Addiction Flashcards

1
Q

What is addiction?

A

A psychological and physical dependency on a behaviour that can include ingestion of a mind-altering substance (alcohol, tobacco, heroin etc)

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2
Q

Hallmarks of addiction

A

-Dependency – potent desire, craving. -Inability to control behaviour.
-Withdrawal – negative feelings upon cessation
-Harm – continued use despite knowledge of harm
-Tolerance – more of the drug/behaviour needed to obtain the same effect.

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3
Q

Why do people appear to do recreational drugs?

A
  • Relieve boredom
  • Temporarily switch off ‘conscious’ mind
  • Become more spiritual
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4
Q

Main areas of the brain affected by addiction ‘drug hijacking’ natural reward systems of the brain

A

Hippocampus - memory
Amygdala - emotion
Nucleus accumbens - pleasure center
Pre-frontal cortex

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5
Q

Difference between strong addictive and weaker less addictive drugs on the Nucleus accumbens

A

Strong addictive drugs = effect within the nucleus accumbens = have stronger effects
Less addictive drugs ie caffine = affect the outside perimeter of the nucleus accumbens has less effects of reward centre

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6
Q

What is the key neurotransmitter involved in addiction?

A

Dopamine

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7
Q

How do individuals become addicted? in terms of psychological and social factors.

A

Psychological = low self-esteem / loneliness
Social = peer pressure
Biological =

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8
Q

In terms of early theories, explain addiction

A

Early theories would explain that addiction would occur due to the positive ‘euphoric’ effects of drugs when first taking them leading us to take them again

As well as the negative effects (withdrawal) that occur when you stop taking the drug.

(ie reinforcement) positive and negative

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9
Q

Limitations of early theory of addiction ie ‘euphoric effect’

A

Argued that not everyone’s first experience of trying drugs for the first time is pleasant. However, the desired effects are learnt over time. The pairing of the pleasurable effect of drug over time.

In terms of taking drugs to avoid negative feelings does not explain relapse after dependency and withdrawal has worn off (Lamb et al 1991)

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10
Q

Explain Incentive sensitisation theory (robinson & berridge, 1993)

A
  • Links biological (esp dopamine system) and psychological mechanisms in addiction
  • Users become sensitized to the rewarding effects of drug
    as well as cues associated with the drug
  • pleasure v craving
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11
Q

Describe how learning theory (Pavlog 1995) explains drug cues.

A

Neutral stimulus = packet of cigarettes
Unconditioned stimulus = nicotine

After frequent pairing of the two…

Packet of cigarettes becomes = conditioned stimulus
and Nicotine becomes = conditioned response

Simply = association of the drug can trigger cravings

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12
Q

How would you test Learning theory for drug addiction?

What did Townshend and Dukas (2001) study find?

A

Pictures of words associated to specific drugs
Townshend and Duka (2001)
study to see if Reaction Times to alcohol pictures differed between heavy versus occasional social drinkers

Findings: Heavy social drinkers = a significantly faster reaction to pictures of alcohol suggesting = more noticeable / important to heavy drinkers in compared to social drinkers

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13
Q

Limitations of Townshends study and possible other study’s (stafford et al 2019)

A

-difficult to replicate effects within other studies
-Another study looked at coffee consumers and found that frequent coffee consumers were significantly better at identifying the lowest odor of coffee detectable compared to non-coffee consumers. (stafford et al 2019)

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14
Q

How does addiction of highly rewarding drugs hijack the natural reward system?

A

Ingesting a highly rewarding drug - morphine/heroin is telling the brain something really good has arrived…
* …so ‘falsely’ signals some advantage/benefit
* Potent control on dulling pain/reducing appetite….add to this severe withdrawal symptoms
* Easy to see how Wanting (craving) increases, though Liking reduces

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15
Q

Describe the importance of context -siegel, 1984

A

-concluded from observation
Explained that taking drugs may not always be a simple chemical reaction
-many heroin addicts were overdosing when not in their usual environment ie home.
-often the same dosage of the drug, but the environment changed
-environment acts a preparation for the consumption of the drug
-Were not prepared = overdose.
Same effects were demonstrated on rodents.

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16
Q

Explain the Vietnam war Robbins (1993)

A

Soldiers were addicted to heroin when in Vietnam
When returned home from the war, many of the soldiers did not continue taking the drug.
Evidence for the environment.

17
Q

List some of the individual differences with addiction

A

-early childhood trauma (Enoch, 2011)
-Heritability - stronger the drug is, the stronger the heritability
-a combination of impulsion and stress (clay et al).

18
Q

Drugs and mental health - uses of cannabis in students

A

most often used for the relief of stress and tension (Kaplan (1986)
And has shown to be one of the main reasons why undergrad students consume cannabis

19
Q

Explain student cannabis and social anxiety

A

students most motivated to take cannabis the more socially / anxious they were
-not related to useage

20
Q

What model explains cannabis and psychosis

A

Vulnerability Stress model

21
Q

Explain the vulnerability stress model

A

someone might have a vulnerability with levels of stress = making them more likely to use cannabis.

22
Q

Schizophrenia and cannabis

A

heightened sensitivity to sound (hearing voices)
Extreme sensitivity to stressful events

23
Q

Psychosis and cannabis

A

Active effect = THC = paranoia / psychosis effects
One theory posits sensitization then follows, where increasing usage induces larger behavioral effects (i.e. psychosis)
Supported by rodent work where increasing cannabis (delta-9-tetrahydrocannabinol ‘THC’ main active drug,) administration led to bigger (physical) behavioural effects (Cadoni et al., 2001)

24
Q

What is psychosis

A
25
Q

What is schizophrenia

A