AV block Flashcards

1
Q

major causes of AV block

A
increased vagal tone
fibrosis and sclerosis of conduction system
IHD
cardiomyopathy and myocarditis
congenital heart disease
familial AV block
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2
Q

other causes of AV block

A
hyperkalemia
inflitrative malignancies, 
neonatal lupus
severe hypo or hyperthyroidism
trauma
degenerative neuromuscular disease
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3
Q

drugs that can cause AV block

A
digitalis 
CaCh blockers
beta blockers
amiodarone
adenosine
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4
Q

other AV block latrogenic

A
cardiac surgery
catheter ablation
transcatherter VSD closure
alcohol spetal ablation for HOCM
TAVr (transcatheter AV replacement)
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5
Q

AV block

A

delay or interruption in transmission of an impulse from atria to ventricles due to an anatomic or fnxal impairment in conduction system

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6
Q

firborsis and sclerosis

A

acounts for 50% of cases of AV block
may be induced by several different conditions
frequently progressive to complete heart block

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7
Q

IHD

A

40% of AV block

conduction disturbances range from 1st degree to complete with both chronic IHD and acute MI

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8
Q

familial AV block

A

autosomal dominant

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9
Q

types

A

1st degree
2nd degree type I
2nd degree type II
3rd degree

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10
Q

2nd degree type I

A

mobitz I

progressive PR interval prolongation followed by non conducted p wave

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11
Q

Ist degree

A

pr interval >.2

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12
Q

2nd degree type II

A

mobitz II

PR interval remains unchanged prior to p wave that fails to conduct to ventricles

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13
Q

3rd degree

A

complete heart block

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14
Q

AF

A
most common cardiac arrhythmia
RR interval no pattern
no distinct P waves
atrial rate 300-600
more common in men
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15
Q

AF risk factors

A

HTN
CH
RF in underdeveloped countries

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16
Q

paroxysmal AF

A

terminated spontaneously or w/intervention w/in 7 days

episodes may recurr

17
Q

persistant AF

A

fails to self-terminate w/in 7 says

episodes often require pharmacological and electrical cardioversion

18
Q

long-standind persistant AF

A

lasted more then 12 months

19
Q

permanent AF

A

persistant with decision made to no longer try to treat

20
Q

low risk AF

A

15-30%
younger males
frequently familial, low risk of thromboembolus

21
Q

recurrent AF

A

90%

asymptomatic recurrent episodes lasting up to 48hrs

22
Q

subclinical AF

A

detected asymptomatic w/o prior diagnosis

many have paroxysmal AF

23
Q

AF Hx

A
palps
syncope
dyspnea
fatigue
precipitating events: exercise, emothional, alcohol
24
Q

AF PE

A

mitral valve disease, especially MS, CHF, findings etc

25
Q

AF echo

A

chamber sizes and fnx, valvular fnx, pulmonary aa pressures

26
Q

AF rate control

A

anti-coagulants and AV blockers

27
Q

rhythem control

A

anti-coagulate and restore NSR via meds, electrical cardioversion
TEE may be used to rule out LA thrombus prior to cardioversion

28
Q

CHADS2

A
CHADS2: 
C-CHF 
H- HTN 
A-Age (76+) 
D- diabetes 
S-stoke/tia (2) 
2+ give anticoagulate
29
Q

CHADS-vasc

A
CHA2DS2vasc: 
C- CHF 
H-HTN 
A-  65-74 (1) 75+ (2) 
D- diabetes 
S-stoke/tia (2),
also add 1 for female
9=15% chance of stoke, 
3= 3% 
3+ give anticoagulate
30
Q

PVC

A
common cadio complaint
present w/palpitatios
syncope is an alarm that it is serious
almost always of no significance
often over emphasized by patient and occasionally by PCP
31
Q

prognosis of PVC

A

dependent on severity of LV fnx, not frequency or complexity of PVC