q

Name the pathologic process and specific cause.

What species is this commonly in?

A

Myocarditis

Cause: Clostridium chauvoei

Cow

28
Q

Name the pathologic process and specific cause.

What species is this commonly in?

A

Myocarditis

Cause: Blastomyces dermatitidis

Dog

29
Q

Name the pathologic process and specific cause.

What species is this commonly in?

A

Myocarditis

Cause: canine parvovirus-2

Dog

30
Q

Name the pathologic process and specific cause.

What species is this commonly in?

A

Myocarditis

Cause: Trypanosoma cruzi “Chagas disease” (protozoa)

Dog

31
Q

Name this pathologic process and underlying disease.

A

Concentric hypertrophy

Feline HCM

32
Q

Name this pathologic process and underlying disease.

A

Concentric hypertrophy

Feline HCM

33
Q

Name this disease process and species.

What is this disease process associated with?

A

Feline DCM

Associated: taurine deficiency

34
Q

Name this pathologic process.

What species is commonly affected and what is likely the underlying cause?

What are the consequences?

A

Excessive moderator bands (false tendons)

Feline - likely congenital, manifests in older cats

Consequences: HF and death

35
Q

Name the disease process and species/signalment.

What is the underlying cause?

What are some common gross findings and consequences?

A

DCM

Species/signalment: dogs - young to middle aged giant/large breeds

Cause: genetic, may be x-linked (some breeds)

Gross findings: LHF, biventricular failure, all chambers dilated +/- subendocardial/atrial fibrosis

Consequences: HF, unexpected death

36
Q

Name the disease process and species/signalment.

What gross finding is notable here?

A

DCM

Species/signalment: dogs - young to middle aged giant/large breeds

Gross finding: subendocardial fibrosis

37
Q

Name the disease process and species/breed.

What do the pale areas represent?

A

Wooly coat cardiomyopathy

Bovine - wooly/curly haired Herefords

Pale areas = necrosis

38
Q

Name the pathologic process.

Name some underlying causes.

A

Hydropericardium

Causes: generalized anasarca, hypoalbuminemia, congestive heart failure, neoplasia

39
Q

Name the pathologic process.

How is this differentiated from the “true form”?

What are the consequences?

A

Hemopericardium

Clotted blood = “true”

Consequences: rapidly results in death

40
Q

Name the pathologic process and underlying cause.

A

Serous atrophy of pericardial fat

Cause: cachexia of any cause

41
Q

Name the pathologic process, species and underlying causes.

A

Fibrinous pericarditis

Horse (Foal)

Causes: Mycoplasma felis, mare reproductive loss syndrome

42
Q

Name the pathologic process, species and underlying cause.

A

Fibrinous pericarditis

Swine

Causes: Haemophilus suis “Glasser’s disease”

43
Q

Name the pathologic process, species and underlying cause.

A

Chronic constrictive pericarditis (purulent pericarditis)

Bovine

Cause: result of traumatic perforation from FB in reticulum → purulent pericarditis → chronic constrictive pericarditis → severe cardiac dysfunction

44
Q

Name the disease process.

A

Aortic body tumor “chemodectoma”

This is a primary neoplasm of the heart

45
Q

Name and describe the disease process

What species can this be seen in?

What is likely the origin?

A

Rhabdomyoma

Definition: anomalous formations of perinatal cardiac myocytes

Species: pigs >>> cattle, sheep, dogs

Origin: possibly Purkinje cell in swine

46
Q

Name the disease process and species.

A

Metastatic lymphoma

Dog