Cardio Images Flashcards

1
Q

Name the pathologic process and how it results/examples.

A

Concentric hypertrophy

Increased systolic loads (increased afterloads)

Examples: aortic stenosis, pulmonic stenosis, pulmonary hypertension in PDA, cats with hyperthyroidism (systemic hypertension)

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2
Q

Name the pathologic process and how it results/examples.

A

Eccentric hypertrophy & dilation

Increase in diastolic load (increased preload)

Examples: AV or semilunar insufficiencies, arteriovenous shunts

Mass is usually increased, but walls are thin

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3
Q

Name the pathologic process and what does this indicate.

What can this be seen with?

A

Subendocardial fibrosis

Best indicator or dilation in atria

Seen with either type of cardiac hypertrophy

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4
Q

This image shows three sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: pulmonary congestion & edema, LA enlargement

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5
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: pulmonary edema

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6
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

LHF

Sequelae: hemosiderosis “heart failure cells”

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7
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

RHF

Sequelae: passive hepatic congestion “nutmeg liver”

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8
Q

This image shows a sequelae to what pathologic process?

Name the sequelae.

A

RHF

Sequelae: hydrothorax

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9
Q

This image shows what pathologic process?

Define the pathologic process.

A

RHF secondary to pulmonary disease (dirofilariasis) = “Cor pulmonale”

(other causes - chronic obstructive pulmonary dz, PTE, neoplasia)

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10
Q

Name the congenital abnormality indicated by the arrow.

What are the consequences?

Name the congenital abnormality indicated by the star.

A

Atrial septal defect

Consequences: excessive flow from LA to RA in neonate → RV dilation → elevated CVP & blood in lungs → pulmonary hypertension → RV hypertrophy → reversal of flow through defect → cyanosis

High ventricular septal defect

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11
Q

Name the congenital abnormality, where is it located?

What are the consequences?

A

Ventricular septal defect in membranous portion

Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death

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12
Q

Name the congenital abnormality, where is it located?

What are the consequences?

A

Ventricular septal defect in muscular portion

Consequences: pulmonary vascular resistance drops after birth → L to R shunt → RV/LV pressures equalize → eccentric hypertrophy of both ventricles → pulmonary hypertension → shunt reversal R to L → cyanosis → death

(none in neonate)

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13
Q

Name the congenital abnormality.

How long after birth is this considered normal?

A

PDA

Normally closes within 5 days after birth

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14
Q

Name the congenital abnormality.

Which arrow indicates the abnormality (top, middle or bottom)?

A

PDA

Middle

Top = aorta

Bottom = pulmonary artery

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15
Q

Name the pathologic process.

What diseases does this commonly accompany?

A

Subendocardial hemorrhage

Associated diseases: septicemia, endotoxemia, anoxia, electrocution, trauma, agonal change

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16
Q

Name the pathologic process.

What are 6 causes of this process?

A

Subendocardial mineralization

Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease

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17
Q

Name the pathologic process.

What are 6 causes of this process?

A

Subendocardial mineralization

Causes: dystrophic (necrotic), metastatic (hyperCa), vit D toxicity, calcinogenic plant toxicosis in cattle, Ca:P imbalance, Johne’s disease

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18
Q

Name this pathologic process.

What are 3 causes, in order of decreasing incidence?

What are some gross findings that are characteristic of this process?

Which anatomic location is most commonly affected?

A

Vegetative valvular and mural endocarditis

Causes: bacteria, parasites, fungi

Characteristics: large yellow/gray friable masses of fibrin on valves “vegetations” (fibrin, leukocytes, bacteria, granulation tissue microscopically)

Mitral > aortic > tricuspid > pulmonary

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19
Q

Name this pathologic process.

Which portion of the heart is affected with this process?

A

Uremic endocarditis

Affected: left atrium

Ulcerative

20
Q

Name this pathologic process.

What is it characterized by and which structure is most commonly affected?

A

Endocardiosis

Myxomatous valvular degeneration

Mitral > tricuspid

21
Q

Name this pathologic process.

What is it characterized by and which structure is most commonly affected?

A

Endocardiosis

Myxomatous valvular degeneration

Mitral > tricuspid

22
Q

This image shows a possible sequalae of endocardiosis, name the sequalae.

What are the other 6 sequelae of endocardiosis?

