Burns Case Conference Flashcards
NOTE: Includes facts that seem important enough, but especially quizzes facts on the slides that the professor starred (i.e. will probably be on the exam). Left out much of the powerpoint.
% TBSA of burn when systemic response is triggered
30%
3 cardiovascular effects
- Increased capillary permeability
- Peripheral and splanchnic vasoconstriction
- Decreased myocardial permeability
Respiratory effect
Bronchoconstriction
Metabolic effect
Increased metabolic rate (up to 3x)
Immunological effect
Non-specific down-regulation of the immune response (both cell-mediated and humoral)
5 steps in initial burn management
Trauma primary survey Secondary survey (history) Estimate TBSA of burn Estimate depth of burn Burn fluid resuscitation
Primary survey: ABCDE meaning
Airway Breathing Circulation Disability (neurologic evaluation) Exposure
Jackson’s Burn Zones
- Zone of coagulation
- Zone of stasis
- Zone of hyperaemia
Define zone of coagulation
Point of maximum damage (initial point of burn) = irreversible tissue loss due to coagulation of constituent proteins
Define zone of stasis
Zone of decreased tissue perfusion that is potentially salvageable at the periphery of the site of direct trauma
Define zone of hyperaemia
Outermost zone where tissue perfusion is acutally increased and where tissue will invariably recover
Define adequate and inadequate burn resuscitation based on Jackson’s burn zones
Adequate = zone of stasis preserved
Inadequate = zone of stasis lost
Parkland Formula for fluid resuscitation
4 cc / kg / % TBSA = total fluid to be administered in the first 24 hours
Purpose of Parkland Formula
ESTIMATION of fluid requirements in burn victim; should also monitor clinically
Fluid administered in burn victims
Ringer’s lactate
Reason for not using NS for burn victims
Risk of inducing a hyperchloremic acidosis
How to clinically monitor fluid requirements
Monitor urine output by means of a Foley catheter and titrate fluids to achieve a specific urine output
What % TBSA burns can be treated with oral fluids only?
<10 - 15% in children
How to administer fluids to burn victims based on Parkland Formula
- 1/2 of fluid given in first 8 h
* 1/2 of fluid given in next 16 h
Goal urine output in adults and in children burn victims
Adult = 30 cc/h Children = 1 cc/kg/h
2 goals of fluid resuscitation in burn management
1) Increase tissue perfusion in the zone of stasis in order to prevent irreversible tissue damage
2) Achieve enough volume to ensure end-organ perfusion while avoiding intracompartmental edema and join stiffness
AMPLE trauma history meaning
Allergies Medications Past medical history Last meal Events
Mechanisms of burn injury
Thermal (scals, flame, contact) Electrical Chemical Cold exposure (frostbite) Radiation burns
Mechanism of injury often associated with inhalation injury
Flame
Method to assess burn size
Standardized Lund-Bower diagram for 2nd and 3rd degree burns (NOTE: Do not include any 1st degree burns in calculation)
May also use rule of 9’s, but less accurate
%TBSA estimated by patient’s palm + fingers
1%
%TBSA of head and neck (rule of 9’s)
9%
%TBSA of trunk (rule of 9’s; anterior AND posterior)
36% total (18% each side)
%TBSA of arms (rule of 9’s)
9% each
%TBSA of legs (rule of 9’s)
18% each
%TBSA of genitalia and perineum
1%
Pathology of 1st degree burns
Involvement of epidermis only
Appearance of first degree burns
Dry, red and warm. Blanches to pressure. No blisters, no scarring once healed.
Sensation of first degree burns
Maybe painful
Healing time of first degree burns
Within 7 days
5 degrees of burns
- First degree
- Superficial 2nd degree
- Deep 2nd degree
- Third degree
- Fourth degree
Pathology of superficial 2nd degree burns
Involvement of epidermis and upper dermis; most adnexal structures intact
Appearance of superficial second degree burns
Pale pink. Smaller blisters. Wound base blanches with pressure
Sensation of superficial second degree burns
Increased; very painful and tender
Healing time of superficial 2nd degree burns
7 - 14 days
Scarring of superficial 2nd degree burns
Color match defect. Low risk for hypertrophic scarring
Pathology of deep 2nd degree burns
Involves epidermis and significant part of dermis. Only deeper adnexal structures intact
Appearance of deep second degree burns
Blotchy red or pale deeper dermis where blisters have ruptured
Sensation of deep 2nd degree burns
Decreased
Healing time of deep 2nd degree burns
over 21 days
Scarring of deep 2nd degree burns
High risk (up to 80%) of hypertrophic scarring
Pathology of 3rd degree burns
Epidermis, dermis and cell adnexal structures all destroyed
Appearance of 3rd degree burns
White, waxy, charred. No blisters. No capillary refill.
Sensation of 3rd degree burns
None
Healing time of 3rd degree burns
Cannot heal spontaneously
Scarring of 3rd degree burns
Wound contraction. Heals by secondary intention.
Pathology of 4th degree burns
Extension through the subcutaneous soft tissue to reach muscle, tendon, or bone. Associated with limb loss or the need for complex reconstruction
Burn Center Referral Criteria in terms of burn depth (4)
- 2nd and 3rd degree burns >10% of TBSA in patient 50 y-o
- 2nd and 3rd degree burns >20% TBSA in other groups
- 2nd and 3rd degree burns with serious threat of functional or cosmetic impairment that involve face, hands, feet, genitals, perineum, and major joints
- 3rd degree burns >5% TBSA in any age group
Burn Center Referral Criteria in terms of mechanism of injury (2)
- Electrical burns, including lightning injury
* Chemical burns with serious threat of functional or cosmetic impairment
Burn Centre Referral Criteria in terms of secondary considerations related to the burn (4)
- Inhalation injury
- Circumferential burns
- Patients with preexisting medical disorders that could complication management, prolong recovery, or affect mortality
- Any patient with concomitant trauma in which the burn injury poses the greatest risk of morbidity or mortality (physician judgment whether trauma or burn priority)
Leading cause of death in adult burn victims
Inhalation injury
Treatment of CO poisoning
High flow oxygen via a non-rebreathing mask
4 indications of hyperbaric oxygen
- CO level > 25%
- Loss of consciousness
- Severe metabolic acidosis
- Concern for end-organ ischemia
Define excharotomy
Release of ONLY burned skin
Define fasciotomy
Release of edematous muscles from their fascial compartments (incision through all involved fascial layers)