advanced vet patient care 🫀 Flashcards

1
Q

what are the stages for the nursing process

A

Assessment
Planning
implementation
Evaluation

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2
Q

what are the sources of information required to effectively assess a px

A

The pet owner to provide information regarding normal activities
Nurse’s observations and clinical examination
Vet’s diagnosis and treatment plan
Patient history and records

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3
Q

what is the nursing model

A

A system that can be used to provide a structure for assessing the patient and standardising the nursing care planned.

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4
Q

what is a nursing diagnosis

A

The nurse identifying the patient’s actual and potential problems/patient needs.

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5
Q

list the names of the 5 types of open wound

A
  1. incision
  2. abrasion
  3. avulsion
  4. laceration
  5. puncture
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6
Q

describe incision

A

a clean, sharp cut created by a sharp object- scalpel

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7
Q

describe abrasion

A

damage with loss of epidermis/ dermis

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8
Q

describe avulsion

A

tearing of tissue away from attachments, underlying tissue and structures

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9
Q

describe laceration

A

irregular wound with damage to superficial and underlying tissue

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10
Q

describe puncture

A

a penetrating wound created by a sharp object- tooth

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11
Q

list the 2 types of closed wound

A
  1. contusion
  2. crushing
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12
Q

describe contusion

A

blunt force trauma which doesnt break skin but causes damage to skin and underlying tissues

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13
Q

describe crushing

A

force applied to the tissue for a period of time

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14
Q

what are the degree

A

class 1: clean wound with minimal contamination- 0-6hr duration

class 2: wound with significant contamination 6-12hr duration

class 3: wound with gross contamination 12+ hr duration

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15
Q

what are the phases of healing

A

haemostasis which occurs immediately after injury

inflammation which occurs within 6hrs, lasts 3-5days

proliferation which is the repair stage, occurs 3-7days post injury

maturation which is the remodelling phase- occurs 5-7 days post injury and lasts up to 2yrs or more

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16
Q

explain haemmostasis

A

Haemostasis – Blood and lymph flows from the damaged blood vessels and lymphatics to fill the wound and cleanse the wound surface. Almost immediately the blood vessels undergo a reflex constriction, and endothelial damage activates the platelet with subsequent formation of the platelet plug. Vasoconstriction only lasts 5-10 minutes until the blood clot has formed. The blood vessels then dilate, and intravascular cells and fluid pass through the vessel walls into the extravascular space. Vasodilation is mediated but histamine released from local mast cells activated by tissue damage. A combination of activated platelets, red blood cells, fluid and fibrin forms the firbin plug within the wound defect.

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17
Q

explain inflammation

A

The inflammatory and debridement phase typically lasts 3 to 5 days after the wound has occurred.
Blood vessels then dilate, increasing blood flow and bringing transudates into the wound, causing the heat, redness, and swelling of inflammation.
White blood cells in the exudate initiate debridement.
Neutrophils help break down bacteria and debris while stimulating monocytes.
Monocytes convert to macrophages, which continue to phagocytize debris and release growth factors that aid in tissue repair

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18
Q

explain proliferation

A

The repair phase (Proliferative), which typically occurs from days 4 through 12, is characterized by replacement of lost tissue with normal, functioning cells of the same type. Only tissue or organs that maintain a cell population – such as epithelia, bone or liver – and are capable of undergoing mitosis are able to heal in this way.
Angiogenesis begins as capillaries grow into the wound from the surrounding healthy vasculature.
Growth factors allow for migration of fibroblasts, which leads to creation of collagen (which provides wound strength) and myofibroblasts (which cause wound contraction).
Granulation tissue begins to form (4-7 days) , followed by epithelialization and wound contraction.
Epithelialisation may take weeks to months to fully stratify, may be incomplete or be thin and delicate
Wound contraction (5-7 days), area of wound reduces, surrounding skin stretches

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19
Q

explain maturation

A

The maturation (remodelling) phase begins when collagen fibres begin to orient along lines of stress, and can continue for years.
Wound edges meet, epithelialisation is completed
Redness reduces
The ultimate strength of the skin will be about 10% at 14 days, 25% by 4 weeks, and up to about 80% at several months

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20
Q

what factors promote healing

A
  • moist warm environment
  • good nutrition
  • tissue oxygenation
  • limited movement of wound edges
  • clean wound with good immune system
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21
Q

what factors delay healing

A
  • excessively dry or exudating wound
  • poor circulation- shock, concurrent conditions, age, recumbency
  • lack of essential nutrients- anorexia, poor perfusion, respiratory problems, lack of mobility
  • excessive wound edge tension, patient interference, damage at dressing changes, if deficit when skin has been lost there can be tension on the stitches which results in wound tension, infection
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22
Q

what is exudate

A

serosanguineaous in appearance
has a pink tingue to it
fluid that comes from wound

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23
Q

what does exudate contain

A

oxygen, nutrients, cytokines, growth factors, chemotactic factors, WBC, enzymes

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24
Q

where is exudate derived from

A

derived from plasma leaking from the cappillaries that leaks into the wound during inflammatory phase

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25
Q

what is exudate

A

exudate consists of wound fluid plus liquified necrotic tissue created by WBC’s via the autolytic debridement and oedema caused by inflammation.

exudate levels are proportionate to the amount of contamination, infection and tissue damage in a wound and should subside as the wound transitions from the inflammatory/ debridement phase to the repair stage.

good bandaging and antibiotics will help wound healing

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26
Q

if exudate is clear and pink tinged what does this tell you

A

its normal

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27
Q

if exudate is cloudy and varied in colour what does this tell you

A

its infected

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28
Q

what is the aim of wound management

A

provide a functional and cosmetic repair, relief of pain and distress to the animal, economic and time efficient procedures and prompt decision making in the event of signs of delayed healing

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29
Q

what do you need to assess in wounds

A
  • assess the whole px initially and stabilise
  • time since injury
  • what caused the wound
  • degree of contamination
  • degree of trauma at the site
  • necrosis
  • concurrent disease/medication
  • is treatment / cost a viable option for the px?
  • euthanasia
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30
Q

what is the first type of wound closure called

A

PRIMARY CLOSURE
- healing by first intention
- this is the immediate closure of a wound after injury. this process can be used to close clean or clean-contaminated wounds with surgery
- classification- clean
- management- immediate closure no tension

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31
Q

what is the 2nd type of wound closure called

A

DELAYED PRIMARY CLOSURE
- this type of closure occurs 2-5days after injury, before granulation tissue is formed. this process can be used to close clean-contaminated or contaminated wounds.
- classification- clean-contaminated or contaminated. questionable tissue viability
- management- lavage + debridement until healthy. appropriate dressing, closure after 2-3 days

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32
Q

what is the 3rd wound closure called

A

SECONDARY CLOSURE
- this type of closure occurs at least 5 days after injury and after healthy granulation tissue has formed. this process can be used to close contaminated or dirty wounds.
- classification- contaminated or dirty
- management- lavage + debridement , appropriate dressing, closure after 5-7 days, granulation bed has begun to form

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33
Q

what is the 4th wound closure called

A

HEALING BY SECONDARY INTENTION
- this type of closure occurs when a wound heals on its own by forming granulation tissue, epitheliasing and contracting.
- second intention is not selected for areas such as a limb on which the wound involves 2/3 or more of the limb because the risk that wound contraction will cause decreased mobility and or a tourniquet effect.
- classification- unsuitable for surgical closure. extensive contamination and devitalisation
- management- open wound management. lavage and debridement. appropriate dressing. allowed to heal.

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34
Q

what are 3 important things to remember with wound healing

A
  1. contaminated wounds should never be closed primarily. if in doubt treat as infected.
  2. don’t manage an open wound for an excessive period.
  3. if trauma caused the wound, let wound declare itself to give it time to disclose what tissue is viable or not.
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35
Q

what is involved in wound prep

A
  1. wear gloves
  2. ensure adequate analgesia is provided and had time to take effect
  3. GA usually required
  4. keep covered with sterile, non-lining dressing prior to prep
  5. swab wound bed for culture and sensitivity
  6. insert sterile water soluble jelly into wound
  7. clip around wound- total injury should be visible, clip passed unviable tissue and provide a 2cm margin
  8. debridement
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36
Q

what is the purpose of a lavage

A

helps to remove debris, reducing contamination and significantly reduces infection risk

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37
Q

what is the pressure amount for a lavage to be effective

A

8-12psi

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38
Q

what ml syringe is needed for lavage

A

20ml syringe

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39
Q

what size needle is needed for lavage

A

19G needle

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40
Q

how does debridement of a wound help

A

debridement prepares the wound bed by removing debris, contamination and necrotic tissue remaining after lavage. Debriding will reduce the risk of infection and promote healing

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41
Q

what is bioburden

A

the number of microorganisms that the wound is contaminated with

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42
Q

what is biofilm

A

forms when multispecies bacterial communities organise onto a wound surface and form an extracellular matrix of polysaccharides, proteins and nucleic acids to provide protection and ensure survival.

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43
Q

what does bioburden cause

A

prolonged inflammation and healing delay

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44
Q

what are the 3 forms of debridement

A

autolytic

mechanical

surgical

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45
Q

what is autolytic debridement

A

Use of primary layer applications such as alginates, hydrocolloids, hydrogels, honey, or sugar. This form of debridement is the most selective because it spares healthy cells and intact matrix molecules while removing damaged cells and matrix with microscopic precision.

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46
Q

what is mechanical debridement

A

Physical removal of tissue adhered to a dried-on
dressing; nonselective and, thus, the least desirable
form of debridement. Also, very painful so requires
anaesthetic and analgesia.

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47
Q

what is surgical debridement

A

Gold standard technique. Tissue removed by surgeon
according to characteristics, such as color, texture,
vascular supply, and temperature; selective on a
macroscopic level.

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48
Q

what is biological debridement

A

sterile environment and are applied to the wound bed. Maggots
may be applied with custom pre-constructed dressings or
Individually created dressing to keep the maggots securely In the
wound bed.

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49
Q

what is active debridement

A

Using a soft purpose made scrubbing brush such as Debrisoft

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50
Q

what are acemannan

A

Acemannan: Derived from aloevera and used on burns, dermal ulcers, lacerations, and radiation therapy wounds.11

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51
Q

what is alginates

A

Derived from kelp and used in the inflammatory and repair stages. Absorb fluid from the wound, convert to a gel, and thus should not be used on dry wounds.

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52
Q

what are bioactive dressings

A

Derived from living tissue and used in the inflammatory and repair stages. Provide a matrix for cell migration.

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53
Q

what is biotherapy

A

Living organisms such as leeches or maggots. Leeches decrease edema and venous congestion. Maggots debride necrotic tissue, and their
secretions provide antimicrobial benefits.

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54
Q

why use honey

A

Nonpasteurized honey (e.g., Manuka) provides antibacterial benefits, reduces edema, hastens sloughing of devitalized tissue, and promotes
granulation tissue formation.

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55
Q

what are hydrocolloids

A

Composed of hydrophilic polymers. Used during the repair stage on low-exudate to medium-exudate wounds. As exudate from the wound is
absorbed, the hydrocolloid liquefies to form a gel. These dressings are usually used in wounds that require additional moisture and natural debridement. Hydrocolloids actively stimulate wound healing and encourage debridement as they degrade on interaction with wound exudate. They are best used in dry to semi-dry wounds, requiring maintenance in an optimal moist environment. Dressings are left in place for several days and provide a near-ideal wound healing environment.

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56
Q

what are hydrogels

A

Hydrogels are used in wounds thought to be at risk of drying out. The main role of hydrogels is as a fluid donator for dry wounds. Hydrogels can both donate and trap water; therefore, they are useful for absorbing wound exudate, as well as hydrating and debriding necrotic material within the wound. A secondary dressing is required for hydrogels to work efficiently – this should ideally be a foam dressing with a semi-permeable film backing to maintain humidity and a moist wound environment.

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57
Q

why use sugar

A

Reduces edema and bacterial proliferation and promotes granulation tissue formation.1 Application should be at least 1 cm thick

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58
Q

list 4 closure techniques

A

sutures

surgical staples

surgical reconstruction- skin flaps or skin grafts

surgical drains - active or passive

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59
Q

what does open wound management include

A

covering the wound with an appropriate dressing and bandage; it doesnotmean the wound is left open to the environment.