A

Left atrial rupture (also shown here)

Others: valvular incompetency, congestive HF (R or L), atrial dilation, jet lesions, hemopericardium, chordae tendinae rupture

23
Q

Name this pathologic process.

What species is this usually seen in?

Why are these not of concern?

A

Hematocyst

Ruminants

Do not cause problems, regress within a few months after birth

24
Q

Name this pathologic process.

What species is this usually seen in?

Why are these not of concern?

A

Lymphocyst

Ruminants

Do not cause problems, regress within a few months after birth

25
Name the pathologic process and specific cause. What species are affected? What do the arrows indicate?
**Myocardial necrosis** _Cause_: nutritional - *vitamin E/selenium deficiency* _Species_: lambs, calves, swine, horses LEFT: areas of necrosis RIGHT: mineralization of necrotic fibers
26
Name the pathologic process and specific cause. What is this disease process called?
**Myocardial necrosis** _Cause_: nutritional - *vitamin E/selenium deficiency* "Mulberry heart disease"
27
Name the pathologic process and specific cause. What species is this commonly in?
**Myocarditis** _Cause_: *Clostridium chauvoei* Cow
28
Name the pathologic process and specific cause. What species is this commonly in?
**Myocarditis** _Cause_: *Blastomyces dermatitidis* Dog
29
Name the pathologic process and specific cause. What species is this commonly in?
**Myocarditis** _Cause_: *canine parvovirus-2* Dog
30
Name the pathologic process and specific cause. What species is this commonly in?
**Myocarditis** _Cause_: *Trypanosoma cruzi "*Chagas disease" (protozoa) Dog
31
Name this pathologic process and underlying disease.
**Concentric hypertrophy** *Feline HCM*
32
Name this pathologic process and underlying disease.
**Concentric hypertrophy** *Feline HCM*
33
Name this disease process and species. What is this disease process associated with?
**Feline DCM** _Associated_: taurine deficiency
34
Name this pathologic process. What species is commonly affected and what is likely the underlying cause? What are the consequences?
**Excessive moderator bands (false tendons)** Feline - likely congenital, manifests in older cats _Consequences_: HF and death
35
Name the disease process and species/signalment. What is the underlying cause? What are some common gross findings and consequences?
**DCM** _Species/signalment_: dogs - young to middle aged giant/large breeds _Cause_: genetic, may be x-linked (some breeds) _Gross findings_: LHF, biventricular failure, all chambers dilated +/- subendocardial/atrial fibrosis _Consequences_: HF, unexpected death
36
Name the disease process and species/signalment. What gross finding is notable here?
**DCM** _Species/signalment_: dogs - young to middle aged giant/large breeds _Gross finding_: subendocardial fibrosis
37
Name the disease process and species/breed. What do the pale areas represent?
**Wooly coat cardiomyopathy** Bovine - wooly/curly haired Herefords Pale areas = necrosis
38
Name the pathologic process. Name some underlying causes.
**Hydropericardium** _Causes_: generalized anasarca, hypoalbuminemia, congestive heart failure, neoplasia
39
Name the pathologic process. How is this differentiated from the "true form"? What are the consequences?
**Hemopericardium** Clotted blood = "true" _Consequences_: rapidly results in death
40
Name the pathologic process and underlying cause.
**Serous atrophy of pericardial fat** _Cause_: cachexia of any cause
41
Name the pathologic process, species and underlying causes.
**Fibrinous pericarditis** Horse (Foal) _Causes_: Mycoplasma felis, mare reproductive loss syndrome
42
Name the pathologic process, species and underlying cause.
**Fibrinous pericarditis** Swine _Causes_: Haemophilus suis "Glasser's disease"
43
Name the pathologic process, species and underlying cause.
**Chronic constrictive pericarditis (purulent pericarditis)** Bovine _Cause_: result of traumatic perforation from FB in reticulum → purulent pericarditis → chronic constrictive pericarditis → severe cardiac dysfunction
44
Name the disease process.
**Aortic body tumor "chemodectoma"** This is a primary neoplasm of the heart
45
Name and describe the disease process What species can this be seen in? What is likely the origin?
**Rhabdomyoma** _Definition_: anomalous formations of perinatal cardiac myocytes _Species_: pigs \>\>\> cattle, sheep, dogs _Origin_: possibly Purkinje cell in swine
46
Name the disease process and species.
**Metastatic lymphoma** Dog