CAD PERIODONTOLOGY Flashcards
What is gingival recession?
The displacement of the marginal tissue where it becomes apical to the CEJ
name the two anatomical structures involved in recession
- Underlying buccal bone
- Marginal gingivae
Recession is a result of…
- Anatomical Abnormalities
- Periodontitis: the primary aetiology
- Tooth position
- Orthodontics: causing tooth movement, arch expanding, pushing the teeth outside the alveolar envelope
Name the Soft tissue anatomy (gingivae) in order from top to bottom
Free gingivae
Attached gingivae (Keratinised mucosa)
Mucogingival junction
Oral Mucosa
compare and contrast the Keratinised mucosa with the oral mucosa
- Keratinised mucosa:
1. Densely organised collagen fibres
2. Attached to the root surface/ periosteum
3. Resist movement by the action of lips/ cheeks - Oral mucosa:
1. Non-keratinised epithelial
2. Mobile tissue
3. Not designed to function like the gingivae
What is the role of keratinised gingivae?
- It is important for Aesthetic
- At least 2mm is required for gingival health
What is gingival biotype?
- Gingival biotype relates to shape, position and thickness of gingivae.
- It can be detected by looking at teeth as welll/ e.g. long crown usually have a thin biotype.
- Thin biotype are more likely to develop recession
Compare and contrast the Thick Flat bio type with the Thin Scalloped bio type
Thick Flat
Flat soft tissue and bony architecture
Thick, dense and fibrotic tissue
Wide band of keratinized tissue
Rectangular/ square crowns
Pronounced emergence profile
Long interproximal contacts to cervical 1/3
Thin Scalloped
Thin and fragile
Narrow band of keratinised
mucosa
Frequent osseous dehiscence
Triangular crowns
Short IP contacts within incisal third
Flat emergence profile
Name the aetiologies of recession
Poor oral hygiene*
Trauma*:
- Toothbrushing
- Iatrogenic: invasion of biological width
- Foreign bodies
- Frenal pull
- Traumatic overbite
- Self induced
Tooth position: when a tooth is outside gingival or buccal bone, it’s more susceptible to recession
Orthodontics
- Loss of buccal plate
- Buccal tooth movement always causes slight recession as a result of stretching of buccal mucosa
- No correlation between amount of keratinised gingivae and amount of recession observed
[PS1] What are the goals of perio tx? Include both clinical and patient-related factors.
1) Reduce BoP (no more than 20-30% BoP) (Clinical)
2) Reduce PPD to less than or equal to 5mm (Clinical)
3) Absence of pain (patient-related)
4) Satisfactory aesthetics + function (patient-related)
[PS1] What is the aim and objective of initial therapy?
Aim: To motivate pt to perform optimal plaque control
Objective: to achieve clean + infection free conditions
[PS1] What is aim of corrective therapy?
Addresses consequences of infection that was treated in initial therapy.
[PS1] What is aim and objective of supportive therapy?
Aim is to prevent re-infection and disease recurrence
Objective: to preserve gingival/periodontal health obtained via regular reviews and appropriate tx.
[PS1] What checks/treatments are involved in Supportive therapy?
-Assess deeper sites with BOP
-Re-instrumentation
-Fluoride application
-Check for integrity of restorations
-Assess vitality
-Take radiographs
[PS1] What is the aim of non-surgical periodontal tx (NSPT)?
To eliminate living microorganisms in the biofilm and calcified microorganism from the tooth surface and adjacent soft tissues
[PS1] What will NSPT achieve?
-Reduced inflammation
-Reduced pocketing and BOP
-Improve plaque control
[PS1] What are surrogate measures and name three.
Surrogate measures are predictors of further attachment loss or stability so we can determine if further tx is required and inform us of the prognosis of the teeth to inform the pt.
They are: PPD, BOP, PFS.
[PS1] What are three factors that may lead to further breakdown of teeth?
1) Patient
2) Tooth
3) Site
[PS1] Describe how further breakdown of teeth may occur at patient level.
-Increased no. of >6mm sites
-Perio tx without LA (less likely to succeed as less thorough debridement provided)
-% of BOP, extent of baseline attachment loss, PPD
[PS1] Describe how further breakdown of teeth may occur at tooth level.
-Furcation
-Mobility
-Limited residual periodontal support due to severe attachment loss
-PRFs
[PS1] Describe how further breakdown of teeth may occur at site level.
-We aim for BOP of no more than 20-30% = periodontal stability. If BOP present, there is 30% likelihood of disease progression.
-PPD >5mm present means risk of further attachment loss (esp. if with BOP).
[PS1] Name 6 factors that you could explain to a pt will make disease progression and tooth loss more likely.
1) Poorer OH
2) Irredular supportive therapy
3) Interleukin polymorphism (genetic factor)
4) Smoking
5) Age
6) Initial diagnosis of severe disease
[PS1] Why do we carry out surgical perio tx?
1) improve prognosis of tooth
2) Eliminate pockets >5mm and BOP
3) Facilitate plaque control
4) Regenerate lost periodontium
5) Resolution of mucogingival problems (overgrowth/recession)
[PS1] Name 4 surgical strategies.
1) Pocket reduction
2) Regeneration
3) Gingivectomy
4) Surgery for recession
[PS1] On a patient level, what describes an appropriate case for surgery?
1) Highly motivated pt, excellent OH
2) Compliance with OHI and appointments (Plaque free scores of > 70% on more than 1 occasion)
3) No medical contraindications
4) Non-smoker
5) Thorough non-surgical periodontal therapy completed
6) Localised residual pocketing
7)Surgery is part of comprehensive treatment plan (Including ongoing SPT)
[PS1] Why is it beneficial to carry out NSPT before perio surgery?
-Reduction in depth and number of pockets
-Reduce inflammation in tissue by removal of plaque/calc.
-Allow assessment of ‘true’ gingival contour
-Tissues firmer -> facilitating surgery (easier to raise flap) and reduce intraoperative bleeding and facilitate post-op healing
-Get better idea of prognosis and pts response to tx and their compliance with OH and form relationship with pt