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adult language Flashcards

1
Q

neurologically based language disorder caused by various types of neuropathies

A

aphasia

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2
Q

neuropathies that cause aphasia

A

CVA, tumors, hydrocephalus, hypoxia, anoxia, infections

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3
Q

caused by a block or interrupted blood supply to the brain

A

ischemic cva

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4
Q

caused by bleeding in the brain due to ruptured blood vessels with hypertension

A

hemorrhagic

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5
Q

types of nonfluent aphasias

A

global
broca’s
transcortical motor aphasia
mixed transcortical

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6
Q

site of lesion for global aphasia

A

Middle Cerebral artery

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7
Q

global aphasia results in

A

severe verbal expression
speech production and comprehension deficits
minimal to noncommunication

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8
Q

broca’s site of lesion

A

posterior inferior left frontal lobe

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9
Q

characteristics of broca’s aphasia

A

verbal expression - decreased length of utterance, nouns and verbs, reduced prosody
auditory comp - relative strength
reading comp - similar to auditory
written expression - predom of content words

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10
Q

broca’s strengths

A

manage to communicate, relevant responses, aware of speech problems

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11
Q

site of lesion for transcortical motor aphasia

A

anterior superior left frontal lobe

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12
Q

characteristics of transcortical motor

A

similar to broca’s but retain ability to repeat

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13
Q

lesion for mixed transcortical

A

arterial border of the brain; damage spares and isolates broca’s

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14
Q

characteristics of mixed transcortical

A

limited spontaneous speech
automatic communication
severe echolalia
severely impaired reading, reading comp, and writing

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15
Q

assessment for aphasia

A

patient interview, chart review
aud comp - point to items, follow commands, y/n
verbal expression - fluent vs nonfluent automatic speech, sentence completion, rep
reading - letter/word/phrase/sentence
written - spelling, grammatical structure

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16
Q

formal aphasia assessment

A

BDAE
WAB
BASA

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17
Q

types of fluent aphasia

A

wernickes
transcortical sensory
conduction
anomic
subcortical

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18
Q

wernicke’s lesion

A

usually the posterior one third of the temporal gyrus

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19
Q

characteristics of wernicke’s aphasia

A

verbal expression - effortless, prosodic
pressed for speech, poor rep
auditory comp - impaired at the word level
reading comp - deficits at word level
written - mirrors verbal expression

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20
Q

strengths of wernicke’s

A

comprehension of repetitive and personal information, maintain correct syntax/artic/prosody

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21
Q

transcortical sensory aphasia lesion

A

temporoparietal region of brain

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22
Q

characteristics of transcortical sensory

A

similar to wernickes but able to repeat and oral reading is relatively spared

23
Q

conduction aphasia site of lesion

A

supramarginal gyrus, upper temporal lobe, lower parietal, damage to arcuate fasiculus

24
Q

conduction aphasia characteristics

A

verbal expression - fluent output, paraphasias, anomia
reading - silent relatively spared
written expression - fluent output, mirrors verbal skills
profound deficits in longer reps

25
Q

anomic aphasia lesion

A

inferior temporal cortex

26
Q

anomic characteristics

A

auditory comprehension - relatively spared
word finding deficits

27
Q

subcortical aphasia lesion

A

basal ganglia, cerebellum, thalamus

28
Q

right hemisphere dysfunction characteristics

A

cognitive communication disorder
right hemisphere handles holistic picture, big picture and visual spatial info

29
Q

etilogies for RHD

A

CVA
tumor
trauma

30
Q

characteristics of RHD deficits

A

do not respond to people/objects to the left

31
Q

right hemishpere disorder characteristics

A

cognition - attention (easily distracted), memory, attention, orientation, executive function (problem solving, reasoning, organization)
left neglect
visual perceptual skills
topographical info
affect/emotions
dysarthria
dysphagia

32
Q

assessment for rhd

A

patient info, identity strengths/weaknesses, does patient have cog/comm deficits
informal: interview to assess understanding, attention, discourse, prag
cookie theft
cancellation, clock drawing
mini right brain inventory
BURNS
RBANS
MOAT
BADS
CLQT
RIPA

33
Q

treatment for severe RHD

A

attention/arousal
short sessions
raise head of hospital to increase arousal
nelgect: while recovering move things to right side, move the nurse situation

34
Q

treatment of neglect for mod

A

compensation of strategies
communication partners stand to the left
important objects to left
neglect
orientation and schedules left

35
Q

treatment of moderate rhd prosody and pragmatics

A

explain patients tone to family
it does not match their meaning oftentimes
have patient identify incorrect aspects of script

36
Q

rhd treatment of mild neglect

A

scanning in community activities
math problems
paragraphs

37
Q

rhd treatment of prosody and pragmatics

A

verbalize emotional states
identify emotional
education role play
pause
reduce pitch

38
Q

tbi is a

A

cognitive communication disorder

39
Q

focal or open-head injury

A

open or penetrative
injury extends brain tissue

40
Q

diffuse or close headed

A

coup/contra-coup, shearing, DAI

41
Q

secondary to tbi

A

hematomas
hemorrhages
hydrocephalus
infarct
seizure
hypoxia

42
Q

characteristics of tbi defecits

A

cognition - orientation, memory, visual spatial, organization, problem solving, pragmatics, confabulations, reasoning

43
Q

GCS 13-15

A

mild
LOC- 20 minutes or less
PTA - 60 minutes or less

44
Q

GCS 9-12

A

moderate
loc 6hrs or less
pta 24hrs or less

45
Q

gcs 8 or less

A

severe
LOC extended
Pta 24hrs
unarousable state

46
Q

rancho levels

A

I no response to external stimuli
II generalized response
III localized but purposeful
IV confused and agitated
V confused non-agitated
VI confused appropriate
VII automatic but appropriate
VIII Purposeful and appropriate

47
Q

etiologies

A

falls are the most common cause
automobile accidents
head striking
assaults

48
Q

treatment for early stages of tbi

A

educate train caregivers
facilitating response to environment
med management
usually in OP

49
Q

patient management for tbi

A

structure environment
no stimulation
one thing at a time
describe actions
maintain routine
monitor response

50
Q

middle stage assessment for tbi

A

GOAT
O-log

51
Q

treatment principles for mild stage tbi

A

minimize agitation
sustain attention
orientation
promote safety
increase independence

52
Q

tbi late stages informally assess

A

selective/alt attention
visual and verbal memory
executive function
communication: discourse

53
Q

formal late stage tbi assessment

A

SCATBI
RIPA
CLQT
GOD

54
Q
A

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