Cardiac Arrest Flashcards

1
Q

What is cardiac arrest?

A

Sudden cardiac arrest is a sudden state of circulatory failure due to a loss of cardiac systolic function.

It is the result of 4 specific cardiac rhythm disturbances: ventricular fibrillation, pulseless ventricular tachycardia (VT), pulseless electrical activity, and asystole.

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2
Q

What are the causes of cardiac arrest?

A

The main underlying causes are ischaemic heart disease, unspecified cardiovascular disease, and cardiomyopathy/dysrhythmias.

Other causes include hypoxia, hypovolaemia, hyperkalaemia, hydrogen ion excess (acidosis), hypothermia, hypo- or hyperglycaemia, trauma, tension pneumothorax, obstructive shock (pulmonary embolism, myocardial infarction), toxins, and cardiac tamponade.

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3
Q

What are the 4 conditions included in sudden cardiac arrest?

A

Sudden cardiac arrest is the term used to describe the ultimate result of 4 different cardiac arrhythmias:

  • Ventricular tachycardia (VT)
  • Ventricular fibrillation (VF)
  • Pulseless electrical activity (PEA)
  • Asystole
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4
Q

What is ventricular tachycardia (VT)?

A

Ventricular tachycardia (VT): ≥3 consecutive complexes originating in the ventricles at a rate >100 bpm. Sustained VT lasts >30 seconds or results in haemodynamic compromise.

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5
Q

What is ventricular fibrillation (VF)?

A

Ventricular fibrillation: rapid, grossly irregular electrical activity with marked variability in waveform; ventricular rate usually >300 bpm (cycle length <200 milliseconds).

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6
Q

What are the risk factors for cardiac arrest?

A
  • Coronary artery disease
  • Left ventricular dysfunction
  • Hypertrophic cardiomyopathy (HCM)
  • Long QT syndrome
  • Medications that prolong QT interval or cause electrolyte disturbances
  • Acute medical or surgical emergency
  • Illicit substances
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7
Q

What are the signs of cardiac arrest?

A
  • Absence of normal breathing
  • Absence of circulation
  • Cardiac rhythm disturbance (including ventricular fibrillation, pulseless ventricular tachycardia, pulseless electrical activity or asystole)
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8
Q

What are the symptoms of cardiac arrest?

A
  • Absence of normal breathing
  • Absence of circulation
  • Cardiac rhythm disturbance
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9
Q

What investigations should be ordered for cardiac arrest?

A
  • Continous cardiac monitoring
  • FBC
  • Serum electrolytes
  • ABG
  • Cardiac biomarkers
  • Echocardiogram
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10
Q

Why is continuous cardiac monitoring important?

A

Identifying the cardiac rhythm is a key step in determining which cardiac arrest treatment algorithm to use.

Identifies shockable rhythm (ventricular fibrillation/pulseless ventricular tachycardia) or non-shockable rhythm (asystole/pulseless electrical activity).

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11
Q

Why investigate using FBC?

A

Haemorrhage may cause hypovolaemia and should be evaluated for.

Low haematocrit can be seen in haemorrhage.

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12
Q

Why investigate using serum electrolytes? And what may this show?

A

Electrolyte abnormalities occur as a result of sudden cardiac arrest and contribute to cardiac arrest.

May show electrolyte abnormalities, particularly hyperkalaemia or hypokalaemia.

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13
Q

Why investigate using ABG?

A

Respiratory and metabolic parameters should be optimised as necessary to normalise acid-base status. Abnormal results may be a result of sudden cardiac arrest and not necessarily the cause. May indicate an underlying respiratory aetiology of pulseless electrical activity/asystole.

May show respiratory acidosis, metabolic acidosis, respiratory acidosis with renal compensation, metabolic acidosis with respiratory compensation, mixed metabolic and respiratory acidosis or can also reveal hyperkalaemia.

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14
Q

Why investigate using cardiac biomarkers?

A

Elevations in markers of myocardial infarction (MI) may be a result of sudden cardiac arrest and do not necessarily indicate that an MI is the cause.

Positive or elevated.

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15
Q

Why investigate using echocardiogram?

A

Cardiac activity may be assessed acutely during resuscitation.

