Avian Non-Infectious Diseases Flashcards

1
Q

Why can broken blood feathers be dangerous? What treatment is recommended?

A

risk of hemorrhaging out and extremely painful

pluck feather and apply Qwick stop or skin glue to promote hemostasis

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2
Q

In what birds are feather cysts most common? What are 2 options for treatment?

A

Canaries —> can be mistaken for xanthomas or fibrosarcomas

  1. lance cyst, remove feather material, and cauterize the follicle with silver nitrate
  2. surgically excise entire feather follicle
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3
Q

What is suspected to be the cause of uropygial gland impaction? What 4 treatments are recommended?

A

improper nutrition —> hypovitaminosis A (check choanal papilla)

  1. lance affected lobe
  2. antibiotic based on C&S
  3. flush with chlorhexidine
  4. heal via secondary intention
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4
Q

What causes pododermatitis in birds? What signs are seen? What birds are most commonly affected?

A

bumblefoot —> Staphylococcus aureus

progressive inflammation of the foot - mild erythema —> abscessation and osteomyelitis

  • raptors
  • psittacines
  • poultry
  • waterfowl
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5
Q

What are 6 predisposing factors to pododermatitis?

A
  1. obesity
  2. overgrown talons/nails
  3. sedentary lifestyle
  4. trauma
  5. improper perching
  6. poor sanitation/husbandry/nutrition - bird requires a variety of perches to avoid sitting on the same part of foot, ground dwellers need clear floors without abrasive material
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6
Q

What are the 5 grades of pododermatitis?

A
  1. early insult, no apparent infection
  2. infection of tissues near insult
  3. infection with swelling and discharge, likely lame
  4. above + involvement of deeper structures
  5. crippling deformity, loss of function
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7
Q

How is pododermatitis diagnosed? What are 5 options for treatment?

A

culture, bloodwork, radiographs

  1. fix husbandry
  2. bandage to remove weight from plantar aspect of foot - interdigital, donut (severe)
  3. surgical debridement - III or above
  4. topical therapy - anti-inflammatories (DMSO, CEH cream, preparation H) + antibiotics
  5. systemic antibiotics + analgesia
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8
Q

What is prognosis of pododermatitis like?

A
  • Gr 1-3 = good with quick recovery
  • Gr 3-5 = guarded to poor with prolonged recovery (can take 2-6 months!)
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9
Q

What is Amazon foot necrosis syndrome? What etiology is suspected?

A

pruritic, ulcerative lesions on feet/legs with self-mutilation common —> poor prognosis

allergic or immune-mediated

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10
Q

What is constricted toe syndrome? What is the most common cause? How is it treated?

A

fibrous band of tissue on the toe of Psittacine neonates constricts one or more digits

low humidity in incubators or brooders

surgical excision of band

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11
Q

What kind of injury are mammal bite wounds to birds? What treatment is recommended?

A

true emergency - Pasteurella multocida is part of the normal flora of a cat’s mouth

antibiotic therapy immediately —> Fluoroquinolones or Penicillins are best choices

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12
Q

Antimicrobials used in avian species:

A
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13
Q

What birds are most commonly affected by xanthomas? What are they? Where are they most commonly found?

A

Cockatiels and Budgerigars

locally invasive, benign, sometimes pruritic, yellow friable masses

ventral coelom, hip, thigh, wing, around face

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14
Q

How are xanthomas diagnosed? How are they treated?

A

cytology - foamy microphage, multinucleated giant cells, cholesterol crystals

  • surgical excision (recurrence possible)
  • low fat/protein diet
    (large = guarded prognosis)
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15
Q

What are 6 possible causes of feather picking/feather destructive behavior?

A
  1. behavioral
  2. pain response - growing, arthritis, cysts
  3. aggression from conspecifics - finches in breeding season
  4. improper husbandry - decreased humidity
  5. infectious - PBFD
  6. endocrine - hypothyroidism causes decreased molting, discoloration, and flaky skin

(diagnosis of exclusion)

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16
Q

What birds commonly develop lipomas? How do they appear? Treatment?

A

birds on high fat seed diets

soft, pale, yellow, encapsulated and lobulated masses in the subcutis, commonly on the sternum

surgical excision and diet correction

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17
Q

What kind of tumors are most commonly found in the uropygial gland? How do they appear? What treatments are recommended?

A

adenocarcinoma +/- SCC

superficial, crusty, proliferative, localized to the lower back and pygostyle region

  • debulk - difficult
  • cryotherapy - recurrence likely
  • radiation therapy - promising
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18
Q

When is polyostotic hyperostosis considered normal? What are 3 pathogenic causes?

