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Blood Bank 1 > BB 1 Midterm 2 > Flashcards

BB 1 Midterm 2 Flashcards

1
Q

ABO Phenotypes

A

A, B, O, AB

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2
Q

B lectin

A

Bandeiraea Simplifiolia

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3
Q

H lectin

A

Ulex Europaeus

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4
Q

A1 lectin

A

Dolichos biflorus

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5
Q

Mixed field (mf)

A

Observation of multiple cell populations in blood

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6
Q

Phenotype

A

Blood group characteristics based on antigens

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7
Q

Terminal sugar of B antigen

A

Galactose

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8
Q

Terminal sugar of A antigen

A

N-acetylgalactoseamine

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9
Q

Terminal sugar of H-antigen

A

Fucose

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10
Q

H substance (most to least)

A

Order of abundance: O>A2>B>A2B>A1>A1B

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11
Q

A frequency

A

45%

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12
Q

B frequency

A

20%

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13
Q

O frequency

A

45%

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14
Q

AB frequency

A

5%

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15
Q

ABO antigens

A

A, B, H

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16
Q

Rh Antigens

A

D, C, c, E, e

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17
Q

Rh disease link

A

Hydrops Fetalis (HDN)

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18
Q

Rh: order of immunogenicity

A

D>c>E>C>e

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19
Q

Anti-f

A

c and e on same haplotype

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20
Q

R1 antigen

A

DCe

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21
Q

R2 antigen

A

DcE

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22
Q

R0 antigen

A

Dce

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23
Q

Rz antigen

A

DCE

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24
Q

r’ antigen

A

dCe

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25
Q

r’’ antigen

A

dcE

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26
Q

r antigen

A

dce

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27
Q

ry

A

dCE

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28
Q

Rh positive

A

D antigen expressed

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29
Q

D frequency

A

85%

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30
Q

C frequency

A

70%

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31
Q

c frequency

A

80%

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32
Q

E frequency

A

30%

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33
Q

e frequency

A

98%

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34
Q

MNS Antigens

A

M, N, S, s, U

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35
Q

MNS disease link

A

Malaria falciparum and Renal disease

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36
Q

MNS chromosome

A

Chomosome 4

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37
Q

M frequency

A

85%

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38
Q

N frequency

A

70%

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39
Q

S frequency

A

50%

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40
Q

s frequency

A

90%

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41
Q

U frequency

A

100%

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42
Q

MNS nulls

A

Mk

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43
Q

MNS disequilibrium frequency

A

Ns>Ms>MS>NS

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44
Q

P/Glob Antigens

A

P1

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45
Q

P/Glob disease link

A

Paroxysmal cold hemoglobinuria and Spontaneous abortions

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46
Q

P1 phenotype

A

P1, P, Pk antigens

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47
Q

P2 phenotype

A

P, Pk antigens

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48
Q

p phenotype

A

Null (no antiens)

