Anatomy Flashcards

1
Q

What is Humphrys ligament

A

Attaches from PCL to lateral meniscus

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2
Q

What tendon passes between LCL and lateral meniscus

A

Popliteal tendon

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3
Q

What type of cartilage lines the knee joint?

A

Hyaline for the synovial bi-condylar joint

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4
Q

Classify the 3 types of synovial joints

A

Simple:
- one pair of articulating surfaces. Usually concave and convex

Compound:
- more than one articulating pair in single capsule, neither will articulate with surface of the other

Complex:
- had an articular disc

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5
Q

Differentiate between closed and open pack position and their clinical significance.

A

Closed:
- most stable, not for mobilization, position for lig testing, # position

Open:
- position for acute sprains, splints mobilizations/ distraction

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6
Q

Special questions for C-spine Ax

A

5D’s, 3N’s, RA (CV lig instability) URTI, cord signs (neck position and signs and symptoms)

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7
Q

Special questions for T-spine Ax

A
  • lifting, eating, breathing, cough/sneezing
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8
Q

Special questions for L-spine

A

B&B, saddle paresthesia, cough/sneeze

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9
Q

Mandatory Red Flag questions

A
  • bilateral/quadrilateral paresthesia (SC syndrome)
  • hemiparesis (CNS dysfunction)
  • hemiparesis + facial paresthesia ( brain stem in fact)
  • dizzy, vertigo, drop attack (vertebrobasilar)
  • loss of consciousness
  • changes in hearing vision and speech (brain stem infarct)
  • changes in bowel and bladder (cause equina)
  • traumatic MOI (verte artery, Cv ligs)
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10
Q

What is Hoffmans reflex?

A
  • Flick of 3rd finger produces opposition of index and thumb
  • positive = CST damage
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11
Q

What is the Openheimer test

A
  • noxious stimulus to tibial crest

- positive = damage to pyramidal tract

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12
Q

What is fibromyalgia?

A

Non articular rheumatism with diffuse MSK aches/stiffness w/ exaggerated tender point
- traps, pec, glutes, greater trochanter, medial fat pad of knee

Tx:
- sleep, exercise, anti-depressants, education

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13
Q

What is myofascial pain syndrome

A

On palpating causes referred pain and autonomic phenomena

  • Stiffness/pain due to taut bands not allowing for stretch, usually unilateral, may be weak

Tx: Heat, ROM, ischemic pressure, IMS

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14
Q

What is polymyositis?

A
  • diffuse inflammation of striated muscle, primarily of shoulder and pelvis
  • Variable pattern of onset of proximal muscle/throat weakness
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15
Q

What is myasthenia gravis

A

autoimmune disease marked by muscular weakness without atrophy, and caused by a defect in the action of acetylcholine at neuromuscular junctions.

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16
Q

What is Duchenes muscular dystrophy

A

Duchenne’s: early childhood, increasing weakness, pseudo- hypertrophy of muscles, followed by atrophy resulting in swaying gait with legs kept wide apart

Growlers sign

17
Q

Myotonic muscular dystrophy

A

Myotonic: Hereditary, muscle membrane pathology. Muscle atrophy esp. neck/ face, cataracts, hypogonadism, frontal balding, cardiac disord

18
Q

Discuss MS

A

85% have optic neuritis, presents as pins/needles in hands/feet w/ hx or relapse/remissions, 20-40 yo

  • Affects membranes of motor axons, sclerotic plaques on myelin sheaths
19
Q

Differentiate acute and chronic compartment syndrome

A

Acute: direct blow involving brachial artery.
Chronic: repetitive UE exercise

  • Acute: swelling, pain, discoloration, decreased pulse, sensory alteration
  • Chronic: pain, numbness, sx, cease when exercise stops

Acute: surgery
Chronic: rest, correct training errors, alignment, balance, flexibility, NSAIDs

20
Q

Capsular pattern of the shoulder

A

Greater loss of:

ER>Abd> IR

21
Q

Capsular pattern of the wrist

A

Equal flexion and extension loss

22
Q

Capsular pattern of 1st CMC

A
  • abduction and extension
23
Q

Capsular pattern of the hip

A

IR > Abd > extn

24
Q

Capsular pattern of 1st MTP

A

Extension > flexion

25
Q

What are the 3 phases of healing

A

1) inflammation (1-4 days)
2) Proliferation (4-15 days)
3) Remodeling (15 day 6 months)

26
Q

What is the function of the inflammatory phase of healing

A

Function: restore homeostasis. Neutralize and destroy toxic agents (neutrophils, monocytes) by release of chemical mediators that increases local blood flow. Death of cell releases lysosome, activating healing process beginning with platelets and mast cells. Platelets helps forms fibrin which forms the clot (to hold wound together and to provide protection against foreign material) but has low tensile strength.

27
Q

Where is the posterior oblique ligament

A

Attaches the PCL to the lateral meniscus