Aging and Exercise Flashcards
age of highest strength levels
20-40 (women and men)
- muscle cross sectional area is largest
- concentric strength of most muscle groups declines, slowly at first, then more rapidly after middle age
- capacity for power generation declines faster than that for maximal strength
declines in strength
- eccentric strength decline begins at a later age and progresses more slowly than concentric strength
- arm strength deteriorates more slowly than leg strength (less walking around/movement lower extremity)
- strength loss relates to limited mobility and fitness status, potential for increased incidence of accidents
muscle mass decrease
- begins to decrease at approximately age 30, decreasing by 10% by age 50
- muscle area usually parallels reduced fiber size, particularly reduced size and number of fast twitch fibers
motor unit remodeling and muscle mass decrease
motor unit remodeling involves motor endplate repair and reconstruction gradually deteriorates in old age
- leads to denervation muscle atrophy which, magnified by reduced physical activity, progressively reduces muscle cross section(strength), mass, and function
- affects type II muscle fibers the most
- leads to an increase in number of type 1 muscle fibers
sarcoponeia
losing muscle fibers
loss muscle fiber type
-lose fastest twitch (type IIB) the fastest, slow twitch maintained with old age
protein synthesis during aging
-slowed during aging
neural function
- aging leads to a 40% decline in spinal cord axons number and a 10% decline in nerve conduction velocity
- these changes likely contribute to the age-related decrement in neuromuscular performance assessed by simple and complex reaction and movement times
- a physically active lifestyle and specific exercise training affects neuromuscular functions positively at any age to slow the age-related decline in cognitive performance associated with speed of information processing
- overall reduction of nervous system ability to conduct impulses
endocrine function
- 40% of those ages 65-75 and 50% of >80 have impaired glucose tolerance that leads to type II diabetes
- pituitary gland decreases release of thyroid stimulating hormone thyrotropin, which affects metabolic function
- hormonal systems changed due to aging
- -hypothalamic-pituitary-gonadal axis, leading to menopause and andropause
- -adrenal cortex leading to a reduced output of DHEA
- -growth hormone/insulin-like growth factor axis leading to somatopause
age dependent changes in pulmonary function
major age related changes in pulmonary function -decrease elastic recoil of lung tissue --changes lung volumes --less surface area for gas exchange -stiffening of chest wall -decrease intervertebral space -weakening of respiratory muscle TV DOES NOT CHANGE WITH AGE -ability to take in as much air decreases, reserves impacted
pulmonary response to exercise
older adults display an attenuated response to exercise
- more flow limitation
- higher ventilatory rates
- approach maximal inspiratory pressure
- increased expiratory resistance
- approach TLC
- response remains the same, just not to the same extent
pulmonary function summary
- mechanical constraints cause deterioration in static and dynamic lung function
- pulmonary ventilation and gas exchange kinetics during the transition from rest to submaximal exercise slow substantially
- in elderly men, aerobic training increases gas exchange kinetics to levels that approach values for fit young adults and older endurance-trained athletes demonstrate greater pulmonary functional capacity than sedentary peers
- -basically still have improvements with old age
cardiovascular function
- VO2 max declines 1% per year and occurs twice as fast in sedentary compared to physically active
- decline in aerobic power with aging
- maximum exercise heart rate declines with age
- maximum cardiac output decreases in the trained and untrained due to lower max heart rate and stroke volume
- compliance of large arteries declines from changes in the arterial wall’s structural and nonstructural properties
- decreased capillary:muscle fiber ratio and arterial cross-sectional area causes lower blood flow to active muscle
cardiac output during exercise
EDV-slight increase
ESV-still increases, just not as much
-contractility decreases, dont eject as much blood, CO decreases, HR does not reach same values as YA
cardiovascular function summary
- attenuated response to exercise largely due to changes in cardiac output
- with aerobic exercise training many of those physiologic changes can be partially reversed
- -parallels improvements in YA