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60
Q

what is the goal of open wound management

A

to work synergistically with the cells, providing the best environment possible to support the body’s wound healing process.

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61
Q

when is second intention healing appropriate

A

Healing is progressing well
Reconstructive surgery isnotneeded to prevent contracture or scarring that might inhibit mobility or be cosmetically unacceptable
The patient tolerates bandaging.

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62
Q

list the 2 types of periwound tissue damage

A

maceration- excessive production of exudate spilling onto adjacent healthy skin increasing susceptibility to infection

excoriation- contact with toxins from the wound causing damage to top layers of skin

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63
Q

how would you reduce maceration and excoriation from occurring

A

-Check underlying cause
-Re-evaluate the best dressing for appropriate absorbency
-Change the dressings more frequently
-Protect peri-wound tissue with barrier cream
-Ensure thorough cleaning at bandage changes

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64
Q

how is granulation built

A

Granulation tissue is built by fibroblasts, which secrete new extracellular matrix molecules (eg, collagen, elastin) and endothelial cells, which build new blood vessels

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65
Q

what colour is healthy granulation

A

bright red and moist. it has a slightly uneven appearance.

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66
Q

what is epithelialisation

A

Process when Epithelial cells on the skin edge migrate onto the granulation tissue, which provides the oxygen, moisture, and surface required for epithelial cells to proliferate, cross the wound, and create a new epidermis.

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67
Q

what is the rate of which epithelialisation occurs

A

1mm/ 10 days - 10cm wound takes 500days to completely epithelialise.

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68
Q

at what point would you change a dressing that has exudate and why

A

medium exudate as low exudate still has absorbency and excess exudate allows the dressing to become fully saturated which will lead to maceration of the surrounding healthy tissues

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69
Q

what does appropriate dressing selection rely on

A

-Effective wound assessment
-Good knowledge of stages of wound healing and how dressings create optimal wound environment
-Location of the wound site
-Consideration of the cost of dressings

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70
Q

what is a polyurethane foam dressing

A

These foams are highly absorbent and act by drawing excess exudate away from the wound, maintaining some moisture through humidity, which keeps the wound moist. They are commonly applied on top of other products – for example, hydrogels or honey. Foam dressings are now available with antimicrobial properties.

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71
Q

what is a polyhexamethylene biguanide

A

PHMB is as an antimicrobial agent exhibiting broad spectrum activity against bacteria and fungi. This PHMB within the dressing attacks bacteria in wound exudate as it is absorbed. This type of foam dressing is effective against Staphylococci (including MRSA), Pseudomonas, Proteus etc.

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72
Q

what do sodium chloride dressings do

A

A relative newcomer to the veterinary dressings market, this is a gauze dressing saturated in a 20 per cent hypertonic saline solution. This dressing promotes biological cleaning and the autolytic debridement process in non-infected and highly exuding wounds.

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73
Q

what do super absorbent dressings do

A

Wounds that produce vast quantities of exudate can be very difficult to manage. New dressings have been designed to cope with very high volumes of exudate by incorporating polyacrylate crystals into the dressings in combination with hi-tech silicone adhesives to make them very “wearable”.

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74
Q

what do silver dressings do

A

Silver and its salts have antiseptic and antibacterial properties. The silver in the dressings ionises to release active silver ions into the wound. The dressings require activation prior to use, by moistening with water for 10 seconds. Effective against Pseudomonas species, MRSA, E coli and common yeasts and fungi, including Candida.

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75
Q

why is laser therapy useful

A

-Use of low level laser waves to enhance wound healing and reduce/prevent infection
-Increases blood flow and oxygenation
-Reduces inflammation and pain
-Speeds up wound healing

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76
Q

why would you use a vacuums assisted closure

A

Use of negative pressure to encourage epithelialisation and contraction of the wound through the use of a vacuum pump sealed within a plastic dressing

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77
Q

list 3 wound complications

A
  1. devitalised tissue
  2. exuding wounds
  3. infection
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78
Q

list characteristics of devitalised tissue

A

-Provide optimum conditions for growth of bacteria
-Delays the inflammatory phase
-Reduces the viability of the wound bed

-Many causes; localised to wound vs systemic issues
Requires debridement

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79
Q

list characteristics of infection

A

Development – contamination, colonisation, critical colonisation, infection
-Signs – erythema, pain, oedema, localised heat
-Biofilm – protective coating produced by bacteria. Causes folding in of skin edges
-Causes damage or deterioration of the wound delaying healing, may cause systemic illness. Care AMR infection
-Check wound is properly debrided and cleaned, exudate level is managed, antimicrobial dressings (consider removal of debris and endotoxins from wound bed), systemic antimicrobials where indicated (culture & sensitivity)

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80
Q

how do you treat an infected wound

A

Environment: Dedicated/Isolated room. Incontinence pads & ventilation

PPE: Gown, gloves, face mask
Vet & Nurse discuss options & plan. Vet would carry out any debridement required.

Aseptic Technique

Swab wound for culture & sensitivity to allow approp antibiotic treatment

Lavage Solution: PHMB breaks down biofilm. If unavailable active

8-12psi – bag of fluids/giving set/3 wat tap/20ml syringe/18-20G needle. (Wound irrigation systems are now available)

debridement & isotonic solution

Amount lavage 100ml/cm2

Dressings: Antimicrobial e.g. PHMB, silver or honey. Good control of exudate

Redress according to exudate volume -2-3 days maximum

Waste – Non-hazardous non-infectious Vs Hazardous Infectious

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81
Q

why are drains used in practice

A
  1. Remove exudate and fluid from surgical sites especially where the surgery has caused dead space
  2. Allow monitoring of the surgical site ie abdominal surgeries- monitor free fluid composition and amount and replace loses
  3. Aid wound healing and reduce the risk of dehiscence
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82
Q

list characteristics of passive drains

A

-Use capillary flow
-Gravity
-Penrose drains
-Made of rubber latex
-Wider= more effective drainage
-Increased risk of infection of both the site and surrounding areas
-Can cause irritation of the skin when in place of from fluid
Sometimes the end is covered in absorbent material to absorb exudate
-Cheap and often used in GP or for minor infected wounds such as bite wounds

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83
Q

list characteristics of active drains

A

-Closed System, collects fluid into a reservoir
-Apply an artificial pressure gradient to pull fluid or gas from a wound or body cavity
-Less risk of ascending infection
-Higher efficacy
-Can be positioned in any position as not relying on gravity
-More expensive
-Continuous or Intermittent Negative Pressure

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84
Q

what is the Jackson Pratt drain

A

Active suction drains
Fenestrated drain attached to tubing that is then attached to a grenade
Air is removed from the grenade to create the negative pressure
When full or if there is an issue with the drain positive pressure will be present
Reduce dead space, remove air and fluid
Used for abdominal surgeries or larger wounds such as STS removal
Stay in until become non productive usually 3-5 days

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85
Q

what is the Redon / Red O pack drain

A

Same as Jackson pratt
Not used as to empty they have to be disconnected from the close circuit tubing unlike Jackson Pratt drains that have a separate area for emptying

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86
Q

what is the redovac drains

A

Completely closed active suction drains
Same as above the drain has fenestrated holes
Tubing to the reservoir bottle
This bottle is already primed as a vacuum DO NOT REMOVE THE CLAMP
A seal must be formed usually 4-6hrs post surgery then the clamp can be released allowing the drain to function
The reservoir has numbers to allow for recording volumes
They are bulky and not well tolerated in smaller species
Can be secured using surgical vests or cardio- vests

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87
Q

what is the fluid production for a drain removal

A

2-4ml/kg/24hrs

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88
Q

when are drains removed

A

when the fluid production is less than 2ml/kg/24hrs

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89
Q

what is the standard operating procedure for handling drains

A

-Barrier nurse
-Excellent hygiene
-Minimum PPE of gloves
-Passive- Clean regularly to prevent irritation and infection, ensure environment clean
-Active- Empty when +ve pressure, DO NOT EMPTY Q4hrs just because the hospital sheet says drain, check if +ve pressure, empty if –ve pressure, check connections and mark on –ve pressure no requirement to drain
-If reservoir/canister is full, they must be replaced not emptied and reused
-Every time they are emptied, fluid/air volume must be recorded if concerned check sample
-Prevent patient interference

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90
Q

how would you protect and cover a drain

A
  • tubular elastic net dressing size 10
    -pet suit
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91
Q

when are tracheostomy tubes used

A

Where complete upper airway obstruction has occured

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92
Q

why are tracheostomy tubes used

A
  • they provide Airway protection
  • Airway patency
  • Mechanical ventilation
  • If unable to intubate ie UAO
  • Laryngeal paralysis sometimes requires a longer term trach stoma
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93
Q

what is the complication of aspiration pneumonia when using tracheotomy tubes

A

The net result of lung exposure to fluid, solid matter and bacteria can be minor, with a temporary physical reduction of the number of alveoli available for gas exchange while the immune system removes the problem. Alternatively, such exposure may lead to local or widespread pulmonary inflammation or infection – termed aspiration pneumonia, acute lung injury or acute respiratory distress syndrome. This complication has serious implications

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94
Q

what are the complications of ongoing URT obstruction and regurgitation when using tracheotomy tubes

A

If the tracheostomy tube is too small, or partially or fully obstructed, the patient will still breathe with increased respiratory effort. The resulting negative intrathoracic pressure during inspiration will continue to predispose the animal to gastro-oesophageal reflux and, if the patient’s head moves below the level of the oesophagus, regurgitation may occur. This phenomenon is a risk in tracheostomy patients, as regurgitation does not involve a coordinated sequence of events causing the pharynx and larynx to protect the airway (as occurs during vomiting). Inhalation of fluid is therefore more likely, and the decreased effectiveness of the cough reflex and mucociliary escalator means the lungs are at greater risk of exposure to fluid, food and bacteria.

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95
Q

what is the complication of inflamed/ infected focus within the tracheal wall when using tracheotomy tubes

A

Wound exudate from the tracheostomy site flows down the trachea, thus increasing lung exposure to fluid and bacteria.

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96
Q

what is the complication of particle filtration is reduced when using tracheotomy tubes

A

More particulate matter is introduced to the respiratory tract;

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97
Q

what is the complication of no effective cough reflex when using tracheotomy tubes

A

Pressure can no longer build up behind a closed glottis, so fluid or solid matter within the airway can no longer be ejected effectively and will accumulate within the airway or flow (under gravity) to the lungs;

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98
Q

what is the complication of inspired air is not warm, humid or with laminar flow when using tracheotomy tubes

A

This causes inflammation of the respiratory epithelium, leading to increased mucus production, which, in turn, dries, causing thicker secretions or solid plugs. The mucociliary escalator no longer functions normally as the cilia are affected by the adverse conditions, particularly in the region of the tip of the tracheostomy tube;

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99
Q

when do you start nursing care for a tracheostomy tube

A

immediately.

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100
Q

explain the nursing care of an tracheostomy tube when using humidification

A

Humidification filters that can be attached to the end of the tube are one of the simplest ways to humidify inspired air for these patients. These are disposable. If the filters are unavailable, there are several alternative techniques which can be employed to provide humidification. One of these is the instillation of sterile isotonic saline (Burkitt Creedon and Davis, 2012). In order to provide proper humidification, 0.5–3 mls of sterile isotonic saline should be instilled into the tracheostomy tube hourly. Prior to instillation, the outside areas of the tracheostomy tube should be cleaned with chlorhexidine solution and sterile gauze. After cleaning and drawing up the sterile saline into a sterile syringe, the needle must be removed quickly and saline should be squirted into the tube without touching the sides. Nebulisation is another alternative method. Nebulising sterile saline for 10–15 minutes every 4–6 hours

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101
Q

explain the nursing care of an tracheostomy tube when using aseptic wound care

A

Good wound care is vital for all tracheostomy tube placements, as these sites become prime locations for bacterial growth. Hands should be washed and gloves worn prior to handling the tracheostomy tube or area surrounding it. Sterile gloves should be used when cleaning the wound. The area should be cleaned around the incision and under the tube with chlorhexidine solution-soaked sterile gauze. Chlorhexidine solution should be diluted to 0.05% concentration for cleaning the wound (Burkitt Creedon and Davis, 2012). The gauze must not be dripping as the solution should not be allowed to get inside the incision. The nurse can begin working at the wound edges and work outward, away from the incision. No ointments should be used in the incision and wound area. Sterile cotton-tipped applicators may also be used instead of sterile gauze squares. Dry gauze pads can be placed around the tracheostomy tube after cleaning to aid in the absorption of exudate. The gauze pads should not be cut as the small fi bres that will be loosened by the cutting action could be inhaled by the patient. Instead, the gauze should be simply folded as needed to fi t around the tube. The tube ties should be checked each time the wound is cleaned, to ensure the tube is secure, and should be changed whenever they become soiled.

