Arrhythmias Flashcards

1
Q

What is an arrhythmia?

A

-Abnormal heart rhythms
-result from an interruption to the normal electrical signals of contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the four cardiac arrest rhythms ?

A

shockable: ventricular tachycardia, ventricular fibrillation
non shockable: pulseless electrical activity, asystole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How do we treat tachycardia briefly ?

A

Unstable patient:

Consider up to 3 synchronised shocks
Consider an amiodarone infusion

In a stable patient:

Narrow complex (QRS < 0.12s)

Atrial fibrillation – rate control with a beta blocker or diltiazem (calcium channel blocker)
Atrial flutter – control rate with a beta blocker
Supraventricular tachycardias – treat with vagal manoeuvres and adenosine

Broad complex (QRS > 0.12s)

Ventricular tachycardia or unclear – amiodarone infusion
If known SVT with bundle branch block treat as normal SVT
If irregular may be AF variation – seek expert help

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is atrial flutter and how is it treated?

A

the electrical signal goes round and round the atrium instead of going through AV node to ventricles
Treated by controlling rate with beta blockers, radio frequency ablation or anticoags

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is SVT and what are the three main types ?

A
  • electrical signal going back into the atrium from the ventricles through the AV node.
  • “Atrioventricular nodal re-entrant tachycardia” is when the re-entry point is back through the AV node.
    “Atrioventricular re-entrant tachycardia” is when the re-entry point is an accessory pathway (Wolff-Parkinson-White syndrome).
    “Atrial tachycardia” is where the electrical signal originates in the atria somewhere other than the sinoatrial node. this is abnormally generated atrial activity not a signal from the ventricles
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

How do we manage stable SVT?

A
  • continuous ECG monitoring
  • Valsalva - blowing hard into syringe
  • Carotid sinus massage- massage carotid on one side with two fingers
    -Adenosine
    -verapamil (calcium channel blocker)
  • direct current cardioversion if treatment fails
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

How does adenosine work?

A
  • slowing cardiac conduction through AV node to reset it back to sinus rhythm
  • given as rapid bolus - so that there is enough impact to interrupt the AV node/ accessory pathway
  • avoid patient with asthma
  • warn about feeling of dying
    -fast IV bolus in a large proximal canula
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How do you long term manage paroxysmal SVT patients?

A

Paroxysmal SVT describes a situation where SVT reoccurs and remits in the same patient over time.
medication: Medication (beta blockers, calcium channel blockers or amiodarone)
Radiofrequency ablation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is wolf-parkinson white syndrome ?

A
  • extra electrical pathway connecting atria and ventricles
    -treatment- radiofrequency ablation of accessory pathway.
    ECG Changes:

Short PR interval (< 0.12 seconds)
Wide QRS complex (> 0.12 seconds)
“Delta wave” which is a slurred upstroke on the QRS complex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How does radiofrequency ablation occur?

A
  • catheter ablation performed, inserting catheter into femoral veins and feeding it through venous system
    -placed in different heart areas to test signals
    -radiofrequency ablation (heat) is applied to burn the abnormally active area - leaves scar tissue
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is torsades de pointes?

A
  • type of polymorphic ventricular tachycardia
  • has prolonged QT interval
  • which is prolonged repolarisation
  • longer repolarisation can cause spontaneous depolarisation in some areas of heart myocytes -> causing ventricular contraction before proper repolarisation
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the causes of prolonged QT?

A
  • long QT syndrome
  • medication e.g antipsychotics, citalopram, flecainide, sotalol, amiodarone, macrolide antibiotics
  • electrolyte disturbance- hypokalaemia, hypomagnesaemia, hypocalcaemia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the acute management of Torsades de pointes

A

Correct the cause (electrolyte disturbances or medications)
Magnesium infusion (even if they have a normal serum magnesium)
Defibrillation if VT occurs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is the long term management of prolonged QT syndrome?

A

Avoid medications that prolong the QT interval
Correct electrolyte disturbances
Beta blockers (not sotalol)
Pacemaker or implantable defibrillator

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is a ventricular ectopic ?

A
  • premature ventricular beats
  • cause: random electrical discharges outside of atria
    -when they occur so frequently its after every sinus beat then it is Bigeminy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How do we manage ventricular ectopics?

A

Check bloods for anaemia, electrolyte disturbance and thyroid abnormalities
Reassurance and no treatment in otherwise healthy people
Seek expert advice in patients with background heart conditions or other concerning features or findings (e.g. chest pain, syncope, murmur, family history of sudden death)

17
Q

What is brugada syndrome

A
  • sodium channelopathy
  • diagnosis: ST elevation
    -can be caused by fever, meds, electrolyte imbalances and ischaemia