Autonomics Flashcards
SA Node parasympathetics
decrease HR by releasing ACh to mAChR
SA Node sympathetics
increase HR by releasing NE to B-1 receptors
AV Node parasympathetics
decrease velocity
AV Node sympathetics
increase velocity by releasing NE to B-1
Myocytes parasympathetics
decease contractility by releasing ACh to mAChR
Myocytes sympathetics
increase contractility acting on B-1 receptors
Cerebral vasculature autonomics
ONLY SYMPATHETIC, act on a-1 receptors to contract vascular to protect from high BP
Coronary parasympathetics
Vasodilation
Coronary sympathetics
short lived vasoconstriction on a-1 receptors due to metabolic need
Cutaneous autonomics
a-1 receptors for vasoconstriction
skeletal m. parasympathetics
dilation of blood vessels
skeletal m. sympathetics
a-1 constrict vessels, b-2 dilate vessels
splanchnic vessels
ONLY SYMPATHETICS, T5-L2 released on a-1 to cause constriction
Venous Return
SYMPATHETICS, a-1 causes constriction to increase venous return, a-2 and b-2 causes relaxation to decrease venous return
Pulmonary vessels parasympathetics
NO release to cause vasodilation
Pulmonary vessels sympathetics
a-1 to vasoconstrict, b-1 and b-2 to vasodilate
Upper Airway Parasympathetics
Via CNV and sphenopalatine ganglion, increase secretions and mucus production
Upper Airway Sympathetics
Via CNVII and cervical ganglia, decrease secretions and mucus production
Lower Airway parasympathetics
vagus nerve to contract bronchioles and increase mucus production
Lower Airway sympathetics
T1-T5 on B-2 to cause dilation of bronchioles
Kidney Sympathetics
a-adrenergics on renal a. and afferent and efferent arterioles cause constriction to decrease urine output
juxtaglomerular uses b-1 receptors to initiate RAA to increase sodium and water reabsorption and increase blood volume