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102
Q

explain the nursing care of an tracheostomy tube when using suctioning

A

Suctioning Patients with tracheostomy tubes produce various amounts of secretions. Regular suctioning is required but must be done very carefully as complications can occur. The patient should be pre-oxygenated for several minutes prior to suctioning. Aseptic technique must be followed (Fudge, 2009). A sterile, soft, long catheter that is pliable with fenestrations should be used. Silicone catheters are often preferred, but even a red rubber catheter may be used. The suction unit should not be turned on until the catheter is in place. Suction should be intermittent and light while moving the catheter in a circular motion to withdraw it. This process should take less than 15 seconds. Oxygen should be supplied and the patient given a break before repeating the process. Suctioning should be discontinued if there is a vagal response, cough, gag reflex, or any other adverse effects (Figure 5). This procedure should be practiced in advance of performing it (Figure 6). Veterinary nurses should use an alcohol based hand solution or disinfectant scrub to cleans hands and fingernails prior to the procedure. Ideally gloves should be worn in addition to proper hand sanitisation protocols.

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103
Q

explain the nursing care of an tracheostomy tube when using removal of secretions

A

The inner cannula of the tracheostomy tube should be removed every 4–6 hours at minimum and replaced with a new sterile inner cannula. The original cannula can then be cleaned and soaked in a chlorhexidine solution to be used for the next exchange. Additional saline may be infused into the outer lumen if copious secretions are noted. If single-lumen tubes are used, the entire tube requires replacement. Care should be taken not to do this too often as it will irritate the wound. Therefore, tubes with inner cannulas are preferred to single-lumen tubes. Coupage and changing the posture of the patient may also facilitate removal of respiratory secretions, especially if done immediately after nebulisation. While care of a patient with a tracheostomy tube is intensive and involved, when the outcome is a healthy patient returning home, this in itself is a reward. Veterinary nurses must remember to start care immediately once a tube has been placed, and to always be on the watch for complications such as dislodgment, obstruction, or occlusion of the tube. They must also be diligent about preventing secretions from building up and blocking the tube, providing aseptic wound care, and providing humidifi cation of air.

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104
Q

why is a thoracosotmy tube needed (chest drain)

A

A Thoracostomy drain is an in-dwelling catheter into the pleural space to drain air or fluid.
Accumulation of fluid or air in the pleural space separates the lungs from the chest wall thereby applying pressure and creating difficultly for the lungs to expand and fill with air, consequently causing clinical signs of dyspnoea, tachypnoea and hypoxia

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105
Q

what is the nursing care for a chest drain

A

All staff must be properly trained or could cause life-threatening iatrogenic pneumothorax
WHO handwash & aseptic technique
The clamp must be compressed with a metal C clamp, and sealed with an adaptor and capped with a 3 way tap
Record volume of air and fluid separately
Frequency – Q4hrs for the first several hours then redice as volume decrease and clinical symptoms improve.
A maximum 3–5ml of negative pressure should be applied to the drain to avoid trauma to the pleura and occlusion of the tube by mediastinal or pleural tissue
Entry site can be cleaned with dilute chlorohexidine
The drain should be. The drain site dressing must be removed and replaced twice daily using a sterile
Monitor tube for clamps becoming disconnected
Monitor Patients Respiratory rate, effort and pattern, mm colour and SPO2 (tissue perfusion) ECG monitoring (cardiac impact)

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106
Q

why are central lines used

A

-These are long stay catehters for use in patients that will
Be hospitlised for longer periods.
-They allow multiple blood draws from the line
-Allow larger volumes or rates of fluid therapy
-Allow higher concentrations of medications in constant rate
infusions that may be more irritant to perivascular tissue.
-Monitor central venous pressures

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107
Q

what are the contraindications when using central lines

A

coagulopathy, raised intracranial pressure, thrombosis,
or a contaminated site/skin condition over the vessel

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108
Q

what is the nursing care for central lines

A

-Aseptic Tehcnique
-Who handwash & Open glove
-Remove bandage material and examine site Q12hrs e.g. phlebitis, haematoma, kinking, dislodgement
-Clean site with dilute chlorohexidine & apply new dressing
-Bandage in place. Any exposed ports should be secured in body stocking
-Alcohol disinfecting caps should be applied over the needle free ports when not in use and should be changed each time the port is flushed
-Swab each port with alcohol prior to use
-Ensure that the catheter gate clamps are CLOSED whilst needle free valve is activated to prevent air embolism!
-All ports not in use must be flushed every 4 hours with heparinised saline. (use patient labelled bag of saline replace Q24hrs)
-If port becomes occluded – change needle free port for a red closed cap to prevent use & label DO NOT USE
-Dogs – walk on harness to avoid lead on jugular region

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109
Q

when would you use an oesophagostomy tube

A

The use of oesophagostomy tubes is indicated when feeding is required for more than 7–10 days in patients with a functioning, unobstructed oesophagus and healthy gastrointestinal tract.
In patients with disorders of the nasal passages, jaw bones, oral cavity or pharynx oesophagostomy tubes bypass the injured site and enable enteral feeding.

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110
Q

what are the complications of an oesophagostomy tube

A

Kinking, Blocking, Vomiting & Tube dislodgement

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111
Q

what is the nursing care when using a oesophagostomy tube

A

Oesophagostomy tube dressings should be changed and the stoma site cleaned daily at the minimum as infection can occur

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112
Q

what is the % of water in ECF

A

33%

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113
Q

what is the % of water in ICF

A

66%

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114
Q

what is the % of water in plasma

A

25%

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115
Q

what is the % of water in interstitial fluid

A

75%

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116
Q

list ways of the body water intake

A

drinking

food

metabolism

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117
Q

list ways of the body water output

A

respiration

urine

faeces

skin

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118
Q

what is an insensible loss

A

Insensible losses are where the body cannot adjust the losses. They will sometimes increase the amount it loses (e.g. with infection), but not decrease.

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119
Q

what are sensible losses

A

Sensible losses are where the body can adjust the fluid loss to conserve or get rid of fluid.

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120
Q

what is the ml/kg/day for insensible losses

A

10-20ml/kg/day

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121
Q

what is the ml/kg/day for sensible losses

A

30-40ml/kg/day

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122
Q

what is a solution

A

a solute dissolved within a solvent

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123
Q

what is a solute

A

a solid, liquid or gas dissolved to make a solution

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124
Q

what is a solvent

A

the liquid portion of a solution

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125
Q

what does isotonic mean

A

concentration equal to plasma

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126
Q

what does hypertonic mean

A

concentration higher than plasma

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127
Q

what does hypotonic mean

A

concentration lower than plasma

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128
Q

what is an electrolyte

A

a substance that dissolves in water and becomes ions which are charged particles. is conducts an electric current when dissolved / melted in water

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129
Q

what are cations

A

positively charged ions

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130
Q

what are anions

A

negatively charged ions

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131
Q

why does normal pH need to be maintained

A

for cell function to occur

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132
Q

how are the levels of hydrogen ions in the blood determined

A

by the amount of bicarbonate and carbon dioxide

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133
Q

what form of carbon dioxide is carried in the blood

A

carbonic acid

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134
Q

what does a change in pH do

A

decrease or increase in hydrogen or bicarbonate

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135
Q

what is osmosis

A

the movement of water from an area of low conc to an area of high conc through a semi-permeable membrane until concs are equal

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136
Q

what is osmotic pressure

A

the pressure with which water molecules are drawn across the semi-permeable membrane

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137
Q

what is hydrostatic pressure

A

the force excreted by a fluid against a wall which causes movement of fluid between compartments

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138
Q

what is the water movement across the capillaries determined by

A

the balance between hydrostatic pressure generated by the heart and the oncotic pressure generated by the proteins present in plasma

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139
Q

describe the movement of water in capillaries

A

In capillaries, hydrostatic pressure (also known as capillary blood pressure) is higher than the opposing “colloid osmotic pressure” in blood—a “constant” pressure primarily produced by circulating albumin—at the arteriolar end of the capillary . This pressure forces plasma and nutrients out of the capillaries and into surrounding tissues. Fluid and the cellular wastes in the tissues enter the capillaries at the venule end, where the hydrostatic pressure is less than the osmotic pressure in the vessel. Filtration pressure squeezes fluid from the plasma in the blood to the IF surrounding the tissue cells.

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140
Q

describe the water movement in cells

A

ECF and cytoplasm conc needs balanced so the osmotic pressure within the cell prevents excess fluid absorption. the fluid and nutrients move into cells from ECF. the fluid and waste is pushed out of cells to ECf. the fluid that is not absorbed into capillaries is then taken to the lymphatic system.

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141
Q

what does a decrease in plasma proteins or ineffective lymphatic drainage cause

A

oedema

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142
Q

what does homeostasis mean

A

any self-regulating process by which an organism tends to maintain stability despite external factors

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143
Q

what is ECF essential for

A

ECF is essential for normal cellular functions, and its composition is tightly regulated by homeostatic mechanisms. These mechanisms ensure that variables like pH, electrolyte concentrations, and fluid volume remain within optimal ranges, allowing our bodies to function effectively

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144
Q

what are the 3 organs in the body that regulate fluid balance

A

brain, adrenal glands and kidneys

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145
Q

what is the composition of ECF

A

The main component of ECF isinterstitial fluid, which surrounds cells.
Blood plasma is another significant component of ECF, especially in animals with a circulatory system.
Lymph constitutes a small percentage of interstitial fluid.
Transcellular fluid accounts for about 2.5% of ECF1.

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146
Q

how is osmolality controlled

A

Osmolality is controlled by hypothalamic osmoreceptors that stimulate thirst and the release of antidiuretic hormone (ADH) from the posterior pituitary gland. If net water loss from the body exceeds net water gain, plasma osmolality will rise and hypothalamic osmoreceptors then stimulate thirst and release of ADH. The augmented water intake and increased reabsorption of water by the kidney combine to decrease plasma osmolality towards normal.

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147
Q

how does the RAAS - renin angiotensin aldosterone system work

A

a drop in blood pressure or in fluid volume causes the kidney to release renin

angiotensin is release from the liver

renin acts on angiotensin to from angiotensin 1

the angiotensin converting enzyme is released from the lungs and it acts on angiotensin 1 to form angiontensin 2.

angiotensin 2 also acts directly on blood vessels stimulating vastcontriction but it also acts on the adrenal gland to stimulate the release of aldosterone.

aldosterone acts on the kidneys to stimulate reabsorption of salt and water.

