Abnormal psych Flashcards

1
Q

Discuss two explanations for one disorder/etiology

Introduction

A
  • Define Major depressive disorder–> (MDD) is a a mental disorder charcterized by **persistent feelings of sadness, reduced interest in activities, feelings of worthlessness, low-selfesteem.
    ** Multiple thories exist to underdstand depression
  • Focus–> genetic explanation and Becks cognitive model of depression.
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2
Q

Describe Becks model

A
  • impact of dyfunctional thoughts on the psychological well-being of an individual.
  • Cognitive triad–> irrational views about one-self, world and future that control the mind.
  • Miantained by three negative self-schemas:
    1. ineptness schema ´´I always fail´´
    2. Self-blame schema ´´Its always my fault´´
    2. Negative self-evaluation ´Ím worthless´´
  • Cognitive schemas (-) lead to cognitive biases.
  • Trigger faulty thinkign patterns –> individual only focus on negative feelings and ignore postive ones.
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3
Q

Empirical evidence 1 (Alloy et al)

A
  • aim: investigate the impact of one cognitive style in the developemnt of depression.
  • Sample: non-depresed studuents (half had. a history of depression)
  • test to identify cognitive style–> seperate as (HR) high risk or (LR) low risk for depression.
  • for sisx years follow up assemsnts were made–> questioanired and structured interviewd to identify symptoms of depression.
  • Results–>17% HR students developed MDD while only 1% in LR. more HR stduents relapsed comapred to LR.
    *
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4
Q

Evalaluate alloy et al study

A

L–> natural experiemnt–> no cause and effect
S—> longitudinal–> mitigates biderectional ambiguity

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5
Q

Applicability of CA

A
  • becks model served in the creation of cognitive behavioral therapy (CBT)
  • restructs faulty thinking patterns in depressive pateints by encuoraging positive thoughts and develop coping strategies to alleviate symptoms.
  • Study–> Hollon et al–> found that relpase rates were lower when treated with CBT compared to antidepressants and placebo.
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6
Q

Describe the genetic explanation

A
  • genes (inheritable segments of DNA) infuelnce speciifc physiological and psychological charcteristic—> determines susceptibility to develop depression.
  • depression–> polygenic trait (multiple genetic factors) and influenced by enviormntaql pressured.
  • This is linked to the 5 HTT gene which is a serotonin transporter, specifically involved in the uptake of serotonin from the synapse.
  • genes interact with enviormental stressors, trigger their expression through epigenetics.
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7
Q

Study: Caspi et al

A
  • aim: investigate the role of the transporter gene 5 HTT in depression and genetic inheritance.
  • It aimed to deduce which alleles are linked to a higher or lower risk of developing MDD.
  • The sample consisted of New Zealand participants which had their alleles measured and were grouped according to either having: two short alleles, two long alleles and one short and one long.
  • The participants had to answer a questionnaire which measured any stressful life events which may have triggered MDD.
  • A correlational analysis was then conducted between allele length and depression and stressful life events.
  • The results showed that participants with 2 short alleles had more depressive symptoms in response to stressful life events comparison to those with long alleles . This meant that the 5 HTT gene when interacting with stressful life events means that individuals have an increased risk of developing depression and those with two short alleles have a higher risk.
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8
Q

Limitations of genetic explanation

A
  • inability to study genes in isolation. This hinders the identification of specific genes associated with depression.
  • offer a more comprehensive explanation as it considers the interplay between biological and environmental factors, which highlights the non-deterministic nature of genes.
  • depression cannot be studied through a single lens, but rather through an integrative approach of social, cognitive, and biological factors.
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9
Q

Discuss one etiology of abnormal psychology

Introduction:

A
  • Etiology can be explained as a cause of a disease or disorder
    * Major depressive disorder (MDD) is one of the major affective disorders
  • Chartacterized by:
    1. persistent feelings of sadness
    2. reduced interests in activiies
    3. worthlessness
    4. low-self esteem
    5. changes in apetite
    6. sleep disturbances
  • Cognitive theory can be expplained can be explained through mental schemas
  • FOCUS–> This essay will discuss the cognitive etiology for depression.
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10
Q

Describe Becks model

A
  • impact of dysfunctional thoughts on individuals well-being
  • Cognitive triad–> individual have irrational views about themselves, future and the world.
  • Cognitive triad is maintained by negative self-schemas.
  1. self-blame–> ´Íts always my fault´´
  2. ineptness–> ´Í always fail´´
  3. negative self evaluation ´´I am worthless´´
  • Negative schemas lead to cognitive biases–> faulty thinking patterns.
  • faulty thinking patterns negelect psoitive thoughts and replace them with negative ones.
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11
Q

