All about the heart Flashcards

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1
Q

ACS definition

A

Unstable angina through AMI

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2
Q

Three principals of UA

A

At rest, <20 minutes usually
New-onset which limits walking 1-2 blocks or 1 flight of stairs
Diagnosed increasing in frequency or duration or threshold is lower

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3
Q

Angina precipitators

A

Exercise, stress, cold

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4
Q

Angina typically lasts

A

<10 mins, up to 20, improves within 2-5 minutes or after nitro

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5
Q

Atypical AMI symptoms

A

Fat beeties woman

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6
Q

S3

A

Present in 15-20% of AMI, may indicate a failing myocardium

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7
Q

TIMI - Thrombosis in Myocardial Infarction

A

0-2 have 2-9% 30 day risk of death, MI, or revascularization
Age 65+
3 or more traditional risk factors
Prior stenosis of >50%
ST segment deviation
2 or more anginal events in 24 hour prior
Aspirin use within 7 days prior to presentation
Elevated cardiac markers

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8
Q

isobar

A
Introduce
Situation/status
Observations
Background
Actions/assessment
Responsibility
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9
Q

AHS inclusion criteria

A

2mm in at least 2 of V1-V6
1mm in 2 of inferior or V4R
1mm in 2 of V5,V6 aVL
1mm depression V1-V2 and 1mm elevation V8, V9

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10
Q

Exclusion criteria (SIBBS)

A

Structural anomaly of brain vessels
Ischemic stroke within 3 months (except within last 3 hours)
Brain tumor
Brain bleed
Significant closed head or face trauma in 3 months

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11
Q

Relative contras ask about

A

BP, strokes, CPR, surgery in 3 weeks, internal bleeding, non-compressible bleeding from IV attempts
Preggo, active ulcers, pts on coumadin

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12
Q

Where da clot?

Elevation III > II plus depression of >1mm in I and or aVL

A

RCA PPV 94%

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13
Q

Where da clot?Elevation III > II plus depression of >1mm in I and or aVL PLUS V1 and or V4R elevation

A

Proximal RCA PPV 100%

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14
Q

Where da clot?

ST elevation in L, aVL, V5 V6 and depression in V1, V2, V3

A

LCA PPV 91%

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15
Q

Where da clot?
Elevation V1, V2, V3 +
>2.5mm V1 and/or RBBB with q wave

A

Proximal LAD PPV 100%

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16
Q

Where da clot?
Elevation V1, V2, V3 +
St depression >1mm in II, III and aVF

A

Proximal LAD PPV 93%

17
Q

Where da clot?
Elevation V1, V2, V3 +
<1mm depression / elevation in inferior leads

A

Distal LAD PPV 78%

18
Q

Reciprocal changes

A

From subendocardial ischemia, means larger area of injury risk, worse underlying CAD, more severe pump failure, increased mortality

19
Q

One of V5, V6, aVL with isoelectric or elevated ST in I

A

strongly suggestive of left circumflex lesion

20
Q

Tins nitro

A

WILL dilate atherosclerotic vessels if they have intact vascular smooth muscle
Inhibits platelet aggregation
If not treated with thrombolytics, it will reduce infarct size and improve regional function
Mortality rate is lowered by 35% with nitrates.
SHOULD be titrated to 10% reduction in MAP, or 30% if baseline is hypertensive NOT symptom resolution

21
Q

B blockers in AMI

A

Antidysrhythmic, anti-ischemic and anti hypertensive properties. Diminish MVO2 from decrease in hrt rate, arterial BP and myocardial contractility. Prolongs diastole for better perfusion

22
Q

When to use B-blockers

A
Increases risk of cardiogenic shock
Don't give if:
Signs of heart failure
Evidence of low CO
Age over 70, SBP<120, tachy >110 or brady <60
23
Q

Atrial kick in AMI

A

Normally lose 10-20% CO when kick is eliminated, lose up to 35% with reduced LV compliance when kick is eliminated

24
Q

Sinus tach in AMI

A

Poor prognosis, common in anterior. Treat anxiety, pain, pump failure etc

25
Q

Complete heart block

A

Anterior/inferior AMI as RCA and septal perforating branch of LAD supply AV conduction system
15% mortality without RV involvement, 30% with
Poor prognosis is due to how much is involved, not the block itself. Still pace though yo

26
Q

Mortality rates from CO in AMI

A

10% no heart failure
15-20% mild failure
40% frank pulmonary edema
50-80% cardiogenic shock

27
Q

Ventricular free wall rupture

A
Pericardial tamponade (90% death rate) 
Severe acute tearing pain. Hypotensive, tachy, possibly confusion and agitation, Becks triad
28
Q

RV infarcation

A
Usually not isolated to RV
From RCA or if left dominant LCX
Use V4R if inferior 
Right ventricle becomes kinda useless, left ventricle bulges into RV and moves blood that way, so if LV is involved thats pretty fucked up I guess
Tins says 1-2L of fluid
29
Q

Post PCI

A

Assume chest pain is abrupt vessel closure until proven otherwise, occurs in 4% of pts 2-14 days postprocedure

30
Q

Cardiogenic shock

A

A combo of damage to myocardium, and inflammatory response which depresses pump function and dilates peripheral vasculature

31
Q

Average ejection fraction

A

Above 50-55%

32
Q

Virchow’s triad

A

Hypercoaguability
Hemodynamic changes - stasis/turbulence
Endothelial injury/dysfunction

33
Q

Cardiogenic shock physical presentation

A
Usually <90SBP (higher if baseline is htn
PP <20
Sinus tach (check if pt is on BB) 
Tachypnea
Rales (unless isolated RV)
JVD
Hepatojugular reflex
Murmur