BCCH Flashcards
NICU pH, pO2, pCO2, bicarbonate, BE, spO2
7.25-7.45, 50-80, 45-55, 22-26, 0+-2, sats <36 = 88-92, >/=36 = 90-95
PICU Vt, RR, Ti, VE, PEEP
6-8ml/kg, 10-30 br/min, 0.6-1.2sec, 100-200ml/min/kg, 5-10 cmh2o
NICU Vt, Ti, RR, VE, PEEP
Vt = 4-6ml/kg, Ti= 0.35 - 0.55, RR 40-60, VE = 200-300ml/min/kg, PEEP = 5-8
Croup: area + degree of obstruction, age, common causes, signs, tx
Laryngotracheobronchitis; inflammation of larynx/cords, trachea, bronchi; partial-complete obstruction; age = 6 mo-3 yr; seasonal due to viral etiology: parainfluenza 1 +3, influenza a+b, rsv, rhinov, corona, HMV, adenovirus; signs: increased WOB, Barky cough, insu stridor, fever, hypoxemia (severe); dx: signs + hx adequate (cxr steeple sign but not necessary). Tx: neb epi, corticosteroids
describe coronary circulation
LV - aortic valve - ascending aorta - brachiocephalic artery>right carotid>RA and RV supply & left carotid>circumflex (LA and posterior LV supply) + left anterior descending artery (critical, supplies anterior and inferior LV)
Why PPV is beneficial for CHF Pts? (hint: aortic wall tension)
PPV generally sucks for heart (collapses IVC & RA, decreased RV preload, increased RV afterload, decreased LV preload and compliance) but PPV actually reduces LV afterload!!!
- this is bc with PPV, Ppl becomes more positive
- LV afterload is represented by aortic wall tension (bc that is what the LV has to pump against)
- aortica wall tension (T) = Ptm x r / 2 according to Laplace’s law: P = 2T/r
- Ptm = vascular pressure - Ppl
- so PPV makes Ppl more positive which makes Ptm smaller and therefore aortic wall tension decreases = LV afterload decreases
- PPV is like mechanical milrinone for the LV in CHF pts: + pressure assists LV contraction to overcome aortic valve opening + decreases the LV afterload
7 acyanotic heart defects + how to tell which ventricle is resulting in dilation
atrial septal defect, VSD, AVSD, PDA, RVOTO/pulmonic stenosis, aortic stenosis, coarctation of aorta
= L to R shunt
-dilation = stenosis of ventricle (pressure loaded) or receiving the volume of shunt (volume loaded)
6 cyanotic heart defects + memory trick
any that starts with "T" -tetralogy of fallot -tricuspid atresia -transposition of the Great Arteries -total anomalous pulm venous return -truncus arteriosus -hypoplastic left heart syndrome = R to L shunt
SVR and PVR eqn
SVR = MAP-CVP / CO PVR = PAP - PCWP/CO
6 factors that affect PVR
oxygen, acid-base status, pulm vasodilators (nitric or sildenafil at childrens), PPV/PEEP, lung volumes, valsalva/crying (increases intrathoracic pressures)
explain why hyperinflation AND atelectasis both cause increased PVR
hyperinflation = compresses capillaries = increased PVR.
atelectasis = decreases tethering and collapses extra-alveolar vessels = increased PVR
-therefore, keep lungs recruited at FRC to keep extra-alveolar vessels tethered open, and capillaries not too compressed to minimize/decrease PVR
most common CHF in pediatric population
VSD!!
main side effect of prostaglandin E/alprostadil therapy?
apnea
-need to be bagged but this will increase PVR
Epiglottitis area + degree obstruction, age, etiology, acute or delayed onset, 4 s&s, dx and cxr, tx
Complete obstruction of upper airway, affects epiglottis “cherry red”; 2-6 y.o. And adults affected; H influenzae B + group a strep; acute onset no prodrome period and not seasonal; signs/sx: stridor, dysphagia, drooling, distress, no cough, muffled voice, tripoding; dx hx and clinical presentation adequate, thumb sign cxr; tx: secure airway awake intubation with lidocaine, oxygen, IV abx and CS
Eqn PEDIATRIC ETT sizing, sxn catheter sizing
Cuffed= (age/4)+3.5
Uncuffed= (age/4)+4
Sxn: IDD X 2