Adult Hip Conditions & Surgery Flashcards

1
Q

What is the surgical sieve?

A
V: vascular
I: infective/inflammatory
T: traumatic
A: autoimmune
M: metabolic
I: iatrogenic/idiopathic
N: neoplastic
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2
Q

Name the layers of cartilage and bone usually found in the head of the femur

A

Hyaline cartilage
Subchondral Bone
Cancellous Bone

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3
Q

What condition is characterised by altered morphology of the femoral neck and/or acetabulum causing them to rub together on movement?

A

Femoroacetabular Impingement Syndrome

FAI

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4
Q

What movements does FAI specifically affect?

A

F - Flexion of hip
A - Adduction of hip
I - Internal Rotation of hip

remember this as its also FAI

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5
Q

What is the difference between a CAM femoroacetabular impingement and a Pincer impingement?

A

CAM = neck of femur has extra bone growing on outside causing deformity

Pincer = acetabulum has extra bone growing down towards femur causing the deformity

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6
Q

What type of FAI is most common?

A

Mixed CAM and Pincer Impingement

=> deformity in both femoral neck and the acetabulum

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7
Q

What type of FAI is more common in males?

A

CAM

Usually young athletic males

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8
Q

What can a CAM deformity in the femoral neck be related to in a patients history?

A

Previous Slipped Upper Femoral Epiphysis (SUFE)

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9
Q

What type of FAI is more common in females and why could this be?

A

Pincer Impingement
this is due to acetabular overhang/ an excessively deep acetabulum

=> females have wider hips and suffer from this deformity more

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10
Q

What do both types of FAI cause?

A

damage/ tears the labrum
damage to cartilage
osteoarthritis in later life

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11
Q

How do patients with femoroacetabular impingement usually present?

A
Activity related pain in the groin 
(especially on flexion and rotation)
Difficulty sitting
C sign positive
FADIR provocation test positive
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12
Q

What methods of imaging are used in the diagnosis of FAI?

A

Radiographs
CT
MRI (better for visualising damage to labrum and bony oedema)

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13
Q

How is FAI managed?

A

Observation if asymptomatic

CAM and Pincer:
Remove excess bone deformity and debride labral tears

Joint replacement in Secondary OA

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14
Q

What is Avascular Necrosis (AVN) in the head of the femur?

A

Failure of the blood supply to the femoral head

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15
Q

What are the two types of AVN?

A

Idiopathic

Trauma-related

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16
Q

Describe the pathophysiology of idiopathic AVN

A
  • coagulation of intraosseous microcirculation
  • venous thrombosis causes retrograde arterial occlusion
    => intraosseous hypertension
  • decreased blood flow to femoral head
    => necrosis
  • chondral fracture and collapse
17
Q

In traumatic AVN, what artery is usually compromised?

A

Medial Femoral Circumflex artery

18
Q

How do patients with AVN often present?

A
  • Insidious onset groin pain
  • Exacerbated by stairs or impact
  • Examination = usually normal
  • *unless disease has advanced to collapse/OA**
19
Q

What imaging methods are used in AVN, and what do they show?

A

Radiographs
(ALTHOUGH these = often normal in early disease)

MRI scan is most sensitive/specific

20
Q

What sign of AVN in children (Perthe’s disease) can be seen on X-Ray ?

A

Hanging rope sign

Thin sclerotic line across femoral neck

21
Q

How many stages of AVN have been identified? What range of these stages are reversible and irreversible?

A

Stages 0-VI

Stages 0-II = Reversible
Stages III-VI = Irreversible

22
Q

How is AVN commonly treated?

A

Drilling into the femoral head to relieve pressure and restore blood flow

Bone grafting

Rotational osteotomy

Total Hip Replacement (THR)

23
Q

What is Idiopathic Transient Osteonecrosis of the Hip (ITOH)?

A
  • Local hyperaemia (excess blood in vessels)
  • impaired venous return
  • marrow oedema
  • increased intramedullary pressure
24
Q

How do patients with ITOH usually present?

A
  • Progressive groin pain over several weeks
  • Difficulty weight bearing
  • Usually unilateral
25
Who is most likely to get ITOH?
Males > Females 2 groups: - Middle aged men - Pregnant women in third trimester
26
What investigations are used to diagnose ITOH?
Elevated ESR Radiographs: Lucent bone on X-Ray Joint space = preserved MRI (gold standard) Bone scan
27
How is ITOH managed?
- Self-limiting condition that resolves in 6-9 months - Analgesia - Protected weight bearing to avoid stress fracture
28
What is trochanteric bursitis?
Repetitive trauma caused by IT band tracking over trochanteric bursa => causes inflammation
29
What type of patients usually present with trochanteric bursitis?
Female patients | Young runners and older patients may be linked to gluteal cuff syndrome
30
How do patients with Trochanteric bursitis present?
Pain on the LATERAL ASPECT of the hip | Pain on palpation of greater trochanter
31
What imaging modalities are used in trochanteric bursitis?
Clinical diagnosis!!! => Radiographs usually unremarkable Visible on MRI but not usually needed (expensive)
32
How is trochanteric bursitis usually treated?
Analgesia NSAIDs Physiotherapy Steroid injection **No proven benefit from surgery!**
33
What conditions can lead to potential secondary OA?
``` DDH SUFE Septic arthritis AVN FAI Trauma ```
34
What is osteoarthritis (OA)?
Degenerative disease of synovial joints => progressive loss of articular cartilage Inflammatory changes in capsule lead to: - thickening - tightness
35
What type of people usually get osteoarthritis?
Female > Males - older age - Genetic element - Pre-existing hip disease
36
How do patients with OA tend to present?
``` Groin pain Worse on activity Pain at night Start up pain Stiff on testing ROM ```
37
What should you asses
Level of symptoms and impact on quality of life Medical comorbidities Social history WOULD THE PATIENT LIKE SURGERY?!
38
What is the management of osteoarthritis?
``` Analgesia Weight loss (decrease joint reaction force) Walking aids Physiotherapy if weakness identified Steroid injections THR ```