Arrhythmias Flashcards
Tachyarrhythmias
> 100bpm
Supraventricular Tachyarrhythmias
o Sinus Tachycardia o Focal Atrial Tachycardia o Multifocal Atrial Tachycardia o AVRT o AVNRT o Atrial Flutter o Atrial Fibrillation
Ventricular Tachyarrhythmias
o Ventricular Tachycardia § Monomorphic § Polymorphic § Normal QT § Prolonged QT- Torsades de Pointes o Ventricular Fibrillation
Bradyarrhythmia
<60 bpm
Sinus Bradycardia • First Degree Heart Block • Second Degree Heart Block • Mobitz I (Wenckebach) • Mobitz II • Third Degree Heart Block
Sick Sinus Syndrome
SA node Dysfunction caused Sinus Bradycardia
Concomitant Compensatory Supraventricular Tachycardia
o Atrial Fibrillation, Atrial Flutter, SVT
• Tachycardia-Bradycardia Syndrome
• Usually require combination therapy
o Beta-blocker or calcium channel
blocker for SVT
o Permanent Pacemaker for
Bradycardia
How does increased SNS activity manifest as in Tachyarrhythmia
o Pain o Exercise o Hypovolemia o Hypoxia o Pulmonary Embolism o Sympathomimetics
what are the effects of increased metabolic activity during Tachyarrhythmia
Fever
Hyperthyroidism
What are the effects of Decreased Automaticity? Increased vagal tone (PSNS) (Bradyarrhythmia)
Increased vagal tone (PSNS)
Sleeping
Athletes
Inferior wall MI (Right Cor. Artery occlusion)
What are the effects of Decreased Automaticity? Slow AV conduction
beta blockers
Ca blockers
digoxin
What are the effects of Decreased Automaticity? Dec Metabolism
hypothermia
Hypothyroidism (myxedema coma)
What are the effects of Decreased Automaticity? Electrolytes
Hyperkalemia
What are the effects of Decreased Automaticity? inc. Intracranial pressure- causes herniation
Cushing’s Triad
Dec. HR, HTN, Irregular RR
SNS tone in Increased Automaticity (Sinus Tachycardia)
Hypovolemia
Hypoxia- Low RBCs, Lung disease, Pul. Emb
Drugs that can increase automaticity?
Sympathomimetics
Psychological factors that increase automaticity
Pain/Anxiety
Increased metabolic activity that causes an increase in Automaticity
Fever
Hyperthyroid
What causes Delayed After Depolarization
o Infarction o Inflammation (myocarditis) o Stretched myocardium (Cardiomyopathy, Mitral Regurgitation) o Hypoxia o Catecholamine excess (Increased SNS)
What causes Early After Depolarization
Hypokalemia, calcemia, magnesemia
Drugs that cause EAD
A- anti-arrhytmics-Type 1a,1c 3 B- Antibiotics- Macrolides- micins C- Antipsychotics- haloperidol D- Antidepressants- TCA and SSRi E- Anti-emetics- Ondansetron
EAD’s EGCs
usually causes Torsade’s- Polymorphic VT (long QT)
DAD’s ECG
Multifocal AT, Focal Atrial
Tachycardia, VTach (normal QT)
Re-entrant Circuit Tachyarrhythmias
AVNRT/AVRT, AFlut/Afib, Vtach and VFib
AVRT (Atrioventricular re-entrant Tachycardia)
Due to accessory pathway- bidirectional between atria and ventricles § Bundle of Kent • WPW syndrome § Bundle of James • LGL (Long-Ganong-Levine) syndrome
Orthodromic AVRT- Down AV node, up Bundle of KEnt
§ More common type § Conduction moves through AV node-ventricles-accessory pathway-atria-AV node § Narrow Complex WPW § Not as dangerous Normal conduction pathway