Cardio Images Flashcards
Name the pathologic process and how it results/examples.
Concentric hypertrophy
Increased systolic loads (increased afterloads)
Examples: aortic stenosis, pulmonic stenosis, pulmonary hypertension in PDA, cats with hyperthyroidism (systemic hypertension)
Name the pathologic process and how it results/examples.
Eccentric hypertrophy & dilation
Increase in diastolic load (increased preload)
Examples: AV or semilunar insufficiencies, arteriovenous shunts
Mass is usually increased, but walls are thin
Name the pathologic process and what does this indicate.
What can this be seen with?
Subendocardial fibrosis
Best indicator or dilation in atria
Seen with either type of cardiac hypertrophy
This image shows three sequelae to what pathologic process?
Name the sequelae.
LHF
Sequelae: pulmonary congestion & edema, LA enlargement
This image shows a sequelae to what pathologic process?
Name the sequelae.
LHF
Sequelae: pulmonary edema
This image shows a sequelae to what pathologic process?
Name the sequelae.
LHF
Sequelae: hemosiderosis “heart failure cells”
This image shows a sequelae to what pathologic process?
Name the sequelae.
RHF
Sequelae: passive hepatic congestion “nutmeg liver”
This image shows a sequelae to what pathologic process?
Name the sequelae.
RHF
Sequelae: hydrothorax
This image shows what pathologic process?
Define the pathologic process.
RHF secondary to pulmonary disease (dirofilariasis) = “Cor pulmonale”
(other causes - chronic obstructive pulmonary dz, PTE, neoplasia)
Name the congenital abnormality indicated by the arrow.
What are the consequences?
Name the congenital abnormality indicated by the star.
Atrial septal defect
Consequences: excessive flow from LA to RA in neonate → RV dilation → elevated CVP & blood in lungs → pulmonary hypertension → RV hypertrophy → reversal of flow through defect → cyanosis
High ventricular septal defect
Name the congenital abnormality, where is it located?
What are the consequences?
Ventricular septal defect in membranous portion
Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death
Name the congenital abnormality, where is it located?
What are the consequences?
Ventricular septal defect in muscular portion
Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death
(none in neonate)
Name the congenital abnormality.
How long after birth is this considered normal?
PDA
Normally closes within 5 days after birth
Name the congenital abnormality.
Which arrow indicates the abnormality (top, middle or bottom)?
PDA
Middle
Top = aorta
Bottom = pulmonary artery
Name the pathologic process.
What diseases does this commonly accompany?
Subendocardial hemorrhage
Associated diseases: septicemia, endotoxemia, anoxia, electrocution, trauma, agonal change
Name the pathologic process.
What are 6 causes of this process?
Subendocardial mineralization
Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease
Name the pathologic process.
What are 6 causes of this process?
Subendocardial mineralization
Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease
Name this pathologic process.
What are 3 causes, in order of decreasing incidence?
What are some gross findings that are characteristic of this process?
Which anatomic location is most commonly affected?
Vegetative valvular and mural endocarditis
Causes: bacteria, parasites, fungi
Characteristics: large yellow/gray friable masses of fibrin on valves “vegetations” (fibrin, leukocytes, bacteria, granulation tissue microscopically)
Mitral > aortic > tricuspid > pulmonary