A

pre-ovulatory females - calcium storage in long bones

  1. cystic ovaries - secrete estrogen
  2. ovarian and oviduct neoplasia - secrete estrogen
  3. paraneoplastic syndrome
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19
Q

What is splay-leg syndrome? What are 3 suspected causes?

A

laxities of the ligaments of the stifle and/or angular deformities of the femur, tibiotarsus, and tarsometatarsus, common in younger birds

  1. parental oversitting - can’t strengthen muscles and ligaments
  2. nutritional deficiencies (MBD)
  3. insufficient support/substrate in nestbox - slippery, lacks traction
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20
Q

What are 3 options for treating splay-leg syndrome?

A
  1. hobbling
  2. coaptation
  3. surgery - risks to circulation and innervation + mutilation common with older birds
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21
Q

What birds develop hypovitaminosis A? What does it lead to?

A

birds fed a seed-based diet

squamous metaplasia —> salivary gland cysts/abscesses, blunting choanal papilla —> predisposes to respiratory infections

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22
Q

What inhaled toxin are birds commonly exposed to in the home? What does this cause?

A

Teflon (polytetrafluoroethylene) - burned nonstick cookware, ovens in self-cleaning mode, burning butter or oils, candles, or air fresheners

  • irreversible diffuse pulmonary edema/congestion
  • regurgitation
  • dyspnea
  • ataxia
  • sudden death
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23
Q

How are birds that inhaled toxins treated? What is prognosis like?

A
  • oxygen therapy
  • diuretics
  • bronchodilators
  • anti-inflammatories
  • antimicrobials

poor —> lung changes likely irreversible

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24
Q

What are the 4 most common cause of nasal discharge in birds?

A
  1. accumulation of dead cells, dust, or debris
  2. FB
  3. squamous metaplasia from hypovitaminosis A
  4. rhinitis

(common for healthy birds to sneeze several times a day)

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25
Q

What is the most common cause of periorbital swelling? What are 4 etiologies?

A

conditions affected the infraorbital sinus

  1. sinusitis
  2. granuloma - associated with hypovitaminosis A
  3. trauma
  4. orbital mass - abscess, neoplasia
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26
Q

What are the 3 most common causes of tracheal disease in birds? How is it treated?

A
  1. infectious - bacterial, fungal
  2. FB
  3. stenosis

air sac cannulation to buy time to allow the removal of tracheal lesion via endoscopy (size limitations!) or transtracheally

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27
Q

Approach to tracheal FB:

A

transtracheal - push FB cranially from behind and pop it out of the mouth

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28
Q

What is indicative of pneumonia on radiographs? When is this most commonly seen?

A

loss of honeycomb pattern

aspiration pneumonia in gavage fed hatchlings/young birds

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29
Q

What are the 2 most common cause of air sac rupture? How are they treated?

A
  1. trauma - SQ emphysema, create a circular defect in the skin and allow healing via second intention
  2. chronic form - occlusion between cervicocephalic air sac and infraorbital sinus, treatment more difficult
30
Q

What are the 3 most common causes of hepatic lipidosis in birds?

A
  1. high fat diet (seeds!)
  2. sedentary lifestyle
  3. nutritional deficiencies (biotin)

(fatty liver disease)

31
Q

What are some clinical signs of hepatic lipidosis?

A
  • lethargy, anorexia
  • dyspnea
  • biliverdinuria
  • regurgitation
  • PU/PD
  • distended coelom
  • neurologic signs
32
Q

What 4 things are seen on blood work in cases of hepatic lipidosis?

A
  1. lipemic serum/plasma
  2. hepatocellular damage - increased AST, GLDH, BA
  3. liver dysfunction - decreased TP, ALB, UA, coagulation factors
  4. impaired gluconeogenesis - decreased glucose
33
Q

What is seen on imaging in cases of hepatic lipidosis? What gives a definitive diagnosis?

A
  • RADIOGRAPH - enlarged cardiohepatic silhouette
  • U/S - hyperechoic liver with rounded margins +/- ascites

histopathology

34
Q

How is hepatic lipidosis treated?

A
  • aggressive supportive care
  • lactulose - binds ammonia
  • improve nutrition (less fats/seeds, supplement deficiencies)
  • gradual weight loss plan with exercise encouragement
35
Q

What are 3 additional causes of liver disease in birds?

A
  1. HEPATOTOXINS - mycotoxins, heavy metals (Zn, Pb), drugs —> fibrosis, cirrhosis
  2. HEPATITIS - bacterial, fungal, viral
  3. NEOPLASIA
36
Q

What are 6 risk factors that increase chances of developing primary crop stasis?