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148
Q

what is a isotonic loss

A

loss of fluid that has similar osmolarity to plasma. e.g. vomiting and diarrhoea

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149
Q

what is hypotonic loss

A

loss of fluid that has a lower conc of water than plasma e.g diabetes insipid and panting

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150
Q

what is a hypertonic loss

A

a loss of fluid that has a higher conc of water than plasma e.g. hypoadrenocorticism

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151
Q

list the 4 types of losses

A

water, water and electrolytes, blood, plasma

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152
Q

what is the pathophysiology of the loss of water

A

plasma become hypertonic. fluid moves from ICF to ECF by osmosis

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153
Q

what is the pathophysiology of the loss of water and electrolytes

A

loss hypotonic, isotonic or hypertonic. movement depends on tonicity of fluid. hypertonic dehydration as water only loss.
hypotonic dehydration - fluid moves from ECF to ICF by osmosis
isotonic dehydration- no fluid movement

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154
Q

what is the pathophysiology of the loss of blood

A

most serious
isotonic loss therefore no fluid movement

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155
Q

what is the pathophysiology of the loss of plasma

A

increased PCV, loss of plasma proteins
proteins must be replaced

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156
Q

what are examples of the loss of water only

A

cannot drink e.g. injury
cannot concentrate urine e.g. diabetes insipidus

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157
Q

what are examples of the loss of water and electrolytes

A

vomiting and diarrhoea
metabolic disorders

158
Q

what are examples of the loss of blood

A

trauma
surgery
warfarin posioning

159
Q

what are examples of the loss of plasma

A

burns
scalds

160
Q

what is dehydration

A

a lack of fluid in the interstitial compartment

161
Q

what is hypovolemia

A

a lock of fluid in the intravascular compartment

162
Q

what are parameters affected by dehydration

A

Mucous membrane moisture
Skin turgor
Eye position within orbit

163
Q

what are parameters affected by hopvolaemia

A

Capillary refill time
Heart Rate
Pulse Quality
Blood Pressure

164
Q

what tests can help estimate fluid loss

A
  1. Packed Cell Volume Test (PCV) – inexpensive and quite revealing. For each 1% increase in PCV a fluid loss of 10 ml/kg has occurred. This can be unreliable where there is preexisting anaemia.
  2. Haemoglobin – dehydration results in an increase in haemoglobin values due to loss of plasma water, but care should be taken when interpreting results from an anaemic animal.
  3. Total Plasma Protein (TPP) – dehydration causes a rise in values. This can be measured from either a biochemistry test or from your refractometer. Measuring the total plasma protein is done in the same way as measuring the specific gravity of urine.
  4. Blood urea & creatinine – parameters will rise in the dehydrated patient but will also be high in an animal with renal disease.
  5. The specific gravity of urine – can also be an indicator of dehydration. If urine is highly concentrated this can be a sign of dehydration.
165
Q

what is the normal rate of urine output

A

1-2ml/kg/hr

166
Q

what is the rate for oliguria

A

under 0.5ml/kg/hr

167
Q

what is the specific gravity for a dog

A

1.015- 1.045

168
Q

what is the specific gravity for a cat

A

1.020-1.060

169
Q

what is a normal cats PCV

A

24-45%

170
Q

what is a normal dogs PCV

A

37-55%

171
Q

when will PCV decrease

A

anaemia

172
Q

when will PCV increase

A

dehydration

173
Q

what is total solids/ total protein

A

is a measure of the amount of plasma proteins in the blood; these include albumin, globulins and fibrinogen. Due to effect of osmosis during dehydration, TS will become elevated

174
Q

what is the normal TP for a dog

A

54-71g/dl

175
Q

what is the normal TP for a cat

A

54-78g/dl

176
Q

what happens when breathing is inadequate

A

When breathing is inadequate, carbon dioxide
(CO2, a respiratory acid) accumulates in the body, contributing to an acid state. The lungs can quite rapidly expel large quantities of carbon dioxide to reduce the quantity of acid in an effort to restore balance

177
Q

explain kidney regulation

A

The kidneys can also excrete more acid in their waste products, and retain more bicarbonate (HCO3−, a base or alkaline chemical), which is an important buffer employed within the body to help resolve or compensate for increased acid accumulation. If alkaline, the kidneys attempt to retain more acid and eliminate more bicarbonate (base substance) in an effort to maintain balance

178
Q

what are aboral acid-base balances indicative for

A

clinical disease processes and
can aid the clinician in identifying underlying causes of illness in the patient.

179
Q

what does the acidity or alkalinity of a soltution depend upon

A

how many hydrogen ions or molecules of C02 are present

180
Q

explain acidity

A

Hydrogen ions (H+) are a product of metabolism of protein and phospholipids. Carbon dioxide (CO2) is a byproduct of the metabolism of fat and carbohydrates. CO2 combines with H2O in the presence of carbonic anhydrase (enzyme/catalyst) to form carbonic acid.

181
Q

explain alkalinity

A

The body contains several mechanisms in order to maintain the desired “normal”, pH level, which is called buffering. A buffer is a compound that can accept or donate protons (H+) and minimize a change in pH

182
Q

what is metabolic acidosis

A

Metabolic acidosis occurs when there is an increase in the amount of acid in the body. This can be due to abnormal metabolic function, or ingestion of an acid or substance that is metabolized into an acid. There is a decrease in HCO3 and a compensatory decrease in CO2.

183
Q

what are examples for metabolic acidosis

A

vomiting
diarrhoea
renal failure
shock

184
Q

what is metabolic alkalosis

A

Metabolic alkalosis occurs when an excessive loss of sodium or potassium affects the kidney’s ability to control the blood’s acid‐base balance. There is an
increase in HC03 and compensatory increase in C02.

185
Q

what are examples of metabolic alkalosis

A

vomiting stomach contents only
over administration of bicarbonate

186
Q

what is respiratory acidosis

A

Where an acid state in the body occurs when respiratory system cannot excrete acid. Respiratory acidosis occurs when the lungs do not expel CO2 adequately. There is an increase in CO2, accompanied by a compensatory increase in HCO3−.

187
Q

give examples of respiratory acidosis

A

Respiratory obstruction
Acute respiratory failure
Hypoventilation for any reason
Anaesthetic problems

188
Q

what is respiratory alkalosis

A

Respiratory alkalosis occurs when too much CO2 is expelled from the bloodstream (typically from hyperventilation). There is a decrease in CO2 with a compensatory decrease in HCO3−.

189
Q

what are examples of respiratory alkalosis

A

Hyperventilation
Pain, stress
Hyperthermia
Excessive IPPV

190
Q

what is the volume of administration equation

A

fluid volume required= Maintenance volume + Deficit volume +
Ongoing Losses

191
Q

what is the maintenance rate

A

50ml/kg/day

192
Q

what is the deficit volume rate

A

10ml x % dehydration or % PCV increase

193
Q

what is the volume of ongoing loss

A

4ml/kg x vomit or diarrhoea or %burns

194
Q

how do you calculate the fluid administer rate

A

fluid volume / 24hr
fluid volume / 60 mins
fluid volume / 20
fluid volume/ 60secs

195
Q

what are the 3 categories of fluid

A

crystalloids
colloids
blood/ blood products

196
Q

what is a crystalloid

A

solution containing water and electrolytes. the electrolytes move easily across the endothelium into the interstitial space. can be classed as isotonic, hypotonic, and hypertonic

197
Q

what is a colloid

A

solution containing large molecules; plasma expanders. they remain in intravascular space longer than crystalloids.

198
Q

what is oxyglobin

A

a plasma expander with 02 carrying abilities

199
Q

when would you use oxyglobin

A

r dogs with anaemia, Oxyglobin acts as an immediate oxygen bridge to stabilise anaemic dogs until the underlying condition can be controlled and the animal’s body can produce its own new red blood cells. The treatment, which is now available in an individual 60ml transfusion bag, as well as a 125ml bag, maintains tissue oxygenation even when there is severe stenosis of blood vessels. It has a viscosity which is more than water but less than blood for ease of flow through vessels and also increases circulatory volume

200
Q

what do you take from blood/ blood products

A

whole blood- haemorrhage, anaemia, haemolysis

plasma - burns, hypovolaemia

packed red cells- anaemia

cryoprecipitate- clotting/ bleeding disorders

201
Q

what are the routes of administartion

A

oral
subcutaneous
intravenous
intraperitoneal
intraosseous

202
Q

what is the management of a drip line

A

Use unopened, sterile materials
Check bag is correct fluid, in date and clear
Prepare aseptically
Insert catheter up to the hilt
Ensure catheter is taped in dry
Change catheters ~ every 48-72hrs and dressings as needed if appropriate.
Flush catheter every 6hrs. DO NOT use fluids from drip bag
Check for signs of Phlebitis. Use Phlebitis Score system. (swelling, bruising, pain and perivascular fluid)
Check line regularly for kinks, blockages and interference
Prevent patient interference
Standard Operating Procedure (SOP) for Care

203
Q

what ways can you monitor a px

A

Check clinical signs for hydration level
Use calculated volumes
Monitor TPR and mucous membranes
Record urine output and SG
Monitor PCV
Monitor ongoing losses
(Central venous pressure)
Record all findings on fluid monitoring chart/ hospitalisation sheet
Review fluid therapy plan regularly
Monitor for signs of overperfusion
Weigh patient daily

204
Q

what is central venous pressure

A

Central venous pressure (CVP) is an estimate of the blood pressure in the right atrium. CVP reflects the amount of blood returning to the heart and the ability of the heart to pump the blood into the arterial system. CVP is directly proportional to the volume of blood in the anterior vena cava and venous tone. This pressure is decreased by hypovolaemia or vasodilation and is increased by fluid therapy or vasoconstriction. It can be used to guide fluid therapy administration in critically ill patients, or in patients with cardiac disease to help prevent volume overload

205
Q

what is over perfusion

A

This is when the patient receives too much fluid. This is quite common in small mammals, cats and small dogs, which is why fluids must be carefully monitored.
Fluid overload can lead to peripheral oedema, which can lead more seriously to pulmonary oedema and can lead to respiratory distress and death.
Excessive administration of colloids can lead to right sided heart failure, and congestive heart failure. Crystalloid overload, in the early stages causes diuresis – the animal may produce lots of very dilute urine resulting in an increased risk of peripheral oedema and then pulmonary oedema, and death

206
Q

what is shock

A

shock is an acute circulatory failure resulting in inadequate tissue perfusion and energy production. as shock develops, pxs deteriorate rapidly

207
Q

what does normal tissue perfusion rely on

A

Cardiac output
Circulating volume
Peripheral vascular resistance

208
Q

what does a reduction of any parameters result in with normal tissue perfusion

A

Inadequate perfusion
Triggers compensatory mechanisms
Altered efficacy of all systems

209
Q

what is compensatory shock

A

when the compensatory mechanisms can fix the underlying problem

210
Q

what are mechanisms to reduce compensatory shock

A

Baroreceptors detect reduced cardiac output, stimulating adrenaline and noradrenaline release - causing increased heart rate and contractility

Hypoxia of tissues results in metabolic acidosis. Ventilation increases to address acid – base balance.

Hypoperfusion of kidney activating the RAA system. Aldosterone acts on the collecting ducts to retain Na and H2O. Also causes peripheral vasoconstriction.

211
Q

what are mechanisms to reduce decompensatory shock

A

Where shock is not treated, compensatory mechanism start to fail
Fluid and proteins leak from circulation into the tissues due to peripheral vasodilation. Viscosity of blood increases.
Acidosis increases
As gut becomes ischaemic, bacteria enters the blood stream – toxic
Increasingly stuporous or comatose

212
Q

what is irreversible shock

A

to much cell death that cannot be reversed

213
Q

what can irreversible shock lead to

A

Systemic Inflammatory Response Sydrome (SIRS)
Inflammtory Injury to one organ system that can cause damage to others. Can have infectious or non-infectious causes
Disseminated Intravascular Coagulation (DIC)
Activation of haemostatic mechanisms inducing a prothrombotic state leading to bleeding tendancies
Multi-Organ Dysfunction (MOD)
SIRS & Septic Shock can lead to MODS. Every organ and system within the body can be affected
Death

214
Q

what are the types of shock

A

Hypovolaemic
Septic/ Distributive
Cardiogenic
Obstructive

215
Q

what’s hypovolaemic shock

A

The heart pumps well but Decreased, inadequate circulating volume
Most common type of shock
Occurs due to loss of blood, fluid or plasma
Results in a severe tissue hypoperfusion
E.g. trauma, ruptured abdominal organs, surgery, V &/or D, burns

216
Q

what are clinical signs of hypovolaemic shock

A

Clinical Signs:
* Tachycardia
* Reduced pulse quality
* Absent peripheral pulses
* Prolonged CRT
* Changes in mm colour
* Reduced urine output
* Obtundation

Signs depend on phase of compensation/decompensation

217
Q

what is septic/ distributive shock

A

Also classed as a Distributive shock due to hypotension
Heart pumps well but there is peripheral vasodilation
Gram –ve (and occasionally +ve) bacterial infection
Endotoxins released from ruptured bacterial cells
Toxins in the circulation increase capillary permeability, causing uneven fluid distribution
E.g. peritonitis, pyometra, intestinal strangulation, gastric haemorrhage

