association cortices Flashcards

add diagrams to notion of where to find each white matter tract type

1
Q

cortex vs lobe

A

cortex = thin outer covering of lobe - esp temporal lobe

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2
Q

insula/ insular cortex

A

(lec recording)
simple function is disgust

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3
Q

what are brodmann areas

A

map of cortex

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4
Q

who first mapped Brodmann areas

A

Korbinian Brodmann - histology variation discovery, observed via microscope - diff strcutures, diff functions

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5
Q

how many Brodmann areas are there

A

52 regions, some are subdivided

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6
Q

brodmann area 17

A

primary visual cortex

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7
Q

brodmann area 37

A

fusiform face area is part of area 37

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8
Q

association cortices ___ the primary cortices

A

are not

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9
Q

Posterior Parietal Cortex function

A

finding loc of sensory info
important for attention
– Especially spatial attention
– Integrates visual, auditory and somatosensory info
Damage results in “neglect” of incoming sensory info

e.g. hemineglect

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10
Q

what brodmann areas make up Posterior Parietal Cortex

A

Brodmann areas 5,7,39,40

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11
Q

what is neglect

A

a sign of posterior Parietal cortex damage

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12
Q

what are the 4 types of neglect

A

sensory neglect

conceptual neglect

hemiasomatognosia

motor neglect

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13
Q

what is sensory neglect

A

incoming sensory info from contralateral hemisphere is ignored

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14
Q

what is conceptual neglect

A

neglect of body and external world in the contralateral hemifield

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15
Q

what is hemiasomatognosia

A

pt denies part of body affected belongs to them

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16
Q

what is motor neglect

A

fewer movements in contralateral space

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17
Q

what is agnosia

A

or loss of knowledge is an inability to
recognize sensory stimuli

caused by inferior or middle temporal cortex damage

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18
Q

3 types of angosia

A

visual agnosia (inferior)

prosopagnosia (inferior)

movement agnosia (middle)

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19
Q

what is visual agnosia

A

pt can see but not identify but can potentially still interact normally with item

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20
Q

what is prosopagnosia

A

face blindness

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21
Q

what is movement agnosia

A

cannot distinguish between moving and stationary

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22
Q

The McGurk Effect

A

subconsious, even if person knows it is different

vision is mostly dominant sense in humans

large variation in how people experience it

reduced in Dyslexia, Autism

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23
Q

prosopagnosia is associated with impaired function of which Brodmann area/areas

A

37

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24
Q

role of frontal cortex

A

selecting and planning an appropriate response

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25
Q

the prefrontal cortex is rostral to

A

primary motor cortex (these are the two parts of the frontal cortex

26
Q

the prefrontal cortex develops when

A

late, 20-30
(is why many mental health conditions develop late)

27
Q

the dorsal part of the frontal cortex regulates

A

thoughts, attention

28
Q

the ventral part of the frontal cortex regulates

29
Q

prefrontal cortex roles - 3

A

restraint
initiative
order

30
Q

restraint

A

role of prefrontal cortex

judgement, foresight, inhibiting inappropriate actions, concentration

31
Q

initiative

A

role of prefrontal cortex

drive
creativity
curiosity
personality
flexibility

32
Q

order

A

role of prefrontal cortex

planning
abstract reasoning
working memory
attention

33
Q

damage to frontal cortex damage can potentially cause (depending on area that is damaged) (12)

A

difficulty planning sequence needed to complete a task (working memory)

loss of spontaneous interactions

loss of flexibility in thought

perseveration - persistence of a single thought or action

inability to focus on the task at hand - erraticness

emotional lability

abulia - passivity, apathy

socially inappropriate behaviour

personality change

difficulty problem solving

epressive aphasia

hemiplegia

34
Q

what is aphasia

A

a condition that makes it hard to speak or write words, even though someone understands what they want to say

35
Q

what is hemiphlegia

A

paralysis that affects only one side of your body

36
Q

where is executive functioning

A

in prefrontal cortex

37
Q

what is executive functioning

A

long term planning
withholding impulsive behaviours
cognitive control

significant in many pathologies - addiction, personality disorders, dementia

38
Q

what are white matter tracts

A

connect the association cortices

myelinated neurons

poorly understood, not easy to see - needs diffusion tensor imaging

39
Q

what are association fibres

A

white matter tracts

connect cortical areas in the same hemisphere

40
Q

example of association fibres

A

superior longitudinal fasciculus

covers lots of area in the brain

41
Q

what are commissural fibres

A

white matter tracts

connect across hemispheres

e.g. corpus callosum - connects two hemispheres

42
Q

what are projection fibres

A

white matter tracts

connect cortex to other brain regions

43
Q

what is language/ what is the process

A

form of sensory info
cortical areas working together:

temporal cortex - what is it aka what am i hearing (after primary auditory cortex processing)

frontal cortex - what am i going to do aka what do i say (sends to motor cortex

muscles in face/throat move to speak back

44
Q

what is dysarthria

A

difficulty moving the muscles of face and tongue that mediate speaking, and therefore speech

musculoskeletal issue

45
Q

what is aphasia

A

difficulty in naming objects , repitition of words is impaired - there are different types

difficulty with language

46
Q

what brodmann areas are primary auditory cortex and where are they found

A

41 and 42

on posterior superior temporal gyrus

47
Q

what is wernicke’s area/ what brodmann area

A

recognition of language/ understanding language

brodmann area 22 - is found next to primary auditory cortex

48
Q

what is wernicke’s aphasia

A

inability/ difficulty to understand language

fluent speech but no sense (if Broca’s area is intact)

little repetition

adequate syntax and grammar

also called fluent, sensory or receptive aphasia

49
Q

cause of wernicke’s aphasia

A

damage to said area

often as result of stroke sometimes alzheimers

50
Q

what is broca’s area/ what brodmann areas

A

creating language

44+45

51
Q

what is broca’s aphasia

A

few problems understanding language (if wernicke’s area is intact)

difficulty contructing own language

repetitive

disordered syntax + grammar + structure of individual words

also called non-fluent, motor, expressive, production aphasia

52
Q

cause of broca’s aphasia

A

damage to broca’s aphasia

often as result of stroke

different branches of the middle cerebral artery

53
Q

for lamguage we are mostly ___ hemisphere dominant

54
Q

wernickes aphasia vs brocas aphasia: regions?

A

wernickes = temporal

brocas = frontal

55
Q

wernickes aphasia vs brocas aphasia: main symptom?

A

wernickes = unable to understand language, fluent speech that does not make sense

brocas area = understands language but cannot structure own, halting speech that somewhat makes sense - self aware

56
Q

wernickes aphasia vs brocas aphasia: repetition?

A

wernickes = little repetition

brocas = repetitive

57
Q

wernickes aphasia vs brocas aphasia: syntax?

A

wernickes = adequate

brocas = disordered

58
Q

wernickes aphasia vs brocas aphasia: grammar?

A

wernickes = adequate

brocas = disordered

59
Q

aphasias affect

A

language - reading, writing, sign language

not convo cues, body language

60
Q

what is arcuate fasiculus

A

white matter tract

connects broca’s and wernicke’s areas

61
Q

arcuate fasiculus is comprised of

A

association fibres (as diff parts of same hemisphere of brain)