A
  1. crop infection
  2. dehydration
  3. crop burn - young hand-fed birds
  4. impaction
  5. FB
  6. over-stretching of crop
37
Q

What are 4 risk factors that increase chances of developing secondary crop stasis?

A
  1. systemic infection causing ileus - Eimeria, Polyomavirus, septicemia
  2. toxicity - lead
  3. PDD - Bornavirus
  4. hypothermia
38
Q

What diagnostics are used for crop stasis? What are 4 treatments?

A

crop cytology/culture to determine underlying cause

  1. crop wash - remove all contents
  2. supportive care - heat support, fluids
  3. treat underlying disease
  4. over-stretched - ingluviectomy, crop bra
39
Q

What 4 structures may be seen within cloacal prolapses? What are 4 causes?

A

oviduct, ureters, intestines/coprodeum, cloacal tissues

  1. chronic egg laying
  2. constipation/tenesmus - cloacitis, cloacaliths, enteritis, obstruction
  3. excessive sexual/masturbatory behavior
  4. idiopathic/behavioral
40
Q

How are cloacal prolapses treated?

A
  • ID tissue
  • assess viability
  • if viable - reduce, replace, retain (lateral vent sutures)
  • if non-viable - debride, euthanize
  • determine underlying cause for prevention
41
Q

In what birds is renal disease most common? What clinical signs are associated?

A

geriatric birds - poor husbandry, diet, infection, neoplasia

  • PU/PD
  • depression
  • lameness
  • visceral/articular gout (advanced, poorer prognosis)
42
Q

What diagnostics are used for renal disease?

A
  • BLOOD WORK = persistent hyperuricemia, leukocytosis
  • RADIOGRAPHS = renomegaly, mineralization
  • BIOPSY = histopath
43
Q

How is renal disease treated?

A
  • fluid therapy
  • improve diet
  • colchicine/allopurinol
  • pain medication for gout
44
Q

What birds are most commonly affected by renal neoplasia? What is the most common presentation?

A

Budgerigars —> nephroblastomas, renal adenocarcinomas (poor prognosis)

unilateral leg lameness, weakness, paralysis, or ataxia (sciatic n)

+/- diarrhea
+/- dyspnea
+/- coelomic distention
+/- weight loss

45
Q

What testicular neoplasia is common in birds? What does this result in? What clinical signs are seen?

A

Sertolic cell tumors —> male Budgerigars

hyperestrogenemai

  • hyperostosis
  • cere color change from blue to brown
  • unilateral leg lameness/paresis (close to kidneys, sciatic n)
  • coelomic distention and dyspnea in other species
46
Q

How is phallic prolapse treated?

A

only reproductive, can be amputated

47
Q

What is the difference between egg binding and dystocia?

A

EGG BINDING = failure of egg to pass through oviduct at a normal rate

DYSTOCIA = egg causing obstruction

48
Q

What are the 3 most common dystocia locations?

A
  1. caudal uterus
  2. vagina
  3. junction of vaginal and cloaca
49
Q

What are 8 predisposing factors to dystocia?

A
  1. muscle dysfunction
  2. sedentary lifestyle
  3. obesity
  4. stress
  5. infection
  6. systemic disease
  7. damage to the uterus from a previous egg
  8. malformed egg
50
Q

What clinical signs are associated with egg binding/dystocia? How is it diagnosed?

A

(metabolic derangement)

  • depression, lethargy
  • unwilling to fly
  • wide-based stance
  • straining
  • fluffed, at bottom of enclosure
  • tail bob
  • compressive effects = lameness, circulatory issues

PE and diagnostic imaging

51
Q

How are patients stabilized prior to treating for egg binding/dystocia? What are 6 options for removing the egg?

A

fluids, antibiotics, lubrication of prolapsed tissue

  1. create a warm, humid, quiet environment
  2. calcium supplementation
  3. oxytocin
  4. prostaglandin - Lutylase, PGE2, Propodyl gel, Misoprostol
  5. manual manipulation - ovocentesis
  6. salpingotomy, salpinohysterectomy (if all else fails)
52
Q

What is egg yolk coelomitis? What history and secondary concurrent diseases are associated?

A

inflammation caused by yolk free in the coelom (typically aseptic)

recent reproductive activity

  • ovarian neoplasia
  • oviductal impaction
  • chronic obesity
53
Q

What clinical signs are associated with egg yolk coelomitis?

A
  • depression/lethargy
  • coelomic distention +/- ascites
  • dyspnea - mass effect,
54
Q

What are 5 options for diagnosing egg yolk coelomitis?