218
Q

what are clinical signs of septic/ distributive shock

A
  • Brick red mucous membranes
  • Tachycardia
  • Rapid CRT
  • Bounding pulses
  • Neurological signs
    o Depressed
    o Collapsed
219
Q

what are the 2 types of distributive shock

A

Hyperdynamic:
Increased cardiac output to improve tissue perfusion
Patient is compensating
Clinical Intervention – fluid therapy
Hypodynamic
Myocardial dysfunction
Patient is decompensating
Prognosis is poor

220
Q

what is cardiogenic shock

A

Reduction in cardiac efficacy
Heart loses its ability to pump effectively, leading to a reduction in cardiac output
Resulting congestion in liver and lungs, and oedema
Not a common type
E.g. cardiomyopathies, pericarditis, congenital defects, arrythmias

221
Q

what are clinical signs of cardiogenic shock

A
  • Dyspnoea
  • Presence of a heart murmur
  • Rhythm abnormality
  • History of heart problems
  • Very high HR (>220bpm in dogs)
  • Ascites
222
Q

what is obstructive shock

A

Obstruction of the normal blood flow
The heart pumps well but outflow is obstructed
Causes of obstructive shock include pericardial effusion and pulmonary thromboembolism
Due to pericardial effusion, blood is unable to fill the ventricles and hence cardiac output is reduced
Removal of the obstruction will cause resolution of the signs of shock

223
Q

what are clinical signs for obstructive shock

A
  • Pale mucous membranes
  • Tachycardic
  • Slow capillary refill time
224
Q

what are clinical signs of general shock

A

Tachycardia
Hypotension
Tachypnoea
Pale mms
Prolonged CRT
Hypothermia + cold extremities
Weakness/depression
Reduced urine output
As progresses, dilated pupils and coma
Different types of shock can display other signs e.g septic shock causes congestion of the mms and an increased CRT

225
Q

how do you evaluate shock

A

History
Physical examination
MM colour, CRT, hydration
Heart rate
Blood pressure
Pulse rate and quality – peripheral pulse
Temperature – core and periphery
Blood tests – Hb, PCV/ TS, Total protein, glucose, BUN, lactate measurement

226
Q

what is the diagnosis for shock

A

Lactate – a byproduct of anaerobic respiration. Therefore a direct indicator of inadequate cell perfusion.

In one study of dogs requiring intravenous fluid therapy, those with lactate concentrations higher than the reference interval 6 hours after presentation were more likely to die (Stevenson et al., 2007).

227
Q

how to manage shock

A

Identify and rectify cause
Establish adequate ventilation and oxygenation
Restore optimum intravascular volume
Maintain adequate cardiac output and renal perfusion
Maintain optimum internal metabolic environment

228
Q

how to treat shock

A

Oxygenate; resuscitation where required
Provide analgesia
Control haemorrhage
IVFT, increase circulating volume + restore perfusion
Bicarbonate to address metabolic acidosis/ Hartmanns
(Antibiotics)
Maintain body temperature

229
Q

what are the shock rate fluids

A

Isotonic Fluid
Dose Dogs: 90ml/kg (full blood volume)
Cats: 50ml/kg
As the patient won’t have lost its full blood volume, it is advised to administer 25% of this and then re-assess cardiovascular parameters. Continue until parameters returned to normal

Hypertonic Fluid
Dose Dogs: 4 to 5 mL/kg
Cats: 2 to 4 mL/kg
Administered over approximately 10 minutes. Infusion rates greater than 1 mL/kg/min may cause a vagally mediated bradycardia, vasodilation, and bronchoconstriction

Hydroxyethyl Starches
Dose Dogs: up to 20 mL/kg (divide into 5ml/kg boluses & reassess)
Cats: up to 10 mL/kg (divide into 2.5-3ml/kg boluses & reassess)
Titrate to effect

Crystalloid & Colloid
Dose Dogs: Colloid @ 5-10ml/kg + Crystalloid @ 4-45ml/kg
Cats: Colloids @ 1-5ml/kg + Crystalloid @ 25-27ml/kg
(equivalent to approx. half the shock dose)
Titrate to effect and continually reassess clinical parameters to adjust type and rate of fluids

230
Q

what is the nursing care for the shocked px

A

Carry out first aid
Select method of oxygen delivery
Organise fluid administration, IV, jugular, bilateral?, central line, intraosseous, lactated ringers / colloids / blood / Oxyglobin
Analgesia – under the direction of the veterinary surgeon
Dressings – haemorrhage control
Warming patient
Stress reduction measures
Constant monitoring vital signs and records
Liaise with owners

231
Q

what parameters would you monitor shock pxs

A
  • heart rate
  • blood pressure
  • mm
  • temperature
  • respiration (visual)
  • repeat bloods- PCV

re evaluating px

232
Q

what is blood transfusion

A

Blood collection
Storage of blood and blood products
Blood types and cross-matching
Blood administration

233
Q

when do we use blood transfusion

A

Indications for Transfusion:
Anaemia- Due to blood loss (regenerative – haemorrhage, haemolysis
Due to failure of production (non-regenerative)
Coagulopathy
Thrombocytopaenia

Packed Red Blood Cells indicated for patients:
showing hypoxia as a result of anaemia (PCV 12-15%=hypoxia)
In acute blood loss due to haemorrhage

Plasma indicated for patients:
Coagulopathy when there is active bleeding
For patients with Von Willebrand’s Disease undergoing surgical procedure

234
Q

what are sources of blood

A

Must be species specific
Donors must meet certain requirements

Methods of acquiring blood or blood products
Practice register of donors
Staff pets
Blood collection organisations
National donor register
Practice protocol for blood transfusion

235
Q

what are the donor requirements

A

Fully vaccinated & reg wormed
Health, fit adult (1 – 8yrs)
Dogs>25kg, cats>4kg, BCS 4-6/9
Normal PCV & blood tests
Not have travelled abroad
No infectious disease, cats, –ve FIV, FeLV, FIP, toxoplasma, Mycoplasma haemofelis
No sub-clinical renal disease
Not on medication
Good temperament
Blood typed (or typed on collection)

236
Q

what % can dogs and cat donate their blood volume

A

20%

237
Q

what is the maximum a cat can give

A

11ml/kg

238
Q

what is the maximum a dog can give

A

18ml/kg

239
Q

what Is the formula to calculate the total volume of blood

A

Volume of donor blood req (ml)

= 80(dog) x BW(kg) x Desired change in PCV
divided by

PCV of transfused blood

240
Q

what is autogenous

A

taken from animal to use later on same animal

241
Q

what is heteroenous

A

blood taken from one animal to give to another animal

242
Q

what equipment needs set up

A

Attire
Clean Scrub Top
2 Pairs of Examination Gloves

Equipment
Clippers/ cat clippers
Comfortable table/location
Local Anaesthetic Cream
Diluted warm chlorhexidine or equivalent and sterile swabs
Surgical Spirit
Collection Equipment (450ml single collection bag with CPDA-1/CPD or syringe primed with anti-coagulant for cats)
Electronic Scales weighing in grams
Metal line clamps (optional – used by pet blood blank)/Guarded Haemostat
Dressing Material for neck bandage
Scissors
Sedation drugs
Eye lubricant
IVFT 100ml crystalloid(for cats/small dogs)

243
Q

what is the process of a blood collection in a dog

A

2 people lift onto table and get comfortable
Lie in left lateral recumbency, chin pulled forwards and front legs pulled caudally
Put on gloves, wipe away local anaesthetic cream, and perform surgical scrub.
Change gloves
Place the collection bag below the patient and place on scales.
Assistant should raise the jugular vein
Phlebotomist should insert the needle bevel up in a caudal direction (cranial can be performed)
Once in the vein, the assistant can remove the clamp on the collection set
Blood should flow easily. Normal rate is 50ml/min so you can collect the full unit in under 10 minutes.
Mix the anti-coagulant with the incoming blood with a gentle rocking motion at 15 second intervals.
Assistant in charge of the collection bag should monitor the weight for the desired volume.
Once full inform the phlebotomist and clamps the line
Place a sterile swab over venipuncture site, remove the needle and have the assistant apply direct digital pressure for 2 minutes.
Strip the blood from the collection line into the collection bag. leaving 5cm in the line from the bag.
Place clamps on the line (can use knots but less secure and doesn’t prevent contamination)
Fresh whole blood should be stored at room temperature and must be used within 4-6 hours from collection
If transfusion is delayed blood can be refrigerated 4-6C, although platelet activity will be reduced.

244
Q

how to monitor the dog donor

A

check and record the donors demeanour, pulse rate and pulse quality and mm colour

allow them to sit up slowly an lift them onto the floor

continue to monitor for 10-15mins and offer them a drink of water and a small meal of food

lead walk only for 24hrs. use a harness instead of collar / lead

245
Q

what is the blood collection process for a cat

A

Cats quite often sedated depending on temperament – if so, lubricate eyes and provide oxygen
A ratio of 1ml citrate based anti-coagulant to 7ml whole blood
Gently restrain in sternal recumbency with neck extended, using minimal restraint.
Disinfect venipuncture site
Change gloves
System of primed butterfly catheter, line clamped with haemostat and syringe. Keeping vein raised throughout.
Flow rate should be a rate of 5ml/minute or greater
Rock the syringe throughout to mix blood and anti-coagulant
Clamp and turn off 3 way tap before removing
Apply digital pressure to venipuncture site for 5 minutes

246
Q

how do you monitor the cat

A

monitor the pxs vitals and bp for hypotension whilst In a warm kennel

donations of 10ml/kg or more will require IVFT. This should be administered post donation via i/v catheter at a rate of double the volume collected over 60-120 minutes

once awake can be offered water and light meal

outdoor cats should be kept indoors overnight

247
Q

what’s involved with blood post collection

A

Bag clamped and blood removed from tubing
Blood bags should be labelled with donor name, collection date and time, blood group and quantity of blood donated, name of phlebotomist
Collected blood may be
Given directly to patient
Stored for future use
Separated to use RBC’s, plasma, platelets and clotting factors
Must be collected and stored in anti-coagulant
Heparin – for immediate use
Citrate phosphate dextrose (CPD)
Acid citrate dextrose (ACD)

248
Q

that blood products are taken in canines

A

Whole blood
Packed red blood cells
Plasma
Fresh frozen plasma (FFP)
Frozen plasma (FP)
Plasma products
Cryoprecipitate (CP) – Factor VIII, vWf, fibrinogen
Cryosupernatant (CS) – albumin, globulin, antithrombin, Proteins S and C, Factors II, VII, IX, X, XI and XII
Platelet products
Platelet rich plasma (PRP)
Other platelet products not currently available in UK

249
Q

how to store blood

A

If not used immediately blood should be stored at 4°C in a temperature controlled fridge
Longer storage,
Plasma may be frozen within one hour at -18°c for under one year (FFP)
Plasma frozen for over a year – frozen plasma (FP)
Cells can be refrigerated for three months

Blood banks process and store blood and blood products to ensure maximum longevity

250
Q

what are the blood types

A

Dogs have DEA (dog erythrocyte antigen). Only DEA 1 can be typed, +ve or -ve
Cats have three blood types (A, B, AB)
60% dogs are universal matches FIRST TIME
Risk of reaction increases at each transfusion
Higher risk of transfusion reaction in cats
Best practice to identify blood type in either species; always determine blood type of cats before blood transfusion and any animal previously transfused
Test kits available to identify if DEA +ve or –ve – Alvedia, Rapid Vet-H, DMEVet, Diamed G

251
Q

in cross matching what is major

A

donor RBCs and recipient plasma

252
Q

in cross matching what are minors

A

donor plasma and recipient RBCS

253
Q

what is the equation to calculate how much product to administer

A

blood volume to be transferred mls=

k (70 for c, 60 for f) x weight x required PCV recipient PCV divided by PCV of red cell product

254
Q

how to administer blood

A

Check blood bag for damage
Ensure it is correct blood type and/or is cross-matched
Warm in water bath to 37°C (max) or room temperature
Gently invert/mix whole blood
MUST Use blood administration set – reduce risk of microclots
MUST use appropriate infusion pump
Administer slowly at start (0.5-1.0ml/kg/hr) and monitor for signs of transfusion reaction
Increase rate (5-10ml/kg/hr) after 30mins if no signs
Continue monitoring
Stop once calculated volume been administered

255
Q

how to set up a transfusion

A

Any blood product must be administered through a 170-260 μm filter to reduce the risk of microclots or debris and set up in an aseptic manner using non-sterile gloves to reduce the risk of iatrogenic bacteraemia. Avoiding disconnection of the transfusion once connected also reduces this risk. A giving set can be inserted into the unit or it can be drawn out into syringes if smaller volumes or rates are required.

