atrial rhythms Flashcards
1
Q
dysrhythmias originated in the atria
A
- wandering atrial pacemaker
- multifocal atrial tachycardia
- premature atrial contractions
- paroxysmal supraventricular tachycardia
- atrial flutter
- atrial fibrillation
2
Q
dysrhythmias originated in the atria: wandering atrial pacemaker
A
- slightly irregular rhythm
- origination varies among the SA node, atrial tissue, and AV junction
- P waves are variable or absent**
- PRI- varies depending on source of impulse
3
Q
wandering atrial pacemaker
A
- etiology- variant of sinus dysrhythmia, which is a natural phenomenon in the very young or old
- may also be caused by ischemic heart disease or atrial dilation
- clinical significance- none but may be precursor to other atrial dysrhythmias
- treatment- typically none required
- rhythms are coming from different foci therefore the rhythm is irregular
4
Q
dysrhythmias originating in the atria: multifocal atrial tachycardia
A
- rate is more than 100
- rhythm is irregular
- pacemaker site is the ectopic sites in the atria
- P waves - organized, nonsinus P waves -> at least 3 forms
- PRI- varies depending on source of impulse
- QRS- variable
- WAP but tachycardic
5
Q
multifocal atrial tachycardia
A
- etiology- often seen in acutely ill patients
- may result from pulmonary disease, metabolic disorders, ischemic heart disease, or recent surgery, COPD
- clinical significance presence of multifocal atrial tachycardia often indicates a serious underlying illness
- treatment- treat the underlying illness
- rhythm is coming from different foci
6
Q
dysrhythmias originated in the atria: premature atrial contractions
A
- rate depends on underlying rhythm
- rhythm usually regular expect for the PAC
- pacemaker site is ectopic sites in atria
- P waves- occurs earlier than expected
- PRI- varies dependent on foci of impulse
- p waves and t waves are combining and colliding
- a lot more off than a dysrhythmia
7
Q
premature atrial contractions
A
- etiology- single electrical impulse originating outside the SA node
- may result from use of caffeine, tobacco, or alcohol, sympathomimetic drugs, ischemic heart disease, hypoxia, or digitalis toxicity, or may be idiopathic
- clinical significance- none, presence of PACs may be a precursor to other atrial dysrhythmias
- treatment- none if asymptomatic
- treat symptomatic patients by administering high flow, high concentration oxygen and establishing IV access
8
Q
dysrhythmias originating in the atria: paroxysmal supraventricular tachycardia
A
- rate is 150-250
- rhythm is regular
- pacemaker site is outside the SA node -> atrial
- P waves are often buried in preceding T wave
- combo of the p and t wave
9
Q
paroxysmal supraventricular tachycardia
A
- etiology- rapid atrial depolarization overrides the SA node
- may be precipitated by stress, overexertion, smoking, caffeine
- clinical significance- may be tolerated well by healthy patients for short peroids
- marked reduction in cardiac output can precipitate angina, hypotension, or congestive heart failure
10
Q
dysrhythmias originating in the atria: atrial fluter
A
- rate is 250-350
- ventricular rate varies
- rhythm is regular
- pacemaker site is outside the SA node (atrial)
- P waves -> F waves are present (flutter wave)
- waves are repeating
11
Q
atrial rate
A
-between the waves before the qrs
12
Q
atrial flutter
A
- etiology- results when the AV node cannot conduct all the impulses
- impulses may be conducted in fixed or variable ratios
- usually associated with organic disease such as congestive heart failure (rarely seen with MI)
- clinical significance- generally well tolerated
- rapid ventricular rates may compromise cardiac output and result in symptoms
- may occur in conjunction with atrial fibrillation
- different ratios
13
Q
dysrhythmias originating in the atria: atrial fibrillation
A
- most common!
- atrial rate 350-750
- ventricular rate varies
- irregularly irregular rhythm
- pacemaker site is outside the SA node (atrial)
- P waves- none discernible
- PRI- none
- QRS- normal
14
Q
atrial fibrillation
A
- etiology- results from multiple ectopic foci
- AV conduction is random and highly variable
- often associated with underlying heart disease
- clinical significance- atria fail to contract effectively, reducing cardiac output
- well tolerated with normal ventricular rates
- high or low ventricular rates can result in cardiac compromise