Appendicitis & Diverticulitis Flashcards

1
Q

What is an Acute Abdomen?

A

A condition that demands urgent attention and treatment in which a patient presents with sudden onset of peritoneal signs.

May be caused by infection, inflammation, vascular occlusion, or obstruction.

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2
Q

What are the peritoneal signs?

A

Rebound tenderness, involuntary guarding, tenderness to percussion.

These signs indicate irritation of the peritoneum.

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3
Q

What are the characteristics of visceral abdominal pain?

A

Slow onset, vague, dull, poorly localized, usually felt midline due to bilateral sensory supply.

Causes include distention of hollow organs, inflammation, ischemia, and malignant infiltration of sensory nerves.

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4
Q

What defines parietal abdominal pain?

A

Acute, sharp, localized to a specific region due to direct irritation of the parietal peritoneum.

Causes include pus, bile, urine, or GI secretions.

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5
Q

What is referred pain?

A

Pain perceived at a different, distant site from the involved organ due to shared innervation from the same spinal level.

It appears with intense stimulation of the affected viscera.

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6
Q

What key history and physical exam factors are important for acute abdomen diagnosis?

A
  • Where is the pain
  • Does it radiate anywhere
  • What does it feel like
  • When did it start
  • What were you doing when the pain began
  • Have you had this type of pain before
  • Associated symptoms
  • Age of Patient
  • Gender
  • Medical Conditions and Medications
  • Previous Surgeries
  • Family and Social History
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7
Q

What is Murphy’s Sign indicative of?

A

Acute cholecystitis.

It is a special test used during the physical examination.

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8
Q

What does Kehr’s Sign indicate?

A

Splenic rupture or ectopic pregnancy rupture.

It is characterized by referred pain to the left shoulder.

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9
Q

What is the significance of McBurney’s Sign?

A

It is associated with appendicitis.

It is located one-third of the way from the ASIS to the umbilicus.

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10
Q

What is the embryological significance of week 6 in intestinal development?

A

Midgut herniates out of the abdominal cavity.

This is a critical stage in the development of the intestines.

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11
Q

What is duodenal atresia?

A

Failure of the bowel to recanalize during gestation.

Clinical presentation includes bilious vomiting and abdominal distention.

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12
Q

What imaging sign is associated with duodenal atresia?

A

Double bubble sign.

It shows dilation of the stomach and proximal duodenum with absent gas distally.

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13
Q

What is hypertrophic pyloric stenosis?

A

Gastric outlet obstruction due to hypertrophy of the pylorus.

It presents with nonbilious projectile vomiting in infants.

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14
Q

What findings are associated with intussusception?

A
  • Crampy abdominal pain
  • Intermittent vomiting
  • Currant jelly stool

It is a leading cause of intestinal obstruction in young children.

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15
Q

What is a common complication of diverticulitis?

A

Perforation leading to peritonitis.

Other complications include abscess, strictures, and fistulas.

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16
Q

What is the primary treatment for uncomplicated appendicitis?

A

Antibiotics with possible delayed appendectomy.

Recent studies suggest non-operative management.

17
Q

What is the gold standard for diagnosing appendicitis?

A

CT Scan.

It shows thickened, inflamed appendix with fat stranding.

18
Q

What is the pathophysiology of peptic ulcer disease?

A

Mucosal injury due to increased luminal acid and/or weakened mucosal defense.

Common causes include H. pylori infection and NSAID use.

19
Q

What is the typical age range for peak incidence of peptic ulcer disease?

A

55-65 years old.

Risk factors include H. pylori infection and NSAID use.

20
Q

What distinguishes gastric ulcers from duodenal ulcers?

A

Gastric ulcers are worse with food and lead to weight loss; duodenal ulcers are better with food and often lead to weight gain.

Gastric ulcers are associated with increased risk of carcinoma.

21
Q

What is the function of the appendix?

A

Immunologic organ that mainly secretes IgA.

It is located at the terminal cecum.

22
Q

What are the common causes of diverticulosis?

A
  • Low fiber diet
  • Disordered motility
  • Alterations in colonic structure
23
Q

What is the clinical presentation of diverticulitis?

A
  • Abdominal pain (typically LLQ)
  • Fever
  • Leukocytosis
  • Nausea and vomiting
24
Q

What imaging modality is standard for diagnosing diverticulitis?

A

CT Scan.

It shows localized bowel wall thickening, increased soft tissue density in pericolonic fat, and colonic diverticula.

25
Q

What is the classic sign of appendicitis on physical examination?

A

Tenderness and guarding upon palpation in the RLQ, classically over McBurney’s point.

26
Q

What is diverticulitis characterized by?

A

Presence of pericolonic inflammation

Diverticulitis can present with various complications such as abscess, fistula, obstruction, or free perforation.

27
Q

What is the management for outpatient diverticulitis?

A

Antibiotics, short-term diet modification

Outpatient management focuses on treating mild cases of diverticulitis without complications.

28
Q

What are the inpatient management strategies for diverticulitis?

A

Bowel rest, IV antibiotics, pain control, serial abdominal exams

Inpatient management is necessary for complicated cases of diverticulitis or when the patient is unable to tolerate oral intake.

29
Q

What must be done 4-6 weeks after a suspected episode of acute diverticulitis?

A

Colonoscopy to rule out malignancy

This procedure is essential to ensure that there are no underlying serious conditions such as cancer.

30
Q

What is the difference between uncomplicated and complicated diverticulitis?

A

Uncomplicated: no abscess, fistula, obstruction, or perforation; Complicated: presence of abscess, fistula, obstruction, or free perforation

Complicated diverticulitis often requires more intensive management, including possible surgical intervention.

31
Q

True or False: Antibiotics are used in both outpatient and inpatient management of diverticulitis.

A

True

Antibiotics are a key component of treatment for both uncomplicated and complicated diverticulitis.