The blood product must be administered via an appropriate infusion pump as some may damage red blood cells (Kisielewicz & Self, 2014). A plasma warmer, such as the SAHARA©, is usually used for defrosting the plasma but can be used to gently warm the pRBCs to combat hypothermia, especially if multiple blood product transfusions are to be given, patients are small or particularly young, if the patient is under general anaesthesia or is already hypothermic (Kisielewicz & Self, 2014; Prittie, 2003).

256
Q

what are the transfusion reactions

A

Acute or delayed
Acute signs – immediate
Intravascular haemolysis – fever, tachycardia, dyspnoea, vomiting, shock, DIC, collapse, haemoglobinaemia
Extravascular haemolysis – milder signs, hyperbilirubinaemia (large amount of bilirubin)
Allery – urticarial type reaction, facial swelling
Delayed signs – 24-48hrs to 21 days
Ineffective transfusion, reducing PCV, fever, anorexia, jaundice

257
Q

how to monitor a transfusion

A

Monitor closely dedicated team member
Obtain baseline parameters prior to transfusion
Check and record all parameters on monitoring sheet
Rate of transfusion: set by vet
0.5-1ml/kg/hr for the first 15-30 minutes
If no reaction- then can administer the remaining over 4 hours
Monitoring intervals: Continuous for the first 30 minutes. Record every 5 minutes. Then can monitor every 15 minutes till end of transfusion
Signs of Reaction:
Tachycardia
Urticaria (facial swelling)
Hypotension
Vomiting or
Diarrhoea,
Dyspnoea/tachypnoea
Pyrexia
Haemoglobinuria.
Transfusion reactions require treatment with glucocorticoids, antihistamines and/or adrenalin may be considered. Antipyretics may be required.
If circulatory overload has resulted in pulmonary oedema, diuretic treatment and oxygen support
may be required.

258
Q

what factors influence the timescale for evaluation of each nursing intervention

A

The severity of the patient’s condition
The age of the patient
The normal frequency of the activity
The timescale of each goal/aim

259
Q

What information may be required in order to effectively evaluate the efficacy of the nursing intervention?

A

The goals set in the planning stage to identify if they are met
Nursing observations and clinical examination
Vet information

260
Q

How is the information from the evaluation used?

A

It identifies which nursing interventions have been successful, and by how much. This is then used to decide whether to change the intervention, alter the frequency, adapt it or stop it completely. Therefore revising the care plan.

261
Q

what are the SMART goals

A

specific – describe the outcome in a focused way. Use an objective view. the outcome should be related to a number, a percentage, fraction or frequency
Measurable - some form of measurable outcome so that a judgment can be made using a numerical element
Achievable – Is the goal set achievable? Has it been done before? Are there any contraindications to doing it?
Realistic - patient-specific factors should be taken into account to assess whether the goal is actually appropriate for the patient in question
Timing - goals should include a date or time by which they will be accomplished or completed.

262
Q

how to assess a cardiax px

A

client info- symptoms seen at home? normal routine for dog, medication,
history - any symptoms?
clinical examination- as we are staying with the px the whole time, we need to know whats normal so when doing the re evaluation is it better or worse
diagnostic tests
assessment of px need and potential problems

263
Q

what are pxs like with cardaic problems

A

fragile, open mouth, panting, a lot of movement in abdomen ( increased effort in breathing) - not just in chest, abdominal muscles are working hard as well. increased movement of chest resulting in reduced movement in lungs possibly due to a mass or fluid resulting in harder breathing

264
Q

what should you do with a px with cardiac distress

A

analyse breathing pattern
any history of breathing problems
reduce stress
place in a quiet area
give 02
provide rest or sedation
open mouth breathing in cats
don’t handle animal - stress- death

265
Q

what do you see in an ultrasound in pxs with cardiac distress

A

thickened mitral valve and tricuspid valve.
you won’t see as much contraction of the muscle around the ventricle

possible fluid around the heart

enlarged heart- cardiomegaly

on the x-ray there should be distinct areas of organs - there shouldn’t be all white or all black

266
Q

what should be assessed in the clinical examination

A

pulse rate- (femoral and sublingual come from the aorta) by using tarsal and metacarpal -peripheral pulse, the pulse might be different to indicate the heart - weak or binding or absent or irregular), rhythm and quality

compare pulse and heart rate- pulse deficit
blood pressure- detects low or high bp, vasoconstriction of blood vessels will maintain bp, if low, the blood can’t get where it needs to go. may have to wait until the animal is calm.

use stethoscope, listen to heart. muffled if fluid in heart, rate, rhythm, quality

capillary refill time- indicated blood is getting to peripheries, delayed or compensatory phase.

mm colour- pale if loss of blood volume

resting respiratory rate

crackling noises in lungs- fluid

engaging abdominal muscles?

is animal sitting up with head and neck extended for lung maximum extension or are they lying on their chest

pulse oximetor- device which is placed on a pulse point which tells the level of 02 in blood- you want it to be above 95, if below they have low 02

abdominal fluid wave- if abdominal is full of fluid- ascites, if one hand is on one side of abdomen and other is tapping the other side of abdomen, you will feel a thrush of fluid

267
Q

what diagostic tests will the vet use

A

radiography- radiogrpah of chest can tell size of heart. the heart takes 3 rib spaces, measure from apex- bottom. can also tell if any mass or fluid in pleural cavity

ECG- electrical activity of heart. is electrical impulses travelling through the heart normally?

echocardiography- ultrasound, can do cross section of heart, measure vessel walls, look at valves, gives accurate presentation of a mass or blot clot.

blood pressure

268
Q

ways of doing an ECG

A

special attachment - halter or telemetry- this goes onto the animal and they go home with it. it takes a continuous ECG of the animal. animal is more relaxed at home therefore beneficial doing it this way.

ECG performed in practice- only seeing what happens in heart in that time. they might be stressed from being in the vet

269
Q

how to do a ECG

A

need a machine
carry out in quiet, stress free environment
place animal in R lateral recumbency with gentle restraint- if too stressful, can do sitting ( may affect ECG if panting) or use sedation however this could affect the ECG.
needs to have a contact medium- ultrasound gel or surgical spirit.
has a sticky pad or crocodile clip
machines will have 3-4 leads that attach under the armpit and groin
clip legs and swab with spirit

270
Q

what colour are ECG cables

A

red- R foreleg
yellow- L foreleg
green- L hind leg
black- R hindleg

271
Q

what is the burst speed for ECG

A

30sec burst

272
Q

what will the ECG pattern look like

A

shoudl be a P for every QRS
should be a T for every QRS
regular pattern

273
Q

what does the echocardiography (ultrasound) scan look like

A

will show
- heart structure. should see each atria and ventricle

274
Q

what does an echocardiography show

A

function and blood flow

275
Q

what sound does the heart make

A

lub dub
lub - closing of the AV valves
dub- closing of aortic and pulomoric valves

systole- time inbetween lub and dub

276
Q

what does a murmur sound like

A

whooshing sounds, like a drum, very abnormal

277
Q

how to monitor blood pressure

A

cuff around leg 40% of leg/tail circumference

278
Q

why does a cuff inflate

A

to stop blood flowing through vessels

279
Q

what is the normal rate for a dog for bp

A

133mmHg systolic

280
Q

what is the normal rate for a cat for bp

A

124mmHg systolic

281
Q

what is a direct method of measuring bp

A

invasive technique using arterial catheters

282
Q

what is an indirect method of measuring bp

A

non- invasive using external cuff

283
Q

what veins can you use for measuring bp with a cuff

A

cerphalic or tail
clip hair
apply surgical spirit
apply ultrasound gel

284
Q

what needs and potential problems are there for a cardiac px

A

urinating and defecating- may not have control as so stressed. animal will use up all of 02 reserves if being taken out

eating as animal too stressed to eat

body temp- as animal is stressed, we need to maintain temp as they will get very hot

give animal 02 - compromised breathing

prepare IV access

place animal in quiet dark room, minimal staff, minimal handling

may be showing abnormal behaviours as so stressed. can become aggressive as they are panicked.

pain relief

285
Q

what are the nursing interventions for a cardiac px

A

stress free environment
minimal handling, reduces stress and avoid restriction of chest/neck, harness, care with restraint technique
controlled exercise- own pace, observe impact, care against syncope. provided minimum activity in advanced disease, use harness instead of collar.
provide oxygenation, lung care
IV access.
warm food as increases palatability.
frequent opportunity for urination, monitor fluid intake and weight (if loosing weight -not eating, dehydrated).
restrict sodium- pulls fluid into areas of a body
maintain body temp- environment temo- window, thermostat, radiator, fans, cool mat, cold towels placed beside them or underneath. DONT place on top as stops evaporation.
support rest or sleep - comfy bed, no lights, don’t have someone coming in all the time
give accurate medication

286
Q

how to provide home care

A

educate the owner on what to do so they have realistic expectations
ensure owner knows how to provide meds- if has side effects, tell owner.
exercise in moderation - reduce walk time by half.
cardiac diet- healthy treat options ( grain free)
no smoking in household
give boosters as don’t want to get kennel cough

287
Q

what are common signs of respiratory diseases

A

nasal discharge
sneezing
stertor
stridor
cough
dyspnoea
tachypnoea

288
Q

what will the lungs sound like with respiratory pxs

A

wheeze
crackles
stridor- HIGH pitched sound created as air passes through a narrowed airway during breathing. may be present during inspiration and expiration.
stertor- LOW pitches sounds (gasp or snore like) heard during inspiration. generally of pharyngeal origin

289
Q

what is eupnoea

A

normal breathing

290
Q

what is orthopnoea

A

adopting an upright or standing or sitting position due to difficulty breathing

291
Q

how to assess a respiratory px

A

Initial Observation
Assess Respiratory Rate, Effort and externally audible noise.
Signs of trauma or abdominal distension
Lung Auscultation
Increased Lung Sounds e.g. crackles, wheezes, harsh lung sounds
Are associated with lower airway and pulmonary parenchymal disease
Decreased Lund Sounds are associated with pleural space disease
Heart Auscultation
Listen for any heart murmur or irregularities in heart rhythm that could indicate underlying heart disease

292
Q

what oxygenation parameters do you assess

A

mm colour for signs of cyanosis

pulse oximetry measures the % of haemoglobin that is saturated with 02

capnography- measures c02 in expired gas and assess partial pressure of c02 in arterial blood. it also assess ventilation.

arterial blood gases- best assessment for ventilation and oxygenation

ultrasonography and radiography

293
Q

what pxs may get respiratory issues

A

any px with dyspnoea

airway obstruction

pleural space disease

pulmonary oedema

pulmonary contusions

pneumonia

feline asthma

pulmonary thromboembolism

diaphragmatic rupture- need the diaphragm to breathe

severe anaemia

cardiac px

shock cases

294
Q

what are the aims for respiratory therapy

A

increase 02 saturation of the blood and tissues and decrease respirator effect. minimise stress, maintain optimum temperature, minimal handling

295
Q

what are other methods of providing 02

A

flow by oxygen - hold tube with 02 coming out of tube
face mask- needs a close seal
nasal prongs- cant have a high flow rate as damages nares
nasal canula
oxygen cage
oxygen tent- cling film around kennel and apply 02
incubator - control temp, humidity, can feed 02
intubation +/- ventilator
tracheostomy/ transtracheal catheterisation

296
Q

what is the rate of flow of 02

A

2-3L/min

297
Q

what % of 02 does the flow by provide

A

25-30%

298
Q

what % of 02 does the face mask provide

A

50-60%

299
Q

what % of 02 does the nasal prongs provide

A

30-50%, rates higher than 4% can be uncomfortable

300
Q

what % of 02 does the 02 tent provide

A

30-80%

301
Q

what are potential complications of o2 therapy

A

Decrease of O2 saturation to vital tissues due to stress and handling

Increased dyspnoea – keep sternal/lateral recumbency, NOT dorsal, do not lean on patient

Drying of nasal mucosa – oxygenation longer than 2hrs, delivered directly into the respiratory tract or at rates of over 4L/min, requires a humidifier

Oxygen toxicity - >50% O2 for over 12hrs. Start 100% then reduce to lowest level that provides effect

Atelectasis – due to alveoli filling with fluid. Low intensity mobilisation, turning the patient, nebulisation and coupage, pain management

302
Q

how would you evaluate 02

A

Maintain blood oxygen levels above 95%
Maintain normal mucous membrane colour
Decrease respiratory rate and effort, reduction of paradoxical breathing
Monitor frequently to assess effect of interventions, review plan and identify complications

303
Q

what is an oesteoblast

A

cell which is involved with bone formation

304
Q

what is an oestoclast

A

a type of bone cell that breaks down bone tissue

305
Q

what is an oestocyte

A

ceel found in mature bone

306
Q

what is cortical bone

A

hard outer shell of bone

307
Q

what is cancellous bone

A

porous inner part of bone

308
Q

what is a fracture

A

a complete or incomplete break of the bone continuity with or without displacement of the resulting fragments. this is often accompanied by other injuries

309
Q

are fractures life threatening?

