BCCH Flashcards

1
Q

NICU pH, pO2, pCO2, bicarbonate, BE, spO2

A

7.25-7.45, 50-80, 45-55, 22-26, 0+-2, sats <36 = 88-92, >/=36 = 90-95

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

PICU Vt, RR, Ti, VE, PEEP

A

6-8ml/kg, 10-30 br/min, 0.6-1.2sec, 100-200ml/min/kg, 5-10 cmh2o

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

NICU Vt, Ti, RR, VE, PEEP

A

Vt = 4-6ml/kg, Ti= 0.35 - 0.55, RR 40-60, VE = 200-300ml/min/kg, PEEP = 5-8

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Croup: area + degree of obstruction, age, common causes, signs, tx

A

Laryngotracheobronchitis; inflammation of larynx/cords, trachea, bronchi; partial-complete obstruction; age = 6 mo-3 yr; seasonal due to viral etiology: parainfluenza 1 +3, influenza a+b, rsv, rhinov, corona, HMV, adenovirus; signs: increased WOB, Barky cough, insu stridor, fever, hypoxemia (severe); dx: signs + hx adequate (cxr steeple sign but not necessary). Tx: neb epi, corticosteroids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

describe coronary circulation

A

LV - aortic valve - ascending aorta - brachiocephalic artery>right carotid>RA and RV supply & left carotid>circumflex (LA and posterior LV supply) + left anterior descending artery (critical, supplies anterior and inferior LV)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Why PPV is beneficial for CHF Pts? (hint: aortic wall tension)

A

PPV generally sucks for heart (collapses IVC & RA, decreased RV preload, increased RV afterload, decreased LV preload and compliance) but PPV actually reduces LV afterload!!!

  • this is bc with PPV, Ppl becomes more positive
  • LV afterload is represented by aortic wall tension (bc that is what the LV has to pump against)
  • aortica wall tension (T) = Ptm x r / 2 according to Laplace’s law: P = 2T/r
  • Ptm = vascular pressure - Ppl
  • so PPV makes Ppl more positive which makes Ptm smaller and therefore aortic wall tension decreases = LV afterload decreases
  • PPV is like mechanical milrinone for the LV in CHF pts: + pressure assists LV contraction to overcome aortic valve opening + decreases the LV afterload
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

7 acyanotic heart defects + how to tell which ventricle is resulting in dilation

A

atrial septal defect, VSD, AVSD, PDA, RVOTO/pulmonic stenosis, aortic stenosis, coarctation of aorta
= L to R shunt
-dilation = stenosis of ventricle (pressure loaded) or receiving the volume of shunt (volume loaded)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

6 cyanotic heart defects + memory trick

A
any that starts with 
"T"
-tetralogy of fallot
-tricuspid atresia
-transposition of the Great Arteries
-total anomalous pulm venous return
-truncus arteriosus
-hypoplastic left heart syndrome
= R to L shunt
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

SVR and PVR eqn

A
SVR = MAP-CVP / CO
PVR = PAP - PCWP/CO
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

6 factors that affect PVR

A

oxygen, acid-base status, pulm vasodilators (nitric or sildenafil at childrens), PPV/PEEP, lung volumes, valsalva/crying (increases intrathoracic pressures)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

explain why hyperinflation AND atelectasis both cause increased PVR

A

hyperinflation = compresses capillaries = increased PVR.
atelectasis = decreases tethering and collapses extra-alveolar vessels = increased PVR
-therefore, keep lungs recruited at FRC to keep extra-alveolar vessels tethered open, and capillaries not too compressed to minimize/decrease PVR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

most common CHF in pediatric population

A

VSD!!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

main side effect of prostaglandin E/alprostadil therapy?

A

apnea

-need to be bagged but this will increase PVR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Epiglottitis area + degree obstruction, age, etiology, acute or delayed onset, 4 s&s, dx and cxr, tx

A

Complete obstruction of upper airway, affects epiglottis “cherry red”; 2-6 y.o. And adults affected; H influenzae B + group a strep; acute onset no prodrome period and not seasonal; signs/sx: stridor, dysphagia, drooling, distress, no cough, muffled voice, tripoding; dx hx and clinical presentation adequate, thumb sign cxr; tx: secure airway awake intubation with lidocaine, oxygen, IV abx and CS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Eqn PEDIATRIC ETT sizing, sxn catheter sizing

A

Cuffed= (age/4)+3.5
Uncuffed= (age/4)+4
Sxn: IDD X 2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Consequences of VSD

A

Right ventricular hypertrophY SECONDARY to pulmonary vascular remodelling resulting in high PVR
+ can deteriorate faster than ASD due to direct flow into pulmonary system leading to increased volume into LV leading to LV hypertrophY and CHF

Major difference between ASD and VSD:
VSD = LV hype trophy bc of increased blood volume back; ASD= RV hypertrophY due to vascular remodelling but happens slower

17
Q

Most common pediatric vs neonatal CHDs

A

Peds: VSD
neonates: PDA

18
Q

What is eisenmenger’s syndrome

A

Reversal of a VSD shunt = instead of left to right shunt it becomes R to L shunt due to PVR > SVR

19
Q

Name of the space between RV and the PA (RVOT)

A

Infudibulum (mal development can lead to VSD)

20
Q

Genetic defect associated with AVSD + post-repair concerns for these pts

A

Trisomy 21 And pulm htn due to shared blood flow;

Post op pts are at risk of pulm htn due to RV becoming so strong even after repair which can cause mitral valve stenosis/insufficiency. Provide O2 to these pts to reducs pulm htn

21
Q

What CHD repaired via Ross procedure?

A

Aortic stenosis:
Pulmonary autograft where pulm valve replaces the stenotic aortic valve, then human donor pulm valve replaces the pts pulm valve
Benefits: anti coagulation therapy not necessary, low regurgitation and dysfunction, valve grows with the patient, same life expectancy as general population

22
Q

What needs to be done for HLHS pt with no ASD?

A

No ASD means no way for oxygenated blood to get to systemic circulation therefore emergency cath lab for BAS= balloon atrial septostomy