Burns Flashcards
How are burns classified?
Type eg thermal, chemical or electrical
Depth - superficial, partial, full
Size of burn - %TBSA rule of nines
Describe a superficial burn
Epidermis destroyed
Red, hypersensitive, rapid capillary return
Wound closes spontaneously in 7-10days
Describe a partial thickness burn
Superficial partial thickness - epidermis and superficial dermis destroyed
Red to pink, hypersensitive, rapid capillary return
Spontaneous closure 14 days
Deep partial thickness - epidermis and deep dermis destroyed
Creamy white, decreased light touch, delayed capillary return, >14 days closure
Describe a full thickness burn
Epidermis, dermis and underlying structures destroyed White, tan, black, leathery No light touch No capillary return >3 weeks closure, needs grafting
Describe pathophysiology of burn healing
Inflammatory phase (1-5/7) Proliferation phase (5/7-3/52) - fibroblasts produce collagen (disoriented) then differentiate into myofibroblasts - contractile, pull edges together. SCAR CONRACTURE. Epidermis and dermis not connected.
Remodelling phase (3wks - 18months) - cross linking collagen in parallel
Describe the initial medical Mx for a burn patient
Ax Tetanus injection Fluid Airway Mx Inhalation injury Mx Escharatomies Skin grafting Prevention and Mx infection
Describe the pathophysiology of an inhalation injury
Acute pulmonary insufficiency (36hrs) - hypoxia due to CO, atelectasis due to coughing and bronchospasm, upper airway oedema Pulmonary oedema (6-72hrs) - surfactant is denatured, decreased lung compliance Bronchopneumonia (3-10days) - pseudo membrane sheds, plugging and decreased secretion clearance, distal atelectasis
Briefly describe the layers of the skin
Epidermis - keratin toughens and waterproofs, no blood vessa, melanin for UV protection
Dermis - papillary and reticular layer, blood and lymph vessels, elastin and collagen
Epidermal appendages - heart follicles, sweat glands, centre for regeneration of epidermis
What are the consequences of an inhalation injury immediately and longer term?
Immediate - bronchospasm, denatured surfactant, high levels of CO bound to haemoglobin and decreased PaO2, pulmonary oedema
Mid - pseudomembrane formation and shedding
Long term - bronchiectasis, recurrent chest infections, pulmonary fibrosis, persistent impairment of lung function, respiratory mm weakness, tracheal stenosis
Discuss positive and negative effects of physiotherapy interventions for a patient with a burn injury
Early mobilisation - prevent contractures and deconditioning, minimise mm catabolism. Causes PAIN and can compromise wound healing and graft take
EOR holds - 1min+ improve ROM and prevent contractures, compromise wound healing
Contract-relax - as above
Exercise and stretch opposite to contracture
Discuss modifications to physio for burns patients to minimise detrimental effects
Time with pain meds
Protect wound with padding and coban for early mobility
NWB activity