ADULT MSD INTRO Flashcards

1
Q

it is caused by injury or disease of one or more cranial or spinal nerve

A

Flaccid Dysarthria

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2
Q

Reflect problems in the nuclei, axons, or neuromuscular junctions that make up the motor units of the final common pathway (FCP)

A

Flaccid Dysarthria

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3
Q

Primary distinguishing deviant speech characteristics in flaccid dysarthria

A

muscle weakness and reduced muscle tone, and their e ffects on the speed, range and accuracy of speech movements.

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4
Q

Flaccid Dysarthria involves problem in ___

A

Neuromuscular execution

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5
Q

FD Primary clinical characteristics

A
  • Weakness
  • Hypotonia
  • Diminished reflexes
  • Atrophy
  • Fasciculations and fibrillations
  • Progressive weakness with use
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6
Q

it would involved lesion in the lower motor neuron (LMN)

A

Flaccid Dysarthria

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7
Q

Give me at least 2 of Etiologies of Flaccid Dysarthria

A

● Congenital: in your childhood MSD; present from birth
● Demyelinating
● Infectious/inflammatory
● Degenerative
● Metabolic
● Neoplastic
● Traumatic
● Vascular diseases: such as stroke

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8
Q

What are the Clusters of Abnormal Speech Characteristics in Flaccid Dysarthrias

A

Phonatory and resonantory incompetence. Phonatory-prosodic insufficiency

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9
Q

Breathiness, short phrases, audible inspiration

A

Phonatory Incompetence (FD)

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10
Q

Hypernasality, imprecise consonants, nasal emission, short phrases

A

Resonatory incompetence

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11
Q

Harsh voice, monoloudness, monopitch

A

Phonatory-Prosodic Insufficiency

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12
Q

Produced by bilateral damage to the direct and indirect activation pathways of the central nervous system (CNS)

A

Spastic Dysarthria

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13
Q

What makes Spastic Dysarthria different?

A

It is a combined e ect of weakness and spasticity in a manner that slows movement and reduces its range and force.

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14
Q

Spastic Dysarthria involves a problem in

A

Neuromuscular execution

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15
Q

Loss of fine, skilled movement, Hypotonia
Weakness (distal > proximal) Absent abdominal reflexes Babinski sign Hyporeflexia

A

Direct Activation Pathway (Pyramidal tract)

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16
Q

Increased muscle tone, Spasticity, Clonus, Decorticate or decerebrate posture Hyperactive stretch reflexes, Hyperactive gag reflex

A

Indirect Activation Pathway, extrapyramidal tract

17
Q

people with spastic paralysis they common exhibit

A

decreased skill in movement and weakness from direct activation pathway. They also exhibit increased muscle tone and spasticity from indirect activation pathway

18
Q

Give me at least two of Etiologies: Spastic Dysarthria

A

● Degenerative
● Vascular
● Congenital
● Traumatic
● Inflammatory
● Toxic and metabolic diseas

19
Q

Patient complaint:

● Speech is slow and e ortful
● Fatigue with speaking
● Must speak more slowly to be understood, but the often admit that they are unable to speak any faster
● Swallowing complaints
● Lowered gag reflex threshold is increased
gagging when brushing teeth
● Drooling
● Pseudobulbar a ect

A

Spastic Dysarthria

20
Q

May be manifest in any or all of the respiratory, phonatory, resonatory, and articulatory levels of speech, but its characteristics are most evident in articulation and prosody

A

Ataxic Dysarthria

21
Q

Reflects the e ects of incoordination and perhaps reduced muscle tone, the products of which are slowness and inaccuracy in the force, range, timing, and direction of speech movements

A

Ataxic Dysarthria

22
Q

Reflects problem in motor control

A

Ataxic Dysarthria

23
Q

Most commonly associated with bilateral or di use cerebellar disease

A

Ataxic Dysarthria

24
Q

Give me at least 1 of Etiologies: Ataxic Dysarthria

A

Damages the cerebellum or cerebellar control circuit can cause ataxic dysarthria
○ degenerative
○ demyelinating
○ vascular
○ neoplastic
○ inflammatory/infectious
○ endocrine
○ structural
○ traumatic
○ immune-mediated
○ toxic or metabolic diseases

25
Q

Patient Perceptions and Complain:

Slurred, they also often refer to the drunken quality of their speech, either as they perceive it or as others have commented

an inability to coordinate their breathing with speaking

Reduced intelligibility

A

Ataxic Dysarthria

26
Q

manifest in any or all of the respiratory, phonatory, resonatory, and articulatory levels of speech, but its characteristics are most evident in voice, articulation, and prosody

A

Hypokinetic dysarthria

27
Q

Reflects the e ects of rigidity, reduced force and range of movement, and slow individual but sometimes fast repetitive movements on speech

A

Hypokinetic Dysarthria

28
Q

Patient Perceptions and Complaints
● Report that others tell them their voice is quieter or weak, they often deny or minimize such changes themselves
● Rate is too fast or that words are indistinct are common
● “Hard to get speech started”

A

Ataxic Dysarthria