AdvMS Cardiac and Fundamental Peds Flashcards
peaked T waves occur in
hyperkalemia
PR interval represents
time for impulse to travel from SA node to ventricular depolarization. Prolongation indicates delay in AV function.
Normal PRI
0.12-0.20 seconds
QRS interval represents
time required for ventricular depolarization
normal QRS interval
0.04-0.10 sec or < 0.12 sec
RR interval represents
regularity of rhythm
EKG small square
0.04 sec (1mm)
EKG large square
0.2 sec
EKG 5 large squares
1 sec
EKG 5 large squares
1 sec
QRS interval measured
from first downturn of Q until reaches baseline after S (J-point)
ST segment measured
from return to baseline after S (J-point) to beginning of T. Should be on isoelectric line, elevation or depression d/t cardiac ischemia/MI
to calculate HR on EKG strips
count # or R waves on a 6-second strip (30 large boxes) and multiply by 10
Best for irregular rhythms
regular rhythm, rate 40-60, normal PRI and QRS
sinus bradycardia
causes of sinus bradycardia
vagal stimulation (vomiting/straining)
MI, IICP, uremia
Dig toxicity, BBs, CCBs
sinus bradycardia treatment
none unless symptomatic Atropine IVP (to decrease vagal stimulation), withhold meds, PM
regular rhythm
HR 100-160
normal PRI and QRS
sinus tachycardia
causes of sinus tachycardia
fever, blood loss, anxiety, HF, meds
Do not ignore, treat cause!
p wave is early, abn in size, shape or direction and sometimes hidden in preceding t wave
PACs
causes of PACs and tx
caffeine, nicotine, alcohol
CHF, MI, hypoxia
emotion, can occur in normal hearts
not treated but watch, can signal future arrythmia
causes of PACs and tx
caffeine, nicotine, alcohol
CHF, MI, hypoxia
emotion, can occur in normal hearts
not treated but watch, can signal future arrythmia
atrial rate 250-400, ventricular rate varies with number of impulses that make it through to AV node
Sawtooth wave forms
Aflutter
“flutter” waves
causes of Aflutter
CHF, MI, CAD, cardiomyopathy, hypoxia, thyrotoxicosis, electolyte imbalance
Aflutter/Afib tx
Dig or Verapamil to convert to A fib, which is easier to treat
RFA (ablation), cardioversion, BBs, dofetilide, ibutilide, IV adenosine or amiodorone
grossly irregular rhythm (unless very rapid)
atrial rate unmeasurable (400+), ventricular rate varies with impulses conducted through to AV node. Wavy deflections called f waves, varying rhythm and shapes. PRI unmeasurable, QRS normal
Afib
causes of Afib
CHF, MI, CAD, after OHS heart is irritated, pericarditis, heart valve disease, hyperthyroid, hypoxia
TEE before cardioversion for afib because
check for clots that could be dislodged once sinus rhythm is restored
TEE before cardioversion for afib because
check for clots that could be dislodged once sinus rhythm is restored
conplications due to afib
increase risk for clots (**CVA), cardiac output is decreased
BIGEMINY
every other beat is a PVC
early, wide QRS that differ from the QRSs of the underlying rhythm
PVCs
causes of PVCs
hypoxia, MI, hypokalemia, hypomagnesia, acidosis, exercise, Dig toxicity, hyperthyroidism, caffeine, CVADs, idiopathic
Regular rhythm, rate 140-250
no visible p waves, hidden in QRS
wide QRS
Vtach
causes of Vtach
same as PVCs but more dangerous because of the decrease in CO and tendency to develop into Vfib
Vtach tx
with pulse: meds (Lidocaine IVP, IV amio)
pulseless: CPR, defib starting at 50j, ICD
chaotic rhythm
rate=0 because no QRS complexes
no p waves, just “fine” or “coarse” deflections
Vfib
Vfib treatment
CPR, defibrillation
causes of Vfib
MI, CHF, CAD, ischemia with increased catecholamine levels (shock)
cardiomyopathy, electrolyte imbalance, etc.
most common cause of sudden cardiac death
Vfib
regular rhythm and p waves but prolonged PRI, normal QRS (can be wide sometimes with DIVC)
First degree Heart Block
DIVC=delay in ventricle
general causes of heart block
MI (can happen initially but then go away)
Dig, BBs, CCBs
treatment of first degree heart block
none, but watch for progression of heart block
regular atrial rhythm but irregular ventricular
more p waves than QRS waves
PRI progressively lengthens until a P wave occurs without a QRS, pause follows dropped QRS
Second degree heart block Type 1: Wenckebach
regular atrial rhythm, ventricular rate less and will depend on the number of impulses conducted–NO WARNING of a dropped beat, less QRS waves than p waves
Second degree heart block type 2:Mobitz II
less common but more serious than Wenchebach
high mortality rate if this is associated with acute MI; pt needs pacemaker and atropine to increase heart rate
Second degree heart block type 2:Mobitz II
regular atrial and ventricular rates but they beat independently of each other. No consistent PRI, some p waves hide under QRS (unique to this arrythmia)
Third degree Heart block
posterior fontanel closes by
2-3 mos
posterior fontanel closes by
2-3 mos
anterior fontanel closes by
18 mos
vaccines at 2,4,6 mos
Hep B Hib Prevnar RV IPV Dtap at 6 mos add Flu
rooting, palmar grasp, tonic neck (fencer) and Moro (startle) reflexes present
birth-4mos
plantar grasp reflex present
birth-8mos
Babinski reflex present
birth-1yr