Cardiac Flashcards

1
Q

Causes of 1st degree heart block

A

normal variant increased vagal tone inferior MI myocarditis hypokaelmia AV nodal blocking drugs: b blockers; ca channel blockers; digoxinl amiodarone

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2
Q

Causes of long QT

A

Idiopathic Acquired including Drugs: antiarrhythmics (incl. amiodarone; sotalol); TCA; Antipsychotics (risperidone); antibiotics (macrolides); antifungals (traizoles); antihistamines MI Hypokalemia myocarditis cardiopmyopathy hypothermia

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3
Q

Causes of mobitz type 2

A

Anterior MI Idiopathic fibrosis of conduction system cardiac surgery inflammatroy conditions (myocarditis, rheumatic fver) Autoimmunie (SLE, SS) Hyperkalemia Drugs: beta blockers, calcium channel blockers, digoxin, amiodarone

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4
Q

Causes of mobitz type I

A

drugs: beta blcoekrs; ca channle blockers; digoxin increased vagal tone inferior MI myocardtitis Post cardiac sugery

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5
Q

Causes of non-cardiogenic pulmonary oedema

A

ARDS high altitude plumonary eedema neurogenic pulmonary edema (narcotic OD; PE;Eclampsia; TRALI) in no-cardiogenic pulmonary oedema its factoris other than high pulmonary capillary pressure that are responsiblt for protein and fluid accumulation in alveoli

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6
Q

Causes of VF

A
  1. Structural Myocardial disease: infraction/ischemia; cardiomyopathy; HOCM; myocarditis; myocardial trauma 2. Metabolic/Electrlyte derangements: hypoxemia of any cause; hypo/hyper K; hypo Ca; hypo Mg; severe acidosis 3. Conduction abnormalities: prolonged WT; WPW and AF together; channelopathies 4. Drugs/Toxins; digoxin OD; TCA OD; 5. Environmental : electrocution; severe hypothermia
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7
Q

Complications of ECMO

A

hemmorraghe; vascular damage; thromboembolism; infection; thrombocytopenia (HITS); VA ECMO - cardiac thrombus VA ECMO: inadeqaue upper body/coronary/ brain perfusion VA ECMO: pulm hemmorhage

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8
Q

Complications of IABP

A

infection hemorhage vascular damage- arotic perforation thromboembolic phenomena renal failure failure to assist cardiac function balloon rupture

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9
Q

Contraindicatins to ECMO

A

irresivble cardiac or respiratroy failure anticoagulation contra-indicated likley futility (MOF; GVHD; Age; prolonged CPR; advanced malignancy)

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10
Q

Contraindications to IABP

A

significant Aoritc regurgitation Aortic aneurysm/ dissection significant PVD tortuous aorta uncontrolled sepsis uncontrolled bleeding

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11
Q

Contraindications to thrombolysis in PE

A

intracrania neoplasm; intracranial surgery/trauma < 2mths; internal bleedgin last 6/12, Hx of hemmorhagic stroke, sever HTN; surgery last 10 days;bleeding diathesis; thrombocytopenia; non-hemm stroke last2/12;

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12
Q

Diagnosis of infective endocarditis

A

2 major or 1 major +3 minor, or 5 minor criteria major criteria: positive BC for infective endocarditis with typical organisms positive echo for IE (oscialting mass on vlalve or adjacent structure; abscess; new dehiscence of prosthetic valve) New valvular regurg Minor crtiera: predisposition (heart condtion; IVDU); Fever>38; vascular phenomena (i.e.emboli); immunologic phenomena (glomerulonephritis; osler nodes; roths spots); microbiological evidence (BC positve not typical for IE); echo findings consitent with IE but does not meet major criteria

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13
Q

ECG features of LVH

A

left axis deviation left atrial enlargement Swave in v1 + R wave in V5/V6 >35 mm

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14
Q

ECg features of RVH

A

right axis deviation V1- dominant R wave V5/V6 dominant S wave

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15
Q

Features of left atrial enlargement on ECG

A

bifid p wave in lead II (2 peaks) bipahsic p wave in V1

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16
Q

Features of right atrial enlargement

A

peaked p wave in inferior leads (>2.5mm) andV1/V2 (>1.5mm)

17
Q

Indications for ECMO

A

VV ECMO: respiratroy failure PaO2/FiO2 <7.2 VA ECMO: refarctory cardiogenic shock ;cardiac arrest; failure to wean from bypass; bridge to cardiac transplant

18
Q

indications for surgery in infective endocarditis

A

hemodynamic instability abscess recurrent emoboli fungal or Qfever (coxiella burnetti)

19
Q

List investigations that help distinguish between cardiogenc and non-cardiogenic pulmonary edema

A

PCWP and CI- in cardiogenic pulmonary edema PCWP>18 mmH Serum BNP Echo PICo and EVLW (raised in both types) - can use pulmonary vascular permeability index to distinguish (raised in non cardiogenic; essentially te ratio of EVLW to Pulm blood voulme)

20
Q

List techniques/measurements available to assess the circulation status of a patient

A

Physical examiation- warm hands, UO, mentation Vital signs ABG- oxygenaion and lactate blood pressure repsonse to fluid challenge or leg raise Invasive monitoring: PICCO; LIDCO; FloTrac/Vigileo PAC Echocardiography -TTE; TOE Other methods: Impedance cardiography; gastric tonometry; trasncutatneous doppler

21
Q

mechanical strategies for supporting myocardial function

A

PEEP
IABP
Pacemaker
ECMO
VAD

22
Q

Potential indications for IABP

A

cardiogenic shock - although negative recent RCT intractable angina low CO post bypass (failure to wean post bypass) Cardiac surgery wth 2 of: left main>70%, LVEF<40%, unstable angina, reop bridge to heart transplant high risk PCI

23
Q

Prognosication following OOH arrest

A

Type of rhythm time to ROSC time to start CPR no sedation or NMBD required After 72 hours: GCS33 at 3 days) (EEG evidence of poor prognosis: burst surpresion; genealised seizures- less useful)

24
Q

Risks of cardioversion

A

failure need for sedation hypotension myocardial damage (usually transient) conduction abnormalities embolic phenomena burns damage to pacemaker

25
Q

Signs of severe Aoti stenosis

A

plateau pulse aortic thrill S4 splitting of S2 LVH- displaced apex beat LVF- late sign

26
Q

What is mobitz type 1

A

2nd degree heart block where PR interval lengthens and then drop every nth beat Treatment rarely needed

27
Q

What is mobitz type 2

A

2ns degree hear block where PR length is normla, but fail to conduct every nth PR interval Lesions is in bundle of His- always pathological