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adult language Flashcards

1
Q

neurologically based language disorder caused by various types of neuropathies

A

aphasia

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2
Q

neuropathies that cause aphasia

A

CVA, tumors, hydrocephalus, hypoxia, anoxia, infections

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3
Q

caused by a block or interrupted blood supply to the brain

A

ischemic cva

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4
Q

caused by bleeding in the brain due to ruptured blood vessels with hypertension

A

hemorrhagic

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5
Q

types of nonfluent aphasias

A

global
broca’s
transcortical motor aphasia
mixed transcortical

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6
Q

site of lesion for global aphasia

A

Middle Cerebral artery

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7
Q

global aphasia results in

A

severe verbal expression
speech production and comprehension deficits
minimal to noncommunication

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8
Q

broca’s site of lesion

A

posterior inferior left frontal lobe

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9
Q

characteristics of broca’s aphasia

A

verbal expression - decreased length of utterance, nouns and verbs, reduced prosody
auditory comp - relative strength
reading comp - similar to auditory
written expression - predom of content words

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10
Q

broca’s strengths

A

manage to communicate, relevant responses, aware of speech problems

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11
Q

site of lesion for transcortical motor aphasia

A

anterior superior left frontal lobe

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12
Q

characteristics of transcortical motor

A

similar to broca’s but retain ability to repeat

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13
Q

lesion for mixed transcortical

A

arterial border of the brain; damage spares and isolates broca’s

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14
Q

characteristics of mixed transcortical

A

limited spontaneous speech
automatic communication
severe echolalia
severely impaired reading, reading comp, and writing

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15
Q

assessment for aphasia

A

patient interview, chart review
aud comp - point to items, follow commands, y/n
verbal expression - fluent vs nonfluent automatic speech, sentence completion, rep
reading - letter/word/phrase/sentence
written - spelling, grammatical structure

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16
Q

formal aphasia assessment

A

BDAE
WAB
BASA

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17
Q

types of fluent aphasia

A

wernickes
transcortical sensory
conduction
anomic
subcortical

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18
Q

wernicke’s lesion

A

usually the posterior one third of the temporal gyrus

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19
Q

characteristics of wernicke’s aphasia

A

verbal expression - effortless, prosodic
pressed for speech, poor rep
auditory comp - impaired at the word level
reading comp - deficits at word level
written - mirrors verbal expression

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20
Q

strengths of wernicke’s

A

comprehension of repetitive and personal information, maintain correct syntax/artic/prosody

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21
Q

transcortical sensory aphasia lesion

A

temporoparietal region of brain

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22
Q

characteristics of transcortical sensory

A

similar to wernickes but able to repeat and oral reading is relatively spared

23
Q

conduction aphasia site of lesion

A

supramarginal gyrus, upper temporal lobe, lower parietal, damage to arcuate fasiculus

24
Q

conduction aphasia characteristics

A

verbal expression - fluent output, paraphasias, anomia
reading - silent relatively spared
written expression - fluent output, mirrors verbal skills
profound deficits in longer reps

25
anomic aphasia lesion
inferior temporal cortex
26
anomic characteristics
auditory comprehension - relatively spared word finding deficits
27
subcortical aphasia lesion
basal ganglia, cerebellum, thalamus
28
right hemisphere dysfunction characteristics
cognitive communication disorder right hemisphere handles holistic picture, big picture and visual spatial info
29
etilogies for RHD
CVA tumor trauma
30
characteristics of RHD deficits
do not respond to people/objects to the left
31
right hemishpere disorder characteristics
cognition - attention (easily distracted), memory, attention, orientation, executive function (problem solving, reasoning, organization) left neglect visual perceptual skills topographical info affect/emotions dysarthria dysphagia
32
assessment for rhd
patient info, identity strengths/weaknesses, does patient have cog/comm deficits informal: interview to assess understanding, attention, discourse, prag cookie theft cancellation, clock drawing mini right brain inventory BURNS RBANS MOAT BADS CLQT RIPA
33
treatment for severe RHD
attention/arousal short sessions raise head of hospital to increase arousal nelgect: while recovering move things to right side, move the nurse situation
34
treatment of neglect for mod
compensation of strategies communication partners stand to the left important objects to left neglect orientation and schedules left
35
treatment of moderate rhd prosody and pragmatics
explain patients tone to family it does not match their meaning oftentimes have patient identify incorrect aspects of script
36
rhd treatment of mild neglect
scanning in community activities math problems paragraphs
37
rhd treatment of prosody and pragmatics
verbalize emotional states identify emotional education role play pause reduce pitch
38
tbi is a
cognitive communication disorder
39
focal or open-head injury
open or penetrative injury extends brain tissue
40
diffuse or close headed
coup/contra-coup, shearing, DAI
41
secondary to tbi
hematomas hemorrhages hydrocephalus infarct seizure hypoxia
42
characteristics of tbi defecits
cognition - orientation, memory, visual spatial, organization, problem solving, pragmatics, confabulations, reasoning
43
GCS 13-15
mild LOC- 20 minutes or less PTA - 60 minutes or less
44
GCS 9-12
moderate loc 6hrs or less pta 24hrs or less
45
gcs 8 or less
severe LOC extended Pta 24hrs unarousable state
46
rancho levels
I no response to external stimuli II generalized response III localized but purposeful IV confused and agitated V confused non-agitated VI confused appropriate VII automatic but appropriate VIII Purposeful and appropriate
47
etiologies
falls are the most common cause automobile accidents head striking assaults
48
treatment for early stages of tbi
educate train caregivers facilitating response to environment med management usually in OP
49
patient management for tbi
structure environment no stimulation one thing at a time describe actions maintain routine monitor response
50
middle stage assessment for tbi
GOAT O-log
51
treatment principles for mild stage tbi
minimize agitation sustain attention orientation promote safety increase independence
52
tbi late stages informally assess
selective/alt attention visual and verbal memory executive function communication: discourse
53
formal late stage tbi assessment
SCATBI RIPA CLQT GOD
54

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