A

no, less priority. you would need to establish px for bleeding, stabilise heart or any life threatening injury

310
Q

how do you suspect a fracture

A

2 radiography views to specifcally diagnose

311
Q

what causes a fracture

A

a force to the bone

312
Q

what is tensile force

A

act to lengthen the bone

313
Q

what is compressive bone

A

shortening of bone

314
Q

what is shearing forces

A

typically parallel or tangential to the bone

315
Q

what is bending forces

A

create a convex aide of the bone and a concave side

316
Q

what are bending forces reffered to

A

moments

317
Q

How would you assess a patient that has arrived at the surgery with a fracture?

A

ABCs

breathing-
panting? open mouth breathing? respiration rate, pattern, effort greater on inspiration or expiration, any noise, stridor or starter? dyspnoea?

pulse-
check heart rate with stethoscope, mm refill time and colour, feel the femoral pulse and peripheral pulse, do they match up?

temperature-
ear thermometer or rectal thermometer.

if collapsed, assess airway

shock-
check bp , blood test if PCV is normal, total solids, lactate test- measure whether there is cell respiration occurring with the presence of 02. of cells don’t have 02, circulation isn’t going to tissues. if lactate is above 2.5, there is anaerobic respiration (lactate buildup).

check for IV

318
Q

How would you stabilise the patient?

A

Check and stabilize vitals (temperature, pulse quality and heart rate, respiration rate, blood pressure, pulse oximetry), if needed.

Perform thorough physical, orthopaedic, and neurologic examinations.

Pursue initial diagnostics, including blood analysis, thoracic and abdominal radiographs, and an AFAST ultrasound.

Resolve any life-threatening issues, which means that surgery may need to be delayed for several days due to conditions, such as pulmonary contusions or hypovolemia.

Administer proper analgesiaas soon as possible

ALWAYS SUPPLEMENT WITH 02

319
Q

what are the 2 types of fracture healing

A

direct and indirect

320
Q

what is direct fracture healing

A

Required rigid internal fixation. Occurs via a combination of contact and gap healing.
Contact Direct Healing occurs when the surfaces of the fracture are in direct contact. The fracture is stable, and there is no compression applied to the fragments.
Gap Direct Healing occurs when an interfragmentary gap of < 1 mm is present.

Direct – bone edges so close together that callus doesn’t form and the bone forms without the interim stage of fibrous tissue and cartilage– usually require surgical fixing asap after trauma.

321
Q

what is indirect fracture healing

A

The most common ‘natural’ healing process, whereby the fracture ends are placed close to each other (but not apposed), with intervening haematoma and variable displacement and/or angulation.

322
Q

what is the healing process of fractures

A

The process for normal fracture healing starts immediately at the time of the fracture with formation of a haematoma.
Inflammation:This phase traditionally lasts 3 to 4 days or longer, and is characterized by a fibrin-rich clot at the fracture site. This clot releases growth factors to simulate bone healing and potentially acts as a scaffold for migration of inflammatory and reparative cells.
Repair:During this phase, the clot is slowly replaced by granulation tissue, which adds slight mechanical strength. As collagen fibers become more abundant, granulation tissue is replaced by connective tissue and, after formation of connective tissue at the fracture site, resident mesenchymal cells differentiate into chondrocytes to form cartilage. With the help of growth factors, such as bone morphogenic proteins, the cartilage begins mineralizing to form woven bone.
Remodeling:This phase is characterized by a slow adaption of the bone to regain its original function and strength. It is a very slow process (up to 6–9 years in humans) that represents 70% of the fracture’s total healing time. The action of osteoclastic resorption and osteoblastic deposition is guided by Wolff’s law.

323
Q

what are the conditions for normal healing

A

For normal fracture healing to occur a number of requirements must be met:
viability of fragments (i.e. intact blood supply, if they dont they will die off and wont heal)

mechanical rest: this can be achieved by not moving and external immobilisation, e.g. cast or internal fixation.

absence of infection

324
Q

what are the 3 types of process of healing

A

spontaneous (indirect/secondary) healing
contact (angiogenic/primary) healing
gap healing

325
Q

wha are the influencing factors on healing times

A

Immature animals –
Improved vascularisation of callus in juvenile increasing rate of healing

Geriatric –
Reduced vascularisation of callus so prolonged healing compared to young and adult

Debilitation -
Hormone/kidney failure

Osteomyelitis –
Inflammation +/-Infection that interferes with healing

Cancellous vs Cortical-
Fractures in cancellous bone heal faster than cortical bone

Fracture sight -
Bone with good muscle coverage will have a good blood supply which will improve fracture healing ability

Type of fracture-
Oblique fractures have a larger surface area than a transverse fracture, improving contact to promote tissue regrowth

Poor reduction/fixation-
Good reduction to allow ‘Direct Healing’ will heal better. Poor reduction will have increased gap and callus formation and increased risk of malunion

Movement-
Movement to the fracture site will delay healing and increase risk of malunion

326
Q

what are the complications of fracture healing

A

Non-union – Complete failure of fractured bone ends to unite

Delayed union – Healing progresses slowly. Clinical union might occur but not achieved within expected time.

Malunion – Healing occurs in an abnormal position

Shortened limb – a form of malunion where the fracture hasn’t been reduced. Might be seen in cases that didn’t have veterinary intervention e.g. stray dogs

Osteomyelitis – Inflammation of the bone characterised by progressive inflammatory destruction of new bone. Bacterial Osteomyelitis = Infection

Fracture disease – A syndrome of muscle wastage and inability to flex joints in a limb after repair. Can occur due to scar formation.

Sequestrum – piece of devitalised bone separated from bone not incorporated into repair.

Implant failure – implants are foreign material and the body’s immune system may reject the implant. Can also occur due to poor choice of implant so it’s not compatible with the bone size/shape, and patient overactivity.

327
Q

what are types of fractures

A

normal, transverse- directly across, oblique- diagonal , spiral, comminuted- numerous fragments, segmental, one segmented part, avulsed, pulling of part of bone, impacted- pressure, torus- compression with a bulge, greenstick- fracture but not all the way through the bone

328
Q

what are the principles of fracture repair

A

Restoring the continuity of the bone

Restoring length

Restoring functional shape

Maintaining soft tissue function of blood vessels, muscles and nerve supply.

329
Q

what are the 3 fixation techniques

A

External coaptation
Internal fixation
External

330
Q

what factors affect repair choice

A

Classification of fracture
Age
Size
Temperament
Underlying diseases
Cost
Expectation of owner

331
Q

what is the aim of external coaptation

A

To limit motion at a fracture sight but immobilizing the joint above and below the fracture.

332
Q

what are the methods for external coaptation

A

casts, splints, and extension splints

rubbing may occur resulting in additional issues- pressure sores

relying on natural healing

only used on stable fractures such as greenstick

333
Q

what are the ADV of external coaptation

A

Technically simpler
Economical
Non-invasive

334
Q

what are the DISADV of external coaptation

A

Limited applications
Not sufficient stabilisation
Decubital ulcers
Slow healing rate and greater callus formation
Fracture disease
Owner Compliance

if cant go above or below the external coaptation isn’t suitable

335
Q

what are the nursing considerations for coaptation

A

Aftercare advice sheets incorporating a care plan to address basic needs

Providing protective cover for example with splint covered with bandage as can get wet. examples include empty drip bags, poo bags, carrier bags, medical pet shirts /anti-chew device such as bitter spray, collar.

Monitor for swelling if too tight/chafing/staining/smell if wound being infected/slipping/collapse

Medications (Analgesia, NSAID’s, Sedatives) may chew bandage if it’s uncomfortable.

Exercise - what is temperament of animal, will need mental stimulation. to limit exercise you cant walk them. only take them out in the garden on a lead. dont let cat out. limit pet on sofa, bed, stairs. keep at crate rest or restrict to one room, puppy pen, if multiplet household, take away other animals. you have to explain the result if they don’t do this e.g. splint falling off, fracture getting worse.

Mental Stimulation- snuffle pads, slow feeder, sit with the dog, radio, toys, activities

Post-op checks

dont have px with you when discussing after care as owner wont listen properly

336
Q

what is an internal fixation

A

something that goes into the bone such as a pin, wire, plate, screw

337
Q

what are the ADV of internal fixation

A

Any closed fracture, any bone

Accurate reduction

Rigid fixation

Early return to full function – minimal risk of fracture disease

338
Q

what are the DISADV of internal fixation

A

Expensive/time consuming
Surgical Skill
Equipment
Surgery risks
Not suitable for open fractures

339
Q

what are the nursing considerations of internal fixation

A

Immobilisation if deemed appropriate- carrying?

Coaptation as protection

Surgical Site Infections (SSI’s), smell, colour of exudate (green yellow or milky purulent), necrotic tissue, inflammation with heat, swelling, temp

Ability to mobilise
sling, vet beds, memory foam mattress, manually lift the dog up to stand with 2-3 people. use rubber matts on floor, carpet to stop slipping. put on lead, does dog pee on or off lead/ certain substrate/ command to toilet?

Ability to access 5 freedoms
Patient Interference
Monitor for complications

340
Q

what is the suggested protocol for surgical site infection

A

Train staff appropriately on protocols

Protocol for cleaning, disinfection, and sterilization of Ortho Equip

Swab patient skin prior to surgery

Pre-operative surgical site preparation e.g. sterile gloves/sterile scrub materials/Chloroprep (remains on skin for 4 days after surgery) /contact time

Peri-operative measures – surgeon double gloves/facemask/cap

Post-operative surgical site care e.g. aseptically clean with sterile gloves & sterile swabs/apply dressing in theatre/residual effect Chloroprep 48hrs

Monitoring & Document Infections that do occur

Swab wound when infections occur

Antimicrobial therapies for prevention of SSI should be based on the risk of SSI

341
Q

what are the indications for external fixations

A

If there is an open contaminated fracture. Where the wound needs to be treated as a open wound and/or needs bandage changes.
If there is a concern that placing internal fixation will increase the risk of osteomyelitis or infection.