A
  1. CBC - variable, hypercholesterolemia
  2. BIOCHEM - variable
  3. COELOMOCENTESIS - cytology + culture
  4. IMAGING - coelomic fluid or hyperostosis on radiographs and U/S
  5. LAPAROSCOPY
55
Q

How is egg yolk coelomitis treated?

A
  • analgesics/anti-inflammatories
  • coelomocentesis
  • fluid therapy
  • antibiotics
  • celiotomy

(common in chickens bred to lay eggs often)

56
Q

What is considered chronic egg laying? In what birds is this most common? What causes it?

A

laying larger than normal clutches or multiple cluthes regardless of the presence of a mate or proper season

Budgies, Cockatiels, Lovebirds

presence of reproductively stimulating toys/objects or mate-like bonding with a particular owner

57
Q

What are 4 aspects to treating chronic egg laying?

A
  1. behavioral/environmental modification - remove reproductively stimulating toys, restrict access to nesting environments/material, decreased photoperiod to 8-10 hours, limit exposure to perceived mate
  2. diet - low fat/reduced calories
  3. hormones - Deslorelin implant, Lupron injections
  4. remove source - salpingohysterectomy
58
Q

What are some common sources of lead?

A
  • weights
  • solder
  • galvanized wire
  • bells
  • shotgun shells
  • costume jewelry
  • batteries
  • lead-based paints
  • mirror backs
59
Q

What are 6 clinical signs associated with lead toxicosis?

A
  1. lethargy/depression
  2. vomiting, diarrhea
  3. PU/PD
  4. anorexia
  5. neurological signs - weakness, ataxia, seizures
  6. hematuria
60
Q

What is the gold standard for diagnosing lead toxicosis? What are 3 other diagnostics that can be used?

A

blood Pb levels

  1. CBC - leukocytosis, hypochromic regeneratice anemia, cytoplasmic ballooning and basophilic stiplling of RBCs in blood smears
  2. BIOCHEM - elevated LDH, AST, BA, and CK
  3. RADIOGRAPHS - radiopaque metal density in GIT
61
Q

What are 3 aspects to treating lead toxicosis?

A
  1. chelation - CaEDTA
  2. supportive therapy - control seizures, antimicrobials, fluids, nutritional support (gavage)
  3. remove lead particles - bulk diets, cathartics, surgical removal
62
Q

What are common sources of zinc?

A
  • hardware cloth in homemade cages
  • clappers in bells of cheap bird toys
  • screws, nuts, bolts
  • US pennies made after 1983
63
Q

What are 6 clinical signs associated with zinc toxicosis?

A
  1. lethargy/depression
  2. vomiting, diarrhea
  3. PU/PD
  4. anorexia
  5. neurologic signs - weakness, ataxia, seizures
  6. acute death

(same as lead)

64
Q

What is the gold standard for diagnosing zinc toxicosis in birds? What are 3 other options? How is it treated?

A

blood Zn levels

  1. CBC - leukocytosis, hypochromic regenerative anemis
  2. BIOCHEM - increased LDH, AST, BA, CK +/- lipase, amylase
  3. RADIOGRAPHS - radiopaque metal density in GIT
65
Q

What toxic principle results in avocado toxicosis in birds? What does it cause? What are 4 clinical signs?

A

R-enantiomer of persin —> present in leaves, fruit, bark, and seeds

myocardial necrosis

  1. dyspnea
  2. SQ edema
  3. pericardial effusion
  4. acute death

(supportive therapy, poor prognosis)

66
Q

Diabetes mellitus is rare in birds. What species typically get it? How is is diagnosed and treated?

A

toucans and smaller psittacines

assess for glucosuria and measure serum insulin

insulin + high fiber diet

67
Q

What is the most common cause of hyperparathyroidism in birds? What are the 2 most common signs?

A

all-seed diets

  1. metabolic bone disease in young growing birds
  2. hypocalcemic associated seizures
68
Q

How is hyperparathyroidism in birds treated?

A
  • correct diet (balance out seeds)
  • supplement Ca and vit D
69
Q

What clinical signs of hypothyroidism are seen in birds? How is it diagnosed and treated?

A
  • obesity
  • alopecia —> lack growing feathers

TSH stim test

thyroxine supplementation

70
Q

In what birds is goiter most common? What is it? How is it treated?

A

Budgies fed a seed-based diet low in iodine

hypertrophy of the thyroid, which causes partial obstruction of the esophagus and trachea —> dyspnea, regurgitation

supplement iodine and fix diet