ACPO Flashcards
what is the most common cause of CPO
LSHF
what happens in left sided heart failure that leads to CPO
LV becomes unhealthy and it can’t pump effectively, blood backs up in the pulmonary veins and capillaries
increased pulmonary pressure leadings to fluid from the interstitum pushing into the lungs
why dose severe HTN lead to CPO
LV cannot overcome the force required due to high after load
what is heart failure
inability of the heart to pump blood/ supply enough blood
what are the two types of heart failure
systolic were the ventricles can’t pump hard enough during systole
and diastolic, where not enough blood fills into the ventricles during diastole
how dose heart failure lead to arrhythmia
muscle wall becomes thins or thickens
heart cell irritation or ishcemia
arrhythmia
guidelines treatment of CPO
Acquire a 12 lead ECG.
Administer 0.8 mg of GTN SL.
Continue to administer 0.8 mg GTN SL every five minutes as above if the patient is not improving. Increase the dosing interval to ten minutes if caution is required.
Apply CPAP if the patient has moderate to severe respiratory distress that is not rapidly improving (see the ‘CPAP’ section). If backup for CPAP is unavailable or delayed, apply PEEP.
Commence a GTN infusion IV if the patient is not rapidly improving.
Fentanyl in 10-20 mcg doses IV or morphine in 1-2 mg doses IV may be administered every five minutes as required for severe anxiety
role of GTN in CPO
reduces preload and after load, off loading some strain from the heart, thus reducing the volume and ejection force required for the heart to pump
role of CPAP in CPO
decreased WOB and increased cardiac output by provoking a constant trend of positive pressure treating alveolar collapse
what is negative pressure pulmonary oedema
during any form of asphyxiation, negative pressure can develop in the thoracic cavity as a result of inspiratory effort against a closed airway
common mistakes with CPO
mistaking COPD for CPO
pneumonia for CPO
fail to identify route cause e.g. STEMI
how to treat negative pressure pulmonary oedema
do not go down CPO pathway, no CPAP or GTN, treat hypoxia
how to treat a CPO who is on a long acting vasodilator
reduce GTN dose to 0.4 mg and increase dose interval to 10 mins, prioritise CPAP
presentation of severe/ life threatning ACPO
Distressed, anxious, fighting to breathe, exhausted, catatonic
Words only or unable to speak
fine crackles - full field, with possible wheeze Marked chest movement with accessory muscle use, intercostal retraction+/- tracheal tugging
alt LOC
tacky ++ or Brady late sign
moderate CPO presentation
distressed
speaking 2-3 words
crackles at bases/ mid zone
decreased SP02
accessory use
pale and diaphoretic