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49
Q

P1k phenotype

A

P1, Pk

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50
Q

P2k phenotype

A

Pk

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51
Q

P2 antibody

A

Anti-P1

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52
Q

p (null) antibody

A

Anti-PP1Pk

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53
Q

P1K antibody

A

Anti-P

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54
Q

P2k antibody

A

Anti-P1 and Anti P2

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55
Q

P/Glob chromosome location

A

Chromosome 22

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56
Q

P1 frequency

A

80%

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57
Q

P2 frequency

A

100%

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58
Q

P frequency

A

100%

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59
Q

Kell Antigens

A

K, k, Kpa, Kpb, Jsa, Jsb

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60
Q

Kell null

A

K null

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61
Q

Kell chromosome

A

chromosome 7

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62
Q

K frequency

A

9%

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63
Q

k frequency

A

100%

64
Q

Kpa frequency

A

0%

65
Q

Kpb frequency

A

100%

66
Q

Jsa frequency

A

1%

67
Q

Jsb frequency

A

100%

68
Q

Kell, MNS, Lutheran

A

DTT destroys what system

69
Q

Lewis Antigens

A

Lea, Leb

70
Q

Lewis expression determines

A

Secretor Status

71
Q

Lewis Chromosome

A

Chromosome 19

72
Q

Lea frequency

A

20%

73
Q

Leb frequency

A

70%

74
Q

Duffy Antigens

A

Fya, Fyb, Fy3/Fy5

75
Q

Duffy disease linkage

A

Malaria vivax

76
Q

Duffy chromosome

A

Chromosome 1

77
Q

Duffy Fy(a-b-) disease correlation

A

P. Vivax resistant

78
Q

Fya frequency

A

60%

79
Q

Fyb frequency

A

100%

80
Q

Fy3/FY5 frequency

A

100%

81
Q

Fy (a-b-) incidence

A

Common in west africa

82
Q

Kidd Antigens

A

Jka, Jkb, JK3

83
Q

Kidd chromosome

A

Chromosome 18

84
Q

Kidd null

A

Jk(a-b-) or Jk3-

85
Q

Jka frequency

A

75%

86
Q

Jkb frequency

A

75%

87
Q

JK3 frequency

A

100%

88
Q

Kidd clinical significance

A

Delayed hemolytic TRXN

89
Q

Kidd System Marked effect

A

Marked Dosage effect

90
Q

Lutheran Antigens

A

Lua, Lub

91
Q

Abnormal Result common in Lutheran

A

Mixed Field common in

92
Q

Lutheran Chromosome

A

Chromosome 19

93
Q

Lutheran nulls

A

In (Ln) dominant, Recessive, X-linked

94
Q

Lua frequency

A

5%

95
Q

Lub frequency

A

99%

96
Q

Lutheran disease correlation

A

Stickiness and Tumor Metastasis

97
Q

Antigens with significant Racial Frequency differences

A

S, C, c, P1, Jsa, Fya, Fyb, Jkb

98
Q

Neutralization principle

A

Uses substance to bind antibody is the principle of

99
Q

Neutralization substance of P1

A

Hydatid cyst fluid

100
Q

Neutralization substance of Lewis

A

Saliva

101
Q

Neutralization substane of Chido/Rodgers

A

Serum (complement)

102
Q

Neutralization substance of Sd3

A

Urine

103
Q

Neutralization substance of I

A

Human Breast milk

104
Q

Gerbich antigens

A

Ge 1-4

105
Q

Gerbich presentation

A

Elliptocytes

106
Q

Diego Antigens

A

Dia/Dib and Wra/Wrb

107
Q

Diego incident

A

Dia high in S.American indians

108
Q

Colton Antigens

A

Coa/Cob, Co3 (null

109
Q

Dombrock antigens

A

Doa/Dob, Gya (null)

110
Q

Dombrock frequency

A

caucasian frequency matches duffy

111
Q

Cartwright antigens

A

Yta/Ytb

112
Q

Cartwright frequency

A

Ytb more common in Israelis

113
Q

Chido/Rodgers

A

Ch 1-6, Rg 1-3

114
Q

Chido/Rodgers action

A

Complement (C4) absorbed

115
Q

Cromer antigens

A

Cra, Tca/Tcb/Tcc, Dra

116
Q

Cromer frequencies

A

Cra high in black pop, Dra high in Japanese

117
Q

Knops antigens

A

Kna, Knb, McCa/McCb, Sla/Vil

118
Q

Indian antigens

A

Ina, Inb

119
Q

Indian prevalency

A

Prevalent in Iran and india

120
Q

Landsteiner Weiner antigens

A

Lwa/Lwb

121
Q

Scianna antigens

A

Sc1/Sc2, Sc3 (null)

122
Q

Xga antigens

A

Xga CD99

123
Q

Xga presentation

A

More common in females (x-linked)

124
Q

Ralph antigen

A

MER2

125
Q

Ralph antigen location

A

Platelets

126
Q

OK antigens

A

OKa

127
Q

OK disease link

A

Receptor for plasmodium falciparum

128
Q

JMH antigen

A

JMH

129
Q

HLA antigen

A

Bga, Bgb, Bgc

130
Q

Warm Reacting antigens

A

Kell, Duffy, Kidd, Lub, some P, some M

131
Q

Cold reacting antibodies

A

ABO, Rh, MNS some, Some P, Lewis antigens Lua

132
Q

Antibody groups that fix complement

A

ABO, Kiss, Lewis, P, Duffy/Lutheran (some)

133
Q

Antibody groups that don’t fix complement

A

Rh, Kell, MNS, P1/p

134
Q

Enzymes enhance these antigen

A

Rh, Kell, Kidd, Lewis, P (enzymes)

135
Q

Enzymes destroy these antigens

A

Duffy and MNS (Enzymes)

136
Q

IgM Antibodies

A

Cold Antibodies

137
Q

IgG Antibodies

A

Warm Antibodies

138
Q

Use of LISS

A

Reduce Zeta Potential

139
Q

Use of PEG

A

Remove water volume

140
Q

Use of Albumin

A

Increase collisions

141
Q

Use of Bromelin

A

Reduce steric hindrance

142
Q

Use of Polybrene

A

Polymer nonspecific binding

143
Q

Gel

A

Precipitation through Polyacrylamide gel matrix

144
Q

Solid Phase

A

Adherence of antibody to RBC antigen on solid surface

145
Q

Alleles

A

SNPs, alternate form of gene

146
Q

Null

A

Absence of antigenic expression

147
Q

Identifiable complement protein on the outside of red cell

A

C3B

148
Q

ABO stimulated

A

IgG

149
Q

ABO Naturally occurring

A

IgM

150
Q

Kx (decreased kell)

A

Acanthocytes

151
Q

K null

A

McLeod syndrome

152
Q

Most common antibody

A

Anti-K

153
Q

Sickle cell pt phenotyping

A

K antigen phenotyping

154
Q

Lutheran is critical for ___ to ___

A

adhesion to laminin

155
Q

Patient plasma with patient cells at IAT

A

Autocontrol

156
Q

Clinically significant

A

Causes TRXN or HDN

Blood Bank 1 (9 decks)

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