Assesses cardiac activity and left ventricular function; may show valvular abnormalities, myocardial scarring, cardiomyopathy, pericardial effusion.

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16
Q

From the four possible cardiac arrest rhythms, which are shockable?

A

Shockable rhythms:

  • Ventricular tachycardia
  • Ventricular fibrillation
17
Q

From the four possible cardiac arrest rhythms, which are non-shockable?

A

Non-shockable rhythms:

  • Pulseless electrical activity (all electrical activity except VF/VT, including sinus rhythm without a pulse)
  • Asystole (no significant electrical activity)
18
Q

Briefly describe the treatment for shockable cardiac arrest (ventricular tachycardia or ventribular fibrillation)

A

A single person performs uninterrupted chest compressions while everyone else prepares for defibrillation: stand clear, move oxygen delivery device 1 m away.

Select the appropriate energy on the defibrillator. When defibrillator is charged and safety check is complete, the rescuer performing chest compression stands clear and the shock is given.

CPR is resumed immediately. Reasess pulse and rhythm only after 2 minutes of CPR.

Repeat if shockable rhythm remains. Give drugs after 3 shocks.

Drugs: adrenaline, amiodarone and magnesium.

19
Q

Why is adrenaline used in cardiac arrest?

A

The use of adrenaline during cardiac arrest has been shown to increase the rate of achieving return of spontaneous circulation (ROSC) and to increase short-term survival.

20
Q

Why is CPR used in cardiac arrest?

A

Circulation of the blood by means of CPR is a demonstrated therapy in patients with sudden cardiac arrest and is thought to work by raising intrathoracic pressure, as well as providing direct cardiac compression.

21
Q

Briefly describe the process of CPR

A

Place the heel of 1 hand on the centre of the chest (lower half of the sternum). Place your second hand on top and interlock the fingers. Use straight arms. Give compressions at a rate of 100-120/min. Aim to compress the sternum 5-6 cm. Afer 30 seconds give 2 rescue breaths. Do not interrupt the compressions for >10s. Continue with a ratio of 30:2 until defibrillation available.

22
Q

Why is defibrillation used in cardiac arrest?

A

Demonstrated to restore normal sinus rhythm and should be initiated promptly, as the success of this therapy diminishes over time.

23
Q

Briefly describe the treatment for non-shockable rhythms (pulseless electrical activity and asystole)

A

Continue CPR. Obtain IV access and secure airway. Once airway is secure switch to continuous compressions and ventilation. Give adrenaline 1mg IV.

Check rhythm every 2 minutes.

Consider reversible causes.

24
Q

What are the complications or cardiac arrest?

A
  • Death
  • Rib and sternal fractures
  • Anoxic brain injury
  • Ischemic liver injury
  • Renal acute tubular necrosis
25
Q

Why is amiodarone used as an anti-arrythmics in ventricular tachycardia (VT) and ventricular fibrillation (VF)?

A

Amiodarone is a class III anti-arrhythmic agent that also has alpha- and beta-adrenergic blocking properties.

26
Q

When is adrenaline given in cardiac arrest?

A

Give adrenaline 1mg every 3-5 mins for both shockable and non-shockable rhythms. In practice this means at every other rhythm check or shock.

27
Q

When is amiodarone given in cardiac arrest?

A

In shockable rhythms give amiodarone 300 mg IV after 3 defibrillation attempts. Consider a further 150 mg after 3 defibrillation attempts.

28
Q

What are the reversible causes of cardiac arrest?

A

4 Hs and 4 Ts

  • Hypoxia (low levels of oxygen)
  • Hypovolemia (shock)
  • Hyperkalemia/hypokalemia/hypoglycemia/hypocalcemia (+ other metabolic disturbances)
  • Hypothermia
  • Thrombosis (coronary or pulmonary)
  • Tension pneumothorax
  • Tamponade (cardiac)
  • Toxins
29
Q

What is the immediate post cardiac arrest treatment?

A
  • Use ABCDE approach
  • Aim for SpO2 of 94-98%
  • Aim for normal PaCO2
  • 12-lead ECG
  • Treat precipitating cause
  • Targeted temperature management