Approach to the patient with Suspected Dysrhythmia Flashcards

1
Q

What are the 4 mechanisms of dysrhythmias

A

Disorders of impulse formation or automaticity
Abnormalities of impulse conduction
Reentry
Triggered activity

Premature / Ectopic / Escape beats or rhythms
Bradyarrhythmias
Tachyarrhythmias

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2
Q

what can lead to arrhythmias?

A

Genetic abnormalities
Acquired structural heart disease
Electrolyte abnormalities
Hormonal imbalances (thyrotoxicosis, hypercatecholaminergic states)
Hypoxia
Drug effects (such as QT interval prolongation or changes in automaticity, conduction, and refractoriness)
Myocardial ischemia

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3
Q

What are ways that palpations can present

A

Palpitations are defined as an unpleasant awareness of the beating of the heart

Forceful
Rapid
Irregular

can ask patients if they can beat

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4
Q

What are most of palpitations?

A

benign :)

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5
Q

What are the 4 goals of evaluation of palpitation

A
  1. an arrhythmia that is minor and transient
  2. significant cardiovascular disease
  3. a cardiac manifestation of a systemic disease, such as thyrotoxicosis
  4. a benign somatic symptom that is amplified by underlying psychosocial characteristics of the patient

should get TSH because most other systemic diseases do not present with palpitations and it is pretty easy to r/o

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6
Q

What are historical risk factors that are red flags for palpitations?

A

Family history of young onset of cardiac
Passing out (history of syncope)
sudden death
History of MI

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7
Q

What are some red flags of PE findings of palpitations?

A

Structural heart disease such as dilated or hypertrophic cardiomyopathies
Valvular disease (stenotic or regurgitant)

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8
Q

What are red flags of ECG findings for palpitations?

A

Prolonged QTC
Bradycardia
Second- or third-degree heart block
Sustained ventricular arrhythmias

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9
Q

What are some common descriptions of palpatations

A

“Flip-flopping” (or “stop and start” or “skipped beats”)
Often a result of premature contractions
Rapid “fluttering in the chest”
Is the fluttering regular (SVT, sinus tach, VT)
Or irregular (afib)
“Pounding in the neck”
Commonly occur with afib and aflutter, as well as PACs

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10
Q

What symptoms do you check for, for atrial flutter?

A

Age at first episode
The rate, duration, and degree of regularity of the heart beat
The circumstances associated with onset and termination
Abrupt onset and termination vs gradual
Setting in which they occur

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11
Q

SVT presentation

A

Abrupt change from normal to like 180

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12
Q

What are some associated symptoms of palpitations

A

Chest pain, shortness of breath
Dizziness, near syncope or syncope

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13
Q

What is a concerning onset of arrhythmias?

A

associated with exercise or syncope

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14
Q

What are some red fag social history for palpitations

A

alcohol
illicit drug use

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15
Q

What medications can cause palpitations?

A

Stimulants, OTC cold medicine
AADs

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16
Q

What diagnostic studies do you order for palpitations?

A

ALL patients get EKGs (even if you are not symptomatic)

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17
Q

Ambulatory Monitoring Devices

A

Holter monitor
Event monitors
Real-time monitors
Patch recorders
Implantable loop recorders
When should each be used? Benefits of each? Concerns / costs?

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18
Q

Holter monitor

A

24 hour - it will be normal

monitor every single heart beat over 24-48 hour period, patient gets diary so you can correlate symptoms with palpitations

longer monitoring is better

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19
Q

Patch, real-time, and event monitor

A

Monitor for longer

patches come with a phone and record if they are having symptoms (and record time) and then say what their symptoms were when there was an episode

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20
Q

Event monitors

A

Only record events or if a patient records it

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21
Q

real-time monitors

A

Record the whole time

more useful

22
Q

If they have daily episodes, what is a good monitor

A

holter monitor (would see within that 24 hour period)

23
Q

If they only have epidoses every now and again, what do you use?

A

Event monitors
Real-time monitors
Patch recorders

24
Q

Loop recorder

A

clean skin
put over drape
make an incision under the skin to the left of the sternum
has an angled scapule that flattens out (so it does not go too deep)
Have a syringe

A couple syncope episodes that was not caught with other monitors

25
Q

Are electrophyisiology pros and cons

A

Pros: measures pathways, can induce rhythms
Cons: very invasive

26
Q

When is a good time to get an EKG exercise testing?

A

If their arrythmia is exerciseinduced

controlled environment

27
Q

Use of echo for palpitations?

