Behavioural Addictions Flashcards
Reilly and Smith
Substance addictions and GD have similar symptom profile, co-morbidities and same heritability
Dong et al
DTI 16 IGD and 15 controls = IGD = greater fractional anisotropy in thalamus and L posterior cingulate = means increased white matter connectivity
Ko et al
IGD = increased bilateral dlPFC, precuneus, L parahippocampus, posterior cingulate and R anterior cingulate. Activation positively correlated with self-reported gaming urge under cue exposure
NOTE similar areas to substance abuse
de Ruiter et al
Stop signal task GD, smokers and controls = no difference in behavioural performance (strange). GD and smokers = hypo-responsiveness of dmPFC during inhibition
Potenza et al
Stroop task cognitive inhibition = lower activation in L vmPFC in GD compared with controls when processing incongruent vs congruent stimuli
Crockford et al
Increased activation in R dlPFC to gambling cues in GD compared with controls. dlPFC implicated in WM and conditioned responses so why increased activity if less control? = looking at ATTENTIONAL mechanisms not executive control
Reuter et al
fMRI 12 GD, 12 controls = guessing card task activates ventral striatum = lower activation in R ventral striatum and vmPFC in GD than controls when receive monetary reward. Activation -vely correlated with gambling severity. Deficient reward system similar to substance abuse?
Steeves et al
PD patients with GD showed greater decreases in dopamine binding potential in ventral striatum (14% vs 8%) = similar to substance abuse
Hewig et al
GD and controls = no difference in response to bust. In response to no-bust = more positive ERP for GD
Tanabe et al
Gambling and non-gambling substance users and controls. Decision making during risk task and fMRI = lower vmPFC in SD and SDGD
Garcia-Garcia et al
Substance vs non-substance addictions = general overlap of brain areas, not specific
