Acute Pain Flashcards
Neuropathic pain
Pain caused by lesion/dysfunction of nervous system
- pathological changes in pain pathway
-usually occurs after injury
Inflammatory: cancer, regional pain syndrome, herpes
Noninflammatory: postherpetic neuralgia, stump pain, trigeminal neuralgia
Pain assessment
Description: throbbing, dull, achy, crushing, shooting
Timing: started, duration, onset, exacerbating/alleviating, frequency, accompanying signs
Localization
Pain measurement
Numerical scale (0=no, 10=worst imaginable) Faces scale (kids
Acute pain
Occurs as direct result from tissue damage,
Physiological effects of pain
CV: HTN, tachycardia, dysrhythmias, MI
Pulmonary: atelectasis, V/Q mismatch, pneumonia
Endocrine: protein catabolism, hyperglycemia, fluid retention
Immune: impaired functioning
Coag: hypercoagulstion, platelet aggregation
GI: ileus
GU: urinary retention
Acute pain treatment
Opioids & nonopioids
Opioid rotation
Using different opioids interchangeably
Organ damage can decrease dose needed to achieve pain control
Essential to use lower than equianalgesic dose
Oral/parenteral doses are different
Itching? -> naloxone and Benadryl
Codeine/acetaminophen
Tylenol #2 (15/300)
Tylenol #3 (30/300)
Tylenol #5 (60/300)
Hydrocodone/acetaminophen
Norco (5, 7, 10/325)
Vicodin (5/500, 7.5/750)
Oxycodone/acetaminophen
Percocet (2.5-10/325-650)
OxyContin (no Tylenol 5-30)
Opioid side effects
N/V Allergic reaction Respiratory depression Pruritis Delirium Constipation Sedation
PO administration of analgesia
Not optimal for immediate post op pain
Opioids commonly combined with cox inhibitors
SC/IM administration for analgesia
Less desirable (pain and erratic absorption) Cyclical periods of sedation -> analgesia
IV administration of analgesia
Requires close respiratory monitoring
Common in pacu, ICU, units
Patient controlled analgesia (PCA)
Allows patient to self-administer opioids with push of button
- set dose and time period between doses