Burns Lecture Flashcards
Function of skin
Protection from external trauma
Resists temperature variation (insulation/cooling)
Blocks entry of microorganisms
Prevents moisture loss from underlying tissues
Immune surveillance function
Decreases penetration of some types of radiation
Aids metabolism
Produces lubes and mositurizes
Touch!
Three zones of injury
Hyperemia
Stasis
Coagulation
Zone of Hyperemia
Outermost or most peripheral area to the site of wound or injury
Tissue is characterized by inflammatory changes and minimal damage
Zone of Stasis
Extends towards the central site of injury
Tissue is ischemic
Vessel endothelial damage –> thrombosis
Drying and infection –> deepen injury
24-48 hours post injury, progressive degeneration can be seen
Zone of Coagulation
Central-most area, where most energy causing the burn was absorbed
Thrombotic vessels and necrotic tissue
Minor burns lack this
Body Surface Area
Severity is proportional to the percent of BSA damage + depth
First degree burn
Injury to the superficial cells of the skin
Mild sunburn
Blisters are NOT seen
Some erythema and mild pain
Partial thickness burn that heals within 3-4 days with no scarring
Second Degree Burn: Superficial
** If damage to dermis is minimal
Involves tissue damage to both epidermis and dermis
Erythematous, blistered, weeping and painful
Blanch with pressure
Spared glands
Within 3 weeks, little or no scarring
Second Degree Burn: Deep
** Deeper damage of dermis
Difficult to distinguish from 3rd degree burns
Burn surface is pale and may be hardened or boggy
Less painful than more superficial burns
Slowly heals over 4-6 weeks
Permanent loss of hair follicles and glands
Third Degree Burns
Destruction of full thickness of skin
Pearly white, gray or brown and is dry and inelastic
Deep pressure = pain
Small: months to heal
Scarring and contracture formation
Excision and grafting to prevent contractures
Fourth Degree Burn
Well belwo the dermis and into subq tissue, fascia, bone
Blackened and charred
Dry and painless (nerve ending destruction)
Great risk of infection and other complications
Fluid loss bc of burns
Capillary damage is widespread and vasoactive mediators are released
Fluid, plasma, and electrolytes move to extravascular compartments
Fall in blood volume, cardiac output and tissue/organ perfusion
Critical in first 24 hours
Infections bc of burns
Threat in patients with stable BP/HR/RESP
- Sepsis and pnemonia
- Loss of mechanical protection of skin permits entry of microorganisms
G+: colonize immediately
G-: Day 5
Treatment of infections
Systemic antibiotics: Not with poor circulation; high bacteria per gram
Topical antibiotics: local wound care and strict infection control
Inhalation Injury bc of Burn
High mortality rate
Bronchospasm, ulceration of membranes, edema and impairment of ciliary clearance of bacteria
24-48 hours before symptoms show: hoarseness, dyspnea, tachypnea, and wheezing
– Needs proper recognition and diagnosis
Diagnosis and therapy of inhalation injury
Diagnosis
Management: endotracheal intubation and mechanical ventilation
Maintenance of fluid status is critical
Corticosteroids DO NOT influence survival rates– NOT RECOMMENDED (increase risk of infection and morbidity/mortality