APACIBLE 04 Flashcards

1
Q

the presence of atmospheric air in the pleural space; its presence causes the alveoli to
collapse (atelectasis)

A

pneumothorax

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2
Q

pneumothorax assessment

A

A P C S

absent or decreased breath sounds

percussion may not be revealing

chest may be hyper-resonant

shift of the apical pulse away from the site of pneumothorax

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3
Q

Pneumothorax treatment

A

CO T W

Chest radiography will show a darkened area of the air-filled pleural space

Oxygen therapy to relieve respiratory distress

Thoracotomy catheter or needle may be placed through the chest wall into the pleural space to
remove accumulated air.

Water-seal drainage with children

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4
Q

A chronic pulmonary involvement that occurs in 10% to 40% of infants who are treated for
acute respiratory distress in the first days of life.

The condition most often occurs in infants who received
mechanical ventilation for respiratory distress syndrome at birth.

A

Bronchopulmonary Dysplasia

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5
Q

The condition is thought to occur from a combination of surfactant deficiency (decreased from lung trauma), barotrauma (lung damage from ventilator pressure), oxygen toxicity (from high levels needed to counteract the original respiratory
distress), and continuing inflammation.

A

Bronchopulmonary Dysplasia

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6
Q

Causes of Bronchopulmonary Dysplasia (occurs from a combination of)

A

Surfactant deficiency
Barotrauma
Oxygen toxicity
Continuing inflammation

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7
Q

Bronchopulmonary Dysplasia Assessment

A

T A T O R N

Tachycardia

Auscultation reveals decreased air movement

Tachypnea

Oxygen dependence

Retractions

Nasal flaring

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8
Q

Bronchopulmonary Dysplasia treatment

A

A I

Administration of a corticosteroid to reduce inflammation and a bronchodilator by nebulizer
greatly improves respiratory function

Infants need to be monitored carefully for nutrition and fluid intake

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9
Q

Caused by bacterium Mycobacterium tuberculosis, is a highly contagious pulmonary disease
that affects children worldwide

A

Tuberculosis

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10
Q

The mode of transmission is inhalation of infected droplets. The
incubation period is 2 to 10 weeks.

A

Tuberculosis

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11
Q

Tuberculosis assessment

A

P S - L A W N

Pulmonary inflammation occurs

Slight cough develops

As the disease progresses,
- low grade fever
- anorexia
- weight loss
- night sweats

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12
Q

TB Treatment

A

T N M S A

tuberculin test as part of basic preventive health care at 9 to 12 months of age

negative tuberculin result before administering this vaccine.

Mantoux test, also called a purified protein derivative (PPD) test, 5 units of protein derivative vaccine is injected intradermally, usually on the left lower arm.

sputum may be analyzed, to confirm a diagnosis of active disease. Done for 3 consecutive days

Anti-tuberculosis drugs include Isoniazid, Rifampin, Pyrazinamide, and ethambutol.

Children should have periodic chest radiographs for the rest of their life to make certain their
disease does not become active again later in life.

The Bacille Calmette-Guérin (BCG) vaccine is available against tuberculosis, but it is not used
routinely in the United States.

A skin test will be strongly positive after effective BCG vaccination.

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13
Q

may produce peripheral neurologic symptoms if pyridoxine (vitamin B6) is not
administered concurrently. Rifampin is a secondary drug often used in combination with this drug

A

INH

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14
Q

used with older children. It must be used with caution with infants because one
side effect is optic neuritis

A

Ethambutol

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15
Q

An inherited disease of the secretory glands.

This is characterized by thick mucus secretions, particularly in the pancreas and the lungs, as well as electrolyte abnormalities in sweat gland secretions.

A

Cystic Fibrosis

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16
Q

It occurs most commonly in Caucasian children and rarely in black or Asian children.

A

Cystic Fibrosis

17
Q

The disorder is inherited as an autosomal recessive trait. It occurs in approximately 1 in 2500
live births.

A

Cystic Fibrosis

18
Q

This means that to have this, a person must have a nutation in both copies of the CFTR gene on
each cell. People with CF inherit one mutated copy of the gene from each parent, who is referred to as a carrier.

A

Cystic Fibrosis

19
Q

Pancreas involvement in CF

A

Without pancreatic enzymes in the duodenum, children cannot digest fat, protein, and some sugars. The
child’s stools become large, bulky, and greasy (steatorrhea).

20
Q

child’s stools become large, bulky, and greasy

A

steatorrhea

21
Q

Cystic Fibrosis assessment
Pancreatic Involvement

A

P S S L

Protuberant abdomen due to bulk of feces in the intestine

Stool is extremely foul in odor

Shows sign of malnutrition: emanciated extremities and loose, flabby folds of skin on their buttocks

Low levels of fat-soluble vitamins particularly A D E

22
Q

The anteroposterior diameter of the chest becomes enlarged in CF. Thickened mucus pools in
bronchioles. Pockets of infection then begin in these secretions.

The organisms most frequently cultured are Staphylococcus aureus, Pseudomonas aeruginosa, and H. influenzae.

Atelectasis occurs as a result
of absorption of air from alveoli behind blocked bronchioles.

A

Cystic Fibrosis
Lung involvement

23
Q

Lung Involvement
CF assessment

A

C

The child’s fingers become clubbed because of the inadequate peripheral tissue perfusion

24
Q

Sweat gland involvement
CF

A

Although sweat glands are not changed in structure, the level of chloride to sodium in perspiration is increased 2 to 5 times above normal, resulting in “saltry” perspiration.

25
CF signs and symptoms Swear glands involvement
C A A P P R C Chromosomal abnormality is evident Abnormal concentration of chlorine in perspiration Absence of pancreatic enzymes in the duodenum Presence of immune-reactive trypsinogen in the blood secondary to pancreatic obstruction and pulmonary involvement Poor weight gain within 7 to 10 days after birth and perhaps not until 4 to 6 weeks of age Respiratory infections develop at 4 to 6 months of age. Even at this early stage of the disease, wheezing and rhonchi may be heard on chest auscultation. Clubbing of the fingers may already be apparent.
26
CF sweat gland involvement Assessment
S D S P Sweat testing Duodenal analysis Stool analysis Pulmonary testing
27
is a time-honored method for detecting the abnormal concentrations in sweat in children with CF. Sweat is collected by placing a filter paper on the skin and analyzed for sodium chloride content.
Sweat testing
28
Analysis of duodenal secretions for detection of pancreatic enzymes reveals the extent of the pancreatic involvement. This is done by passing a nasogastric tube into the duodenum and then aspirating secretions for analysis. The secretions removed from the duodenum are sent to the laboratory for analysis of trypsin content, the easiest pancreatic enzyme to assay. Keep the secretions cold during transport. They should be analyzed immediately for accurate results.
Doudenal analysis
29
Stool may be collected and analyzed for fat content and lack of trypsin, although description of the large greasy appearance may be all that is necessary.
Stool analysis
30
A chest radiograph generally con- firms the extent of the pulmonary involvement (pockets of emphysema and perhaps beginning pneumonia infiltration are present). Pulmonary function tests may be done to determine if atelectasis and emphysema are present.
Pulmonary testing
31