Bio-Neuro Flashcards
Medulla
Very important
Vital signs
Pons
Connects halves of cerebellum
RL movement integration
Colliculi
Visual superior
Auditory inferior
Routes
Substantia Nigria
Motor activity
Reward
Reticular formation
RAS
Sleep arousal consciousness
Thalamus
Relay station
Hypothalamus
Everything
Drives homeostasis emotion
Basal ganglia
Voluntary movements reflexes facial recognition planning and organizing
Amygdala
Emotion and emotional memory
Hippocampus
Learning and memory
Cingulate cortex
Pain signals
Acetylcholine
Muscannic and nicotinic receptors
Nervous system and memory
Acetylcholine disorders
Alzheimer’s
Myasthenia Gravis
Dopamine
Personality mood memory sleep movement
Dopamine disorders
Schizophrenia
Tourette’s
Parkinson’s
Norepinephrine
Mood attention dreams learning depression
Serotonin
Mood hungry temperature sex sleep aggression
GABA
Inhibitory
Eating sleep motor control
GABA disorders
Anxiety
Huntington’s
Seizures
GABA medication
Benzos neurotransmitter
Glutimate
Excitatory
Long term memory exotoxicity
Glutimate (exotoxicity) disorders
Seizures stroke Huntington’s Alzheimer’s
Endorphins
Inhibitory neuromusculator
Analgesic pleasure
Huntingtons etiology
decreased GABA, increased glutamate in basal ganglia
Huntingtons symptoms
- emotional cognitive 2. motor (fidget, klutz) 3. chorea.- jerky involuntary movements 4. degenerative (ends with athetosis- slowed movements and dementia)
Parkinsons etiology
decreased dopamine in the substantia nigra (thalamus and frontal lobe too)
Parkinsons symptoms
tremors, rigidity, akathisia (restlessness), slowed speech and movement (akinesia), depression
multiple sclerosis (MS) etiology
progressive degeneration of myelin
MS symptoms
autoimmune; relapsing-remitting type evolves into secondary progressive type; tremors, speech, swallowing, hearing, mood, it all goes
Alzheimers NT
acetylcholine
Alzheimers stages
steady and progressive decline in memory and 1+ other cognitive area. 1. anterograde/declarative memory 2. retrograde amnesia 3. severe deterioration of memory and functioning
brain damage to dorsolateral PFC
dysexecutive syndrome (DUH) = simple minded, apathetic, impaired judgement, planning, insight, and organization
brain damage to orbitofrontal pfc
disinhibition syndrome= pseudopsychopathy, inappropriate behavior (ORBIT around ME)
damage to mediofrontal PFC
apathetic syndrome (MEH)= pseudodepression
damage to Broca’s area PFC
Broca’s aphasia (BUH, BUH, BROKEN, non-fluent)- can’t produce speech, frustrated
Wernicke’s aphasia
fluent aphasia (WHAT), can’t understand language, produce language but it doesn’t make sense
damage to parietal cortex
somatosensory cortex, all the “ias”, apraxia, agnosia, etc.