Acute Respiratory Problems Flashcards
What can we do for hospitalized smokers?
Welbutrin: antidepressant that helps ease nicotine withdrawal
Nicotine Patch
-SE = dizziness, sleeping problems, vivid dreams
Are cigarette quitting treatments more effective in men or women?
Men b/c women are less likely to quit b/c of weight gain that happens after quitting.
Traumatic Problems of the Respiratory System
chest trauma - pneumothorax epistaxis caused by: -dry mucous membranes -substance abuse -infections (local or systemic) -HTN -bleeding disorders -anticoagulant therapy -trauma -high flow O2
Tx for nosebleeds
- calm the person
- sit up with head slightly forward
- apply pressure for 5-15min
- seek medical attention if bleeding doesn’t stop
Vascular Problems of Respiratory System
PE (leading cause is DVT) Pulmonary HTN (increased pulmonary pressure d/t pulmonary vascular resistance to blood flow through arteries and arterioles) Cor Pulmonale (enlargement of R ventricle secondary to disease of lung, thorax or pulmonary HTN)
Pulmonary Edema
abnormal accumulation of fluid in alveoli and interstitial spaces
complication of heart and lung diseases
most common cause = left-sided HF
PE: Etiology and Pathophysiology
- blockage of one or more pulmonary arteries by thrombus, fat, or air embolus or tumor tissue
- clot in venous system into pulmonary circulation then lodges in small blood vessel and obstructs alveolar perfusion
- most often affects lower lobes (cause vessels are smaller than in upper lungs)
Types of PEs
DVT: deep vein thrombosis
VTE: venous thromboembolism (originates in one place a piece breaks off and travels until it gets stuck)
Saddle Embolus: large thrombus at arterial bifurcation
Risk Factors for PE
- immobility or reduced mobility
- surgery w/in 3 months (esp. pelvic or lower extremity)
- history of VTE
- cancer
- obesity
- oral contraceptives/hormone therapy
- smoking
- prolonged air travel
- heart failure
- pregnancy
- clotting disorders
Manifestations of PE
depends on type, size, and extent of emboli
- dysnpea (most common manifestation)
- mild-moderate hypoxemia
other symptoms include: tachypnea, cough, chest pain, hemoptysis, crackles, wheezing, fever, tachycardia, syncope, pulmonic heart sound
massive PE symptoms = change in mental status, hypotension, impending doom, death
PE Diagnostic Tools
D-Dimer (blood test): normal value <250ng/mL
- elevated w/ clot degradation
- could have false negatives with small PE as it may not be releasing enough to show up on blood work
Spiral (helical) CT scan/CT angiography or CTA:
- most common, requires contrast
- 3D pic of pulmonary vasculature
Ventilation Perfusion (V/Q) scan:
- used if pt cannot have contrast
- 2 components: perfusion scanning with radioisotope injection, ventilation scanning with radioactive gas inhaled
Additional tests to look at for PE (not diagnostic)
arterial blood gas chest x-ray electrocardiogram (ruling out MI) troponin levels b-type natriuretic peptide
Drug Therapy for PE
Anticoagulation: stops clot from getting bigger
- heparin: monitor PTT (antidote = protamine sulfate)
- warfarin: monitor PT
Fibrinolytic agents: dissolve clot
-ex) tPA and Alteplase (activase)
Drug Therapy Monitoring for PE
monitor for bleeding in unusual places:
- gums
- stool
- hematuria
- hematemesis
- bruising for no reasons
- nose bleeds
- tachycardia and hypotension
- severe HA
monitor CBC (can show bleeding we can’t see)
avoid IM injections
use electric razor
Surgical Therapy for PE
Pulmonary embolectomy for massive PE
-when pt is unstable hemodynamically and thrombolytic therapy is contraindicated
Percutaneous catheter embolectomy or endovascular u/s delivered thrombolysis
Inferior vena cava (IVC) filter: prevents migration of clots in pulmonary system
Nursing Management of PE
prevention:
- SCDs
- ambulation
- anticoagulation
immediate treatment:
- bed rest in semi fowlers
- assess cardiopulmonary status
- O2
- fluids
- medication
pt support for anxiety, dyspnea, fear
education:
- long term anticoagulation therapy
- measures to prevent VTE
- importance of follow ups
Acute Bronchitis
self-limiting inflammation of bronchi
most caused by viruses, but can be triggered by pollution, smoking, sinusitis, asthma
symptoms = cough, clear/purulent sputum, HA, fever, malaise, dyspnea, chest pain
Acute Bronchitis: Diagnosis and Tx
Diagnosis: based on assessment of breath sounds (crackles or wheezes)
Tx: symptoms relief and prevention of pneumonia
- cough suppressant, oral fluids, humidifier
- beta 2 agonist inhaler
- avoid irritants
- if due to influenza, can take antiviral within 48 hours
- see HCP sx last longer than 4 weeks
Flu: Risk Factor
- older than 50
- younger than 3
- chronic cardiac or pulmonary disease
- long-term care facility residents
- hospitalization in past year
- immunocompromised
- pregnant women in 2nd and 3rd trimester
- heath care workers (transmit it to high risk groups)
Flu: Subjective/Objective Data
abrupt onset of:
- fever
- HA
- aches
- cough
- sore throat
- fatigue
lung sounds are usually clear
symptoms last about a week (longer for elderly)
Flu: Interventions
Prevention: vaccine and sneeze protection
Supportive measures: tx symptoms
Antiviral med (Tamiflu): decreases length and severity of sx by at least 1 day (need to take w/in first 48 hours)
Flu: Complication
Secondary Bacterial Pneumonia
- pt starts to improve but then worsens
- tx w/ antibiotics is usually effective
also could get bronchitis, otitis media, sinusitis (can be treated)
Pneumonia: Definition
Acute Inflammation of alveolar spaces causing consolidation of lung tissue as alveoli fill with exudate
Gas exchange difficult -> hypoxia and hypercapnia
Can be:
lobar = affects section of lung
bronchopneumonia = affects patches throughout lung
cause can be microbes, chemical (aspiration), fungal
3 ways organisms can reach the lung:
- aspiration (normal flora lives in airway)
- inhalation: inhaling microbes like black mold
- hematogenous: spread from one place of the body to another