342
Q

what are the ADV of external fixations

A

Minimal instrumentation/reuseable
Minimal disruption to soft tissues/foreign materials
Can manage open wounds
Complements other techniques
Adjustable/easy to assess
Easy to remove

343
Q

what are the DISADV for external fixations

A

Soft tissue issues
Skill/difficult to apply to proximal limbs
Xrays can be hard to view
Premature pin loosening

344
Q

what are the nursing considerations for external fixations

A

Open wound care
Compression bandages for 2-3days, daily change
Cover the pins!
Air
Cage rest/lead walks
Scab formation – leave
Excess exudate – clean and see VS
Wound interference
Written instruction – what’s next?
Contact information

345
Q

what are the aims of rehabilitation

A

restoring maximum function, quality of life and independence following injury or disease.

to limit pain

build muscle

return the animal to normal function where possible- all activities of daily living

reduce recovery time

346
Q

what are the types of rehabilitation

A

physiotherapy

acupuncture

hydrotherapy

mctimoney

347
Q

who in the team are involved with rehabilitation

A

all members of the team including multi-disciplinary teams

348
Q

what types of pain are there

A

inflammatory response
articular cartilage damage
increased joint stress
muscle atrophy less ROM
decrease in exercise

349
Q

what is the aim of acupuncture

A

to promote natural body healing

350
Q

what is the method os acupuncture

A

insertion of a special needle into acupoints which are rich in nerve endings and blood vessels

351
Q

what are the effects of acupuncture

A

enhances blood circulation and tissue blood flow/ oxygenation and removal of waste products/ toxins

stimulate nervous system

reduces swelling

relives pain

encourages healing by correcting energy imbalances in the body

relaxes muscles

352
Q

what is the aim of hydrotherapy

A

take the weight off joints and enable flexion and extension

353
Q

why is a thermal effect of hydrotherapy good

A

aids muscle relaxation for exercise

354
Q

what are benefits of hydrotherapy

A

relives pain

reduction of swelling and stiffness

circulatory benefits

improved cardiovascular fitness

muscle strength

joint mobilisation

increased mental stimulation

improve gait pattern

355
Q

list some conditions hydrotherapy can be used for

A

orthopaedics
spinal
neurological
muscle atrophy
obesity
show dog conditioning
behaviour issues by relieving excess energy and promoting focus

356
Q

what is mctimoney

A

animal manipulation that’s hands on adjustments

357
Q

what does the RVN need to asses in a px

A

Assess your patient – nose to tail health check with some enhancements –
physio assessment – which includes….
Static Assessment: Posture (head, spine, tail) Conformation (spine, limbs, joint angle) Limb placement (cow hocked, wide, narrow)

Dynamic Assessment Watch in walk and trot - Spinal Movement, Posture/Tail,Limb Placement, Weight Baring, Lameness, Swing Phase, Sit to Stand

BCS!!!

Lameness scoring -which scale? Force plates/gait analysis

Measurements – pros and cons

358
Q

what are considerations for pxs

A

Companion/working
Multi animal/children
Floor
Stairs
Bedding
Exercise
Previous therapy
Aversion to water/touch/noises…

359
Q

why is hot heat useful for

A

chronic pain
relaxes tense muscle
aches
arthiritis

360
Q

what is cold heat useful for

A

acute injury
after surgery
reduces swelling
sprains
bruises
pain

361
Q

list methods to maintain temperature regulation

A

Heat pad
Bear Hugger
Bubble Wrap
Blankets
Hot Water Bottle/Microwave heated pads
Thermal socks/bubble wrap on feet
Humidifiers attached to GA circuit- warm air so preserved body temp

362
Q

what can hypothermia impact

A

recovery and depth of anaesthesia

363
Q

temperature can be affected as soon as …

A

presumed drugs administered

364
Q

what are types of physio techniques

A

Thermotherapy
Assisted standing
PROM
Massage
Turning 2-4 hourly

However, physiotherapy and rehabilitation after orthopaedic surgery can aid recovery by increasing muscle strength and reducing pain, swelling and inflammation (Connell and Monk, 2010).

Restrict blood flow – reduce swelling – reduce pain - Ice packs are wrapped in a towel to prevent damage to the skin, and then placed on the area for 10–20 minutes (Connell and Monk, 2010; Drum et al, 2015) repeatedly throughout the day. The use of ice packs on cats can be difficult because of their reluctance to lie still for long periods of time (Drum et al, 2015), and can be painful on application. However, this may be easier during the recovery period when the patient is drowsy. ice cubes

Slings/towels

If the patient was recumbent for 2 hours, they were turned onto a different recumbency to aid patient comfort, prevent decubitus ulcers and hypostatic pneumonia (Murrell and Ford-Fennah, 2011).

365
Q

what is the role of the scrubbed nurse

A

Works directly with VS within sterile field

Ensures sterility maintained and liases with
Circulating nurse

Organises sterile instruments and passes sterile
Equip to vet

366
Q

what is the role of the circulating nurse

A

Tasks performed outside sterile field
Organises nursing care with rest of team
Tasks could include –
passing instruments in sterile manner to scrubbed nurse
Organising equipment
Monitoring and recording
Consumables

367
Q

how do you prepare a theatre

A

Area checked and clean
All theatres damp dusted at start of day
Spot cleaned after each procedure
Room set appropriately for procedure
Correct equipment and instruments
Checking sterilised equipment
Equipment
Patient checked
Check lists!!!!!
Planning is key!!

368
Q

what are the pre and postoperative nursing considerations

A

Surgical preparation of skin
Positioning of patient- bratty- elevated so less chance of regurgitation. nerve damage. breed. surgery.
Equipment
Drapes – “strike-through”
Monitor patient for hypothermia and dehydration
Swabs counted and weighed for blood loss calculations
Post op bedding/environment
Post op analgesia and close observations
Wound interference
Drains – dressings
Factors that promote/delay healing

369
Q

what are the nursing considerations in a theatre

A

Aftercare advice sheets

Providing protective cover/anti-chew device

Monitor for swelling/chafing/staining/smell/slipping/collapse

Medications

Exercise

Post-op checks

369
Q

what are some anti- lick/ chew devices

A

Buster collar
Soft
Inflat
Stocking
T-shirt/underwear
Covers

370
Q

what questions should you ask the owner

A

Cats – what litter type and can they get into the tray? Lids/inco sheets
How do they drink
Decrease stress – pheromones/calming aids/things from home (label!!)
Normal behaviour? Breed type

what is the usual bedding at home
what does the animal eat
what does the animal eat out of
is there any allergies or dislikes
is the animal on meds
does the animal have any previous history
is the animal fully vaccinated
how often does the animal toilet
how active is the animal
does the animal have any preferences of males or females
what is the animals normal behaviour
does the animal have a preference of where or how they toilet
does the dog have high or low stress levels
is there any mobility issues
is there any command words
Is there a food release word
is there any grooming issues, like matts
any health conditions
has the dog had any vomiting or diarrhoea in the past 2-3 days
is there any aggressions
does the dog have issues with dogs or cats
does he react okay around strangers
when did he last eat and how much
when was the last time he defecated or urinated
any issues with respiration
why does he take any medications
how much water does he drink normally
is there any toys he gets when he is anxious
has he destroyed any bedding
has he ever been away from owner
any treats he prefers or gets after a walk etc
how fast does he eat
raw fed?

371
Q

what questions should you ask the vet

A

how did surgery go
any complications
open sutures?
how long do you want sutures in for and when do you want them removal
any medication to be given and how much
how did anaesthetics go
hypo/hyperthermic?
have you phoned owner to let them know how surgery went
any restrictions for the dog
happy for nurse to discharge px
how long do you need px to rest before toileting
how your going to manage the medical case
any particular diet
how slow/fast to give IVFT
when do they start meds
any side effects of meds
can I use ice packs
do you need me to apply a bandage such as Robert jones
how often do you need drain emptied and managed
do you tell owner or shall VN tell owner for open drain or closed drain
how do you want external fixation managed
how long should animal rest
how long should animal exercise for
is it on fluids
rate for fluids
is it needing blood work
how often is blood work needed
pain score?
does VN have to administer any med
do they need to come in for post op checks
is it allowed to jump

372
Q

what are the clinical symptoms of acute renal px

A

Sudden-onset anorexia, lethargy and depression.
Oliguria and anuria, followed by polyuria.
Vomiting and diarrhoea.
Polydipsia.
Dehydration.
Uraemic breath.
Abdominal pain.

373
Q

what is the clinical symptoms of chronic renal failure

A

Polyuria/nocturia (as the kidney loses its ability to concentrate the urine).
Polydipsia.
Uraemia – anorexia, vomiting, lethargy and depression.
Weight loss.
Dehydration.
Oral ulceration and halitosis.
Non-regenerative anaemia (due to lack of erythropoietin production by the kidney).
Hypertension.
Rubber jaw (renal hyperparathyroidism)

374
Q

what is cystitis

A

inflammation of the bladder

375
Q

what are the causes of cystitis?

A

Idiopathic
Feline lower urinary tract disease (see ‘Feline lower urinary tract disease (FLUTD)’ below)
Trauma
Urolithiasis (see ‘Urolithiasis (urinary calculi)’ below)
Neoplasia
Primary bacterial infection – often ascending, common in female dogs
Bacterial infection secondary to other diseases (e.g. diabetes mellitus, hyperadrenocorticism (Cushing’s disease), immunosuppressive infection such as FIV and FeLV).

376
Q

what are the clinical symptoms of cystitis

A

Pollakiuria = Abnormally frequent urination
Urinary tenesmus.
Haematuria.
Stranguria.
Incontinence.
Dysuria.
Inappropriate urination.
Excessive grooming.

377
Q

what are the clinical symptoms of urinary incontience

A

Loss of control over urination
Passing of urine when lying down or walking.
Urine around perineum which can cause urine scalding.

378
Q

what are the clinical signs for urolithiasis

A

Pollakiuria.
Urinary tenesmus= feeling of incomplete emptying of bladder after urination
Haematuria.
Dysuria.
Distended bladder.

379
Q

what are the clinical symptoms of feline lower urinary tract disease

A

Distress – vocalizing and signs of abdominal pain
Anuria
Distended hard bladder
Clinical signs of renal damage
Anorexia and vomiting
Lethargy and depression
Dehydration
Collapse and death if untreated.
Hyperkalaemia- high K+ levels
kidney damage

380
Q

what are the nursing interventions for urinary pxs

A

Record abnormal output, appearance, dipstix/SG results, micturition posture and frequency- collecting urine and comparing what’s normal, volume, appearance
Express bladder/catheterise where indicated
Use aseptic techniques
Take steps to reduce trauma
Regularly check bladder size- not commonly used now a days as bladder may burst, catheter siting etc. Flush if required
Empty bag every 4hrs, measuring output where required
Check lines are not kinked or blocked, bag not full. urine will go on animal if not closed catheter.
Prevent self-mutilation
Keep patient and kennel clean and dry, use kennel liners under vet bed
Clip perineum and apply barrier cream where risk of scalding
Provide accurate medication

381
Q

what equipment and prep is needed of a catheter

A

Equipment
Analgesia
Sedate/GA
Gloves
Catheter
3 way tap
Closed urinary system
Sterile lubricant
Syringe
Kidney dish
Saline flush
Suture material & instrument

Preparation
Bloods
GA
Clean site

382
Q

what additional care is needed for a catheterisation

A

Maintain fluid therapy, observe for fluid overload & care if potassium added!
Monitor fluid ins+outs
Assisted feeding
Palatable diets, warming food
Soft diets
Hand feeding
Pain management
Feeding tubes
Maintain body temperature
Stress reduction

383
Q

how do you evaluate a catheterisation

A

Monitoring urine output (1-2ml/kg/hr)
Washable litter
Weighing newspaper/kennel liner
Catheter attached to closed system - collection bag
Output, appearance, dipstix/SG results, micturition posture and frequency, signs of UTI infection
Monitor body weight, condition and muscle mass
Monitor hydration status
Monitor water intake
Pain levels and vital signs

384
Q

what is SG used for

A

evaluate kidney function

385
Q

what is the SG for dogs

A

1.001-1.060

386
Q

what is the SG for cats

A

1.005-1.080

387
Q

In animals with dehydration and normal renal function, urine specific gravity should be..?

A

> 1.030 in dogs and >1.035 in cats.

388
Q

what are some further care to provide for urinary pxs

A

Appropriate diet (restricted protein and phosphate for CRF, restricted minerals for urolithiasis), ideally wet diet

Encourage increased water intake
Fresh water ad lib
Broth (salt-free)
Water fountain

Renal nurse clinics
Monitor food and fluid intake, BW, BCS & MCS
Blood sampling, signs of deterioration and IRIS staging
BP monitoring

389
Q
A