Anesthesia Week 2 Ventilator-Diana Flashcards
Flow
Volume/Time
DELIVERY OF VENTILATION IS CONTROLLED BY FLOW
Resistance
Change in pressure/Flow
total driving pressure=gradient between the outside and inside pressure EX. inspiratory pressure and alveolar pressure
Total sum resistance
patient circuit, tubes conencting to the patient to the ventilator, to the bronchi, chest wall, the lung parenchyma, the distended abdomen
Volume
FlowXTime
Flow rate is fixed and where the volume control is done by adjusting the time
Pressure
FlowXResistance
Alpha angle in ETCO2
Increased alpha angle signifies an expiratory airflow obstruction such as COPD, bronchospasm, or a kinked entotracheal tube
Beta angle in ETCO2
Fresh gas does not contain carbon dioxide, so when a patient inspires, the capnograph should immediately return to zero. Causes: absorbent exhaustion.
What are the methods of CO2 analysis?
Mainstream attached to the ETT and Sidestream located outside the airway and it requires a water trap
Low ETCO2
Hyperventilation: light anesthesia, metabolic acidosis
Decreased CO2 production: hypothermia
Increased alveolar dead space: hypotension, pulmonary embolism
Elevated ETCO2
Increased CO2 production: MH, sepsis, fever, hyperthyroidism
Decreased alveolar ventilation: hypoventilation, narcotis
Rebreathing
Causes are exhausted CO2 absorbent, incompetent expiratory valve, hole in the inner tube of a Bain system, inadequate FGF with Mapleson circuit.
Causes of increased ETCO2
Increased CO2 production and delivery to the lungs
Increased BMR, malignant hyperthermia, thyrotoxicosis, fever, sepsis, seizures, laparoscopy, tourniquete or vascular clamp removal, sodium bicarbonate administration, pain, shivering, anxiety, increased muscle tone, medication side effect
Causes of decreased end tidal CO2
Decreased CO2 producrtion and delivery to the lungs
Decreased BMR, increased anesthetic depth, hypothermia, decreased pulmonary blood flow, decreased cardiac output, hypotension, pulmonary embolus, V/Q mistmatch, medication side effect
Increased End Tidal CO2
Decreased alveolar ventilation
Hypoventilation, CNS depression, residual neuromuscular blockade, COPD, high spinal anesthesia, neuromuscular disease, metabolic alkalosis, medication side effect
Decreased End Tidal CO2-
Increased Alveolar Ventilation
Hyperventilation, inadequate anesthesia, metabolic acidosis, medication side effect