Anesthesia Week 2 Ventilator-Diana Flashcards

1
Q

Flow

A

Volume/Time

DELIVERY OF VENTILATION IS CONTROLLED BY FLOW

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2
Q

Resistance

A

Change in pressure/Flow

total driving pressure=gradient between the outside and inside pressure EX. inspiratory pressure and alveolar pressure

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3
Q

Total sum resistance

A

patient circuit, tubes conencting to the patient to the ventilator, to the bronchi, chest wall, the lung parenchyma, the distended abdomen

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4
Q

Volume

A

FlowXTime

Flow rate is fixed and where the volume control is done by adjusting the time

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5
Q

Pressure

A

FlowXResistance

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6
Q

Alpha angle in ETCO2

A

Increased alpha angle signifies an expiratory airflow obstruction such as COPD, bronchospasm, or a kinked entotracheal tube

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7
Q

Beta angle in ETCO2

A

Fresh gas does not contain carbon dioxide, so when a patient inspires, the capnograph should immediately return to zero. Causes: absorbent exhaustion.

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8
Q

What are the methods of CO2 analysis?

A

Mainstream attached to the ETT and Sidestream located outside the airway and it requires a water trap

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9
Q

Low ETCO2

A

Hyperventilation: light anesthesia, metabolic acidosis
Decreased CO2 production: hypothermia
Increased alveolar dead space: hypotension, pulmonary embolism

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10
Q

Elevated ETCO2

A

Increased CO2 production: MH, sepsis, fever, hyperthyroidism

Decreased alveolar ventilation: hypoventilation, narcotis

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11
Q

Rebreathing

A

Causes are exhausted CO2 absorbent, incompetent expiratory valve, hole in the inner tube of a Bain system, inadequate FGF with Mapleson circuit.

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12
Q

Causes of increased ETCO2

Increased CO2 production and delivery to the lungs

A

Increased BMR, malignant hyperthermia, thyrotoxicosis, fever, sepsis, seizures, laparoscopy, tourniquete or vascular clamp removal, sodium bicarbonate administration, pain, shivering, anxiety, increased muscle tone, medication side effect

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13
Q

Causes of decreased end tidal CO2

Decreased CO2 producrtion and delivery to the lungs

A

Decreased BMR, increased anesthetic depth, hypothermia, decreased pulmonary blood flow, decreased cardiac output, hypotension, pulmonary embolus, V/Q mistmatch, medication side effect

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14
Q

Increased End Tidal CO2

Decreased alveolar ventilation

A

Hypoventilation, CNS depression, residual neuromuscular blockade, COPD, high spinal anesthesia, neuromuscular disease, metabolic alkalosis, medication side effect

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15
Q

Decreased End Tidal CO2-

Increased Alveolar Ventilation

A

Hyperventilation, inadequate anesthesia, metabolic acidosis, medication side effect

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16
Q

Increased End Tidal CO2

Equipment malfunction

A

Rebreathing, CO2 absorbent exhaustion, unidirectional valve malfunction, leak in breathing circuit, increased apparatus dead space

17
Q

Decreased End Tidal CO2

Equipment Malfunction

A

Ventilator disconnect, esophageal intubation, poor seal with ETT or LMA, sample line leak, airway obstruction, apnea

18
Q

SPO2 calculation

A

SpO2 90%= PaO2 60 mmHg
SpO2 80%=PaO2 50 mmHg
SpO2 70%=PaO2 40 mmHg

19
Q

Left shift= Increased affinity

A
Decreased Temperature
Decreased 2,3 DPG
Decreased CO2
Decreased H+
Increased PH
HgbMet
HgbCO
Hgb fetal
20
Q

Right shift=decreased affinity

A
Increased temperature
Increased 2, 3 DPG
Increased CO2
Increased H+
Decreased PH