A

Pretty much every patient

See if their are structural/valvular issues

28
Q

What labs should you always order for palpitations

A

Thyroid
Electrolytes

29
Q

Goal of dysrhythmia management

A

Goal #1: prevent sudden death
Goal #2: reduce symptoms improve QOL
Goal #3: reduce hospitalization

Weigh benefits/harms based on patient

30
Q

What are the management for dysrhythmias (read don’t memorize)

A

Antiarrhythmic drugs
Cardioversion
Electrical or chemical
Synchronized or unsynchronized (defibrillation)
Catheter ablation
Pacemaker
Temporary – transcutaneous or transvenous
Permanent/implanted
Cardioverter-defibrillator
Portable AED
Wearable – Life Vest
Permanent/implanted

31
Q

Cardioversion use

A

Patches placed rather than paddles

only for tachyarrythmias (SVT, AF, VT, VF)

Atrial fibrillation / flutter
SVT
Ventricular tachycardia

32
Q

What is the range of Jouels for cardioversion?

A

50-360 J

33
Q

Preparation of cardioversion?

A

Pads placed or conduction gel applied to paddles
ALL staff/personnel must be clear of touching patient
Requires informed consent, except in unstable emergency / cardiac arrest
Requires sedation, except in unconscious unstable patient

34
Q

Risk/complications of cardioversion

A

VT/VF due to general anesthesia or lack of synchronization between the DC shock and the QRS complex
Thromboembolus due to insufficient anticoagulant therapy
Arrhythmias: non-sustained VT, atrial arrhythmia, heart block, bradycardia, transient left bundle branch block
Myocardial necrosis, myocardial dysfunction (every shock causes death of tissue)
Transient hypotension
Pulmonary edema (pressure no long moves into the heart)
Skin burn

35
Q

catheter ablation

A

laser or cryo (freezing) can map a PVC cell and burn it

invasive
pricy
First-line for some arrhythmias
need sedation because it hurts and is long

36
Q

What conditions are catheter ablation first-line

A

AV nodal reentrant tachycardia
Paroxysmal atrial tachycardia
Atrial flutter

37
Q

How do we get to the left atrium?

A

Through the right, which is why a catheter ablation is CI in people with a congental defect

38
Q

Complications of catheter ablation

A

Generally fairly safe

Major vascular damage during catheter insertion occurs in < 2% of patients.
There is a low incidence of perforation of the myocardial wall resulting in pericardial tamponade.
Sufficient damage to the AV node to require permanent cardiac pacing occurs in < 1% of patients.
A rare but potentially fatal complication after catheter ablation of atrial fibrillation is the development of an atrio-esophageal fistula resulting from ablation on the posterior wall of the LA just overlying the esophagus.

39
Q

Pacemakers are indicated for

A

Bradyarrythmias

can ONLY speed up the heart - kicks in with a small impulse if <60 BPM

40
Q

Implantable cardioverter-defibrillator (ICD)

A

are indicated to prevent SCD, and ALL include pacemakers

lower power and internal

41
Q

Indications for pacemakers

A

Symptomatic bradycardia
High-grade AV Block
Sinus pauses or afib pauses with symptoms
No reversible causes identified – this is important (treat the reversible cause first - whether it is meds or something else)

42
Q

ICD Indications

A

Primary prevention of Sudden Cardiac Arrest (VT/VF)
EF < 35% or other at-risk population (Long QT, Brugada, Hypertrophic Cardiomyopathy - no waiting period)
Secondary prevention of Sudden Cardiac Arrest (VT/VF)

40 days after MI
90 days if others

43
Q

where do you put an ICD

A

non-dominant side that is less active (so that there is less tearing)

normally on left side because most people are right handed

44
Q

What are the different types of ICD

A

All devices include the can and leads (wires)

45
Q

What lead is always included

A

Right ventricular lead

depending on the indication, it can be:

Single chamber pacemaker
RV lead only
Dual Chamber ICD
RA and RV leads
Bi-Ventricular ICD (CRT-D)
RV and LV leads +/- RA lead

46
Q

How to know that leads are placed correctly?

A

Fish hook in right atrium

heal arch toes of apex of RV

47
Q

How to know if there is a pacemaker?

A

Tiny thickness on end

Shock coil is larger and is seen in defibrillator

48
Q

Patient education for shocks?

A

Pacing should not be detectable by the patient
ICD shocks are substantial!!!
The devices are METAL and are affected by MAGNETS
Therefore, patients will set off metal detectors
MRIs may be contraindicated (for the most part – newer devices are MRI-safe)
Pacing is detectable on EKG and Telemetry, marked by “pacer spikes”
Patients with a pacemaker or ICD should be following with a cardiologist, electrophysiologist, or the surgeon who implanted the device
Home wireless monitoring has improved safety

if you get out of rythym, you have a 95% chance of dying with out it, if you have it, you have a 95% chance of surviving

49
Q

What do you see on pacemaker EKG

A

Pacemaker spikes followed by wide QRS complex

If it does not cause any EKG changes and it’s rhythm is independent to the patient’s EKG (random pacemaker spikes), then it is not working or is placed incorrectly

50
Q

What should a pacemaker spike cause

A

depolarization of atria or ventricles

otherwise it is not working

def don’t wanna see a spike at the T wave as it can cause torsades

51
Q
A