Empirical evidence: Alloy et al

A
  • investigate if ones thinking pattern affect the onset of depression.
  • RM–> longitudinal quasi experiment.
  • participants: non-depressed students–> half had history of depression.
  • given test to identify if they were high or low risk for depression.
  • assesemnt for 6 years–> questionaires and interviews to identify symtoms of depression.
  • Results: 17% pf HR developed depression and only 1% in LR. In group of history with depression 29% HR relapsed and 6% in LR relapsed.
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12
Q

Evaluation of study 1

A
  • S–> highly standarized and increases relaibility
  • L–> natural exp–> no cause effect relationship.
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13
Q

Study 2–> Joiner et al

A
  • the role of negative thinking patterns and enviormental stressors in onset of depression.
  • participants–> college students pre-post midterms
  • given three tests:
  • DAS after exams —> CCL and BDI was before exams
    1. DAS–> measures thinking patterns
    2. CCL–> thoughts llinked to depression
    3. BDI–> symptoms linked to depression.

Results:
* High DAS score–> failed exam —> increase in BDI
* High DAS + not failed–> no change in BDI
* Low Das+ fail–> no change in BDI

  • showed how neg thinking patterns and enviorment stressors cause more preveleance in MDD symptoms
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14
Q

Applicability: CBT

A
  • cognitive behavioural therapy (CBT)
  • type of therpay that helps depressive patients restructure their thinking patterns by encouraging positive thinking patterns +alleviate symptoms.

STUDY: HOLLON ET AL
* results: relapse rates of patients receving CBT was lower with 31% than antidepressants with 47% and placebo with 76%.
* suggests that CBT was long-lasting effects as it teaches patients effective coping skills that help change behavior.

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15
Q

Limitations Beck

A
  • reductionsist–> does not considedr biological aspects sociocultural aspect.
  • correltional data–> no cause-effect
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16
Q

Discuss the biological etiology of one disorder

Introduction

A
  • Etiology can be explained as a cause of a disease or disorder
    * Major depressive disorder (MDD) is one of the major affective disorders
  • Chartacterized by:
    1. persistent feelings of sadness
    2. reduced interests in activiies
    3. worthlessness
    4. low-self esteem
    5. changes in apetite
    6. sleep disturbances
  • Cognitive theory can be expplained can be explained through mental schemas
  • FOCUS–> This essay will discuss the biological etiology for depression.
17
Q

Desciption

A
  • proposes idea that genes–> inheritable segments of DNA.
  • infleunce phisological and psychological characteristics that dtemrine onset of depression.
  • MDD is a polygenic trait–> influenced by multiple factors
  • genes dont act in isolation–> interact with enviormental stressors
  • enviormental stressors trigger the expression of genes by epigenetics which increases liklyhood of having genetics.
  • One gene associated with depression is the 5HTT-gene.
  • The 5HTT gene is a seratonin transporter that is responsible in the reuptake of seratonin in the synapse.
  • Seratonin is associated with satisfaction, hapiness and optimism.
18
Q

Study 1: Caspi et al

A
  • aim: investigate the role of the transporter gene 5 HTT in depression and genetic inheritance.
  • It aimed to deduce which alleles are linked to a higher or lower risk of developing MDD.
  • The sample consisted of New Zealand participants which had their alleles measured and were grouped according to either having: two short alleles, two long alleles and one short and one long.
  • The participants had to answer a questionnaire which measured any stressful life events which may have triggered MDD.
  • A correlational analysis was then conducted between allele length and depression and stressful life events.
  • The results showed that participants with 2 short alleles had more depressive symptoms in response to stressful life events comparison to those with long alleles . This meant that the 5 HTT gene when interacting with stressful life events means that individuals have an increased risk of developing depression and those with two short alleles have a higher risk.
19
Q

Study 2: Kendler et al

A
  • investigate the role of genetics in depression using twins.
  • participants: 15,000 twin pairs
  • procedure: they performed telephone interviews to assess lifetime major depression using the DSM-IV.
  • concordance rate–> rate of probability that if one twin has – the other twin will have it as well.
  • Results: Female MZ–> concordance rate of 0.44
  • Female DZ—> rate of 0.16
  • Male Mz–> rate of 0.31
  • Male DZ–> rate of 0.11
  • As Mz share 100% of their genes then. the cordance rate is higher compared to DZ as they share 50%. Shows that MDD has a genetic component.
20
Q

Limitations of study 2

A

S—> large sample size –> high external validity
L–> as it was a phone telephone interview it may not be relaible

21
Q

Genetic explanation

A
  • difficult to identify which genes are associated with depression and how they interact with eachother.
  • genetic theory is still in investigation
  • Reductionsist–> genes are not the only explanation (cogntive and socioultural).