Cardiac Arrest Flashcards
What are the 2 different algorithm paths used to treat Adult Cardiac Arrest?
-VF/pVT
-Asystole/PEA
Because many pts will sudden cardiac arrest demonastrate VF at some point in their arrests, most ACLS providers will often follow the VF/pVT algorithm—Why is this the best approach?
Rapidly treating VT according to this sequence is the best approach to restoring spontaneous circulation.
VF/pVT will require CPR until a defibrillator is available to deliver high-energy Unsynchronized shocks.
What are atonal gasps a S/O cardiac arrest?
-Agonal gasps are not normal breathing
-A pt who gasps usually appears to be drawing air in very quickly
-The mouth may be open and the jaw, head or neck may move w/ gasps
-Gasps may appear to be forceful or weak, usually a slow rate
-The gasp may sound like a snort, snore or groan
-Gasping is a S/O cardiac arrest.
Describe the shock energy for VF/pFVT for a biphasic defibrillator:
-Manufacturer recommendation (Zoll—120/150/200)
-If unknown, recommended shock energy use maximum available; second and subsequent shock doses should be equivalent & higher doses may be considered.
What is the shock energy dose for VF/pVT for a Monophysite defibrillator?
-360 J
What is the drub therapy for VF/pVT?
-Epinephrine 1 mg Q3-5 minutes IV/IO
-Amiodarone 300 mg IV/IO first dose; 150 mg IV/IO second dose
OR
-Lidocaine 1-1.5 mg/kg IV/IO first dose; 0.5-0.75 mg/kg IV/IO second dose
Coronary perfusion pressure (CPP) is Aortic Relaxation (diastolic) pressure minus Right Atrial relaxation (diastolic) pressure. During CPR, PCC correlates w/ both may cardiac blood flow & ROSC. In 1 human study, ROSC did not occur unless a CPP 15 mg Hg or higher was achieved during CPR. What can providers do to achieve the best CPP possible?
-Provide good chest compressions w/ minimal interruptions.
In adult cardiac arrest but do VF/pVT, the heart is quivering & not effectively pumping blood to vital organs. What can be done so that these pts have a much higher survival rate?
-Immediate chest compressions and CPR
The first 4-6 minutes after cardiac arrest, referred to as ‘Clinical Death’, no damage occurs to the brain. In 6-10 minute period, ‘biological death’ after cardiac arrest, damage is likely to occur to the brain. Brain damage is usually irreversible, except in special circumstances such as accidental hypothermia or cold water drowning.
Why is the interval from collapse (cardiac arrest) to defibrillation one of the most important determinants of survival from cardiac arrest?
-A common initial rhythm in out-of-hospital witnessed cardiac arrest is VF.
-pVT rapidly deteriorates to VF and then the heart is quivering & does not pump blood
-Electrical defibrillation is the most effective way to treat VF (delivery of shock to stop the VF) & pVT.
-The probability of successful defibrillation decreases over time.
-VF deteriorates to Asystole if not treated.
When VF is present, CPR can provider a small amount of blood flow to the heart & brain but cannot restore an organized rhythm. Why is early defibrillation important?
-The earlier defibrillation occurs, the higher the survival rate.
-Restoring a perfusing rhythm is more likely w/ immediate CPR & defibrillation within a few minutes after the initial arrest.
What should be done after defibrillation?
-Immediately resume CPR beginning w/ compressions. Do not perform a rhythm check at this point unless the pt is showing S/O life, such as ROSC.
What is the effect of Epinephrine of the body and how does it help w/ cardiac arrest?
-Epinephrine causes vasoconstriction which will increase cerebral & coronary blood flow during CPR by increasing mean arterial pressure and aortic diastolic pressure.
When is Magnesium Sulfate given?
-for Torsades de Pointes
-Loading Dose 1-2 g IV/IO over 20 minutes.
For the intubated pt, what is the best way to monitor CPR quality, optimize chest compressions & detect ROSC.
-Quantitative waveform caphography