Arrhythmias Flashcards

1
Q

What are the different types of arrhythmias

A

Tachy arrhythmias - fast

Brady arrhythmias - slow

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2
Q

Give examples of tachy arrhythmias

A

Atrial fibrillation

Ventricular tachycardia

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3
Q

Give examples of Brady arrhythmias

A

Heart block

Drug induced - Beta blockers, digoxin

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4
Q

What is the most common pulse in bradyarrhythmias

A

50bpm

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5
Q

Describe heart block

A

Slow or no conduction through the AV node to ventricles of impulse from SA node

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6
Q

What can be seen on an ECG of a patient with heart block

A

Prolonged PQ interval

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7
Q

What are the different levels of heart block and how are they classified

A

1st degree, 2nd degree
3rd degree - complete heart block as no impulse is passed to the ventricles
Classified by the length of signal delay

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8
Q

What is the correlation between heart rate and risk of arrhythmia

A

As heart rate increases, the risk of arrhythmia increases

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9
Q

How does ventricular tachycardia relate to ventricular fibrillation

A

VT is a precursor for VF

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10
Q

Describe the ECG of a patient with 3rd degree heart block

A

Regular P intervals but they are not necessarily tied to QRS complexes

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11
Q

Describe how atrial fibrillation appears on an ECG

A

Electrical activity arises throughout the atria rather than just the SA node
Narrow QRS, waves aren’t a fixed interval apart
No p waves as disorganised atrial activity
Irregular pulse - termed irregularly irregular

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12
Q

How does atrial fibrillation affect heart rate

A

Rapid atrial impulses conducted to ventricles giving high heart rate

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13
Q

How does ventricular tachycardia appear on en ECG

A

Broad QRS complex

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14
Q

Why is ventricular tachycardia particularly dangerous

A

Can lead to ventricular fibrillation and death

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15
Q

What abnormality occurs in the cardiac conduction system in AF

A

Many P waves are trying to get through to the ventricle through the AV node, but the AV node is only allowing a small number through

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16
Q

What abnormality in the cardiac conduction system causes VT

A

The time taken to activate the ventricular muscle is longer and as soon as it is finished, it starts again
This leads to electrical activity becoming uncoordinated
No coordinated muscle activity to allow the heart to work as a pump

17
Q

What are cardiac pacemakers used to treat

A

Bradyarrhythmias

18
Q

What is the purpose of a cardiac pacemaker

A

To keep the heart rate at a minimum level

19
Q

How is a cardiac pacemaker fitted

A

Box fitted in chest wall on left side
Wires are passed through the vena cava, through the atria, one placed within the atrial wall another placed in the ventricle to trigger ventricular contraction

20
Q

How do cardiac pacemakers work

A

Stimulate heart action by pacing atria and ventricles in sequence

21
Q

Which dental equipment should be avoided with patients with pacemakers and why

A

Induction scalers as they generate a strong EM field

22
Q

What has a theoretical risk of shutting a cardiac pacemaker down

A

Electrical fields such as an MRI or diathermy

23
Q

When is an implanted defibrillator useful

A

Useful for patients with unstable cardiac rhythms

24
Q

Where is an implanted defibrillator placed

A

Subcutaneously on the right side

25
How does an implanted defibrillator work
Has wires passing to the heart and will look for evidence of VF Where there is, it will use a low energy pulse to coordinate the electrical activity and shock the heart out to VF very rapidly
26
What cardiac rhythms should I be able to recognise
Sinus rhythm Asystole Ventricular Fibrillation
27
In what cardiac arrhythmias do i need to know their clinical presentations
Atrial fibrillation | MI
28
What does the P wave correlate to in sinus rhythm
Atrial depolarisation
29
What does the QRS complex correlate to in sinus rhythm
Ventricular depolarisation
30
What does the T wave correlate to in sinus rhythm
Ventricular repolarisation
31
Why is the P wave in sinus rhythm small
There isn’t much muscle in the atria
32
Why is the QRS complex in sinus rhythm narrow and large
The bundle of His carries the electrical activity to the whole ventricle muscle very rapidly so the muscle contracts in a single unit creating a large electrical signal
33
How is ventricular fibrillation treated
Defibrillation
34
Describe ventricular fibrillation
No cardiac output Electrical heart activity but disorganised Muscle fibres contracting in ventricles at random so there is no emptying of ventricles
35
What can cause ventricular fibrillation
Heart attack Electrocution Long QT syndrome - can be drug induced Wolf-Parkinson-White syndrome
36
Describe asystole
No cardiac output No electrical activity Defibrillation not possible Low chance of survival
37
How can a patient in asystole be treated
Treated with adrenaline to try and produce some electrical activity in the heart so that a defibrillator can